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Joshua D. Ohrtman, Ph.D. - Publications

Affiliations: 
University of Colorado Anschutz Medical Campus, Denver, Aurora, CO 
Area:
Excitation-contraction coupling
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10 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2017 Polster A, Perni S, Filipova D, Moua O, Ohrtman JD, Bichraoui H, Beam KG, Papadopoulos S. Junctional trafficking and restoration of retrograde signaling by the cytoplasmic RyR1 domain. The Journal of General Physiology. PMID 29284662 DOI: 10.1085/Jgp.201711879  0.694
2015 Ohrtman JD, Romberg CF, Moua O, Bannister RA, Levinson SR, Beam KG. Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus. The Journal of General Physiology. 145: 303-14. PMID 25779869 DOI: 10.1085/Jgp.201411292  0.71
2013 Polster A, Ohrtman JD, Beam KG, Papadopoulos S. The Cytoplasmic Foot of RyR1 without the Membrane Spanning Domain Targets Junctionally and Retrogradely Enhances DHPR L-Type Ca2+ Currents Biophysical Journal. 104: 445a. DOI: 10.1016/J.Bpj.2012.11.2470  0.723
2012 Polster A, Ohrtman JD, Beam KG, Papadopoulos S. Fluorescence resonance energy transfer (FRET) indicates that association with the type I ryanodine receptor (RyR1) causes reorientation of multiple cytoplasmic domains of the dihydropyridine receptor (DHPR) α(1S) subunit. The Journal of Biological Chemistry. 287: 41560-8. PMID 23071115 DOI: 10.1074/Jbc.M112.404194  0.649
2012 Polster A, Ohrtman J, Beam KG, Papadopoulos S. FRET Reveals Substantial Reorientation of the Cytoplasmic Interface of the Skeletal Muscle DHPR in the Presence of RyR1 Biophysical Journal. 102: 362a. DOI: 10.1016/J.Bpj.2011.11.1975  0.683
2010 Ohrtman JD, Beam KG, Allen PD. Both Voltage- and Ca2+-Dependent Inactivation of Cav1.2 Ca2+ Channels are Suppresed in Myotubes, Likely Because of Triad Junction Proteins other than RyR1 Biophysical Journal. 98: 516a. DOI: 10.1016/J.Bpj.2009.12.2807  0.685
2009 Bannister RA, Ohrtman JD. Reassessing the molecular mechanism of beta-adrenergic stimulation of cardiac L-type Ca2+ current. Channels (Austin, Tex.). 3: 146-8. PMID 19535897 DOI: 10.4161/Chan.3.3.8818  0.637
2009 Ohrtman JD, Beam KG. Depolarization-induced Potentiation Of Cav1.1 Does Not Require The Distal C-terminus Biophysical Journal. 96: 184a-185a. DOI: 10.1016/J.Bpj.2008.12.865  0.652
2009 Bannister RA, Ohrtman JD. Reassessing the molecular mechanism of β-adrenergic stimulation of cardiac L-type Ca2+ current Channels. 3.  0.653
2008 Ohrtman J, Ritter B, Polster A, Beam KG, Papadopoulos S. Sequence differences in the IQ motifs of CaV1.1 and CaV1.2 strongly impact calmodulin binding and calcium-dependent inactivation. The Journal of Biological Chemistry. 283: 29301-11. PMID 18718913 DOI: 10.1074/Jbc.M805152200  0.705
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