Year | Citation | Score | |||
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2006 | Loewen CA, Lee SM, Shin YK, Reist NE. C2B polylysine motif of synaptotagmin facilitates a Ca2+-independent stage of synaptic vesicle priming in vivo. Molecular Biology of the Cell. 17: 5211-26. PMID 16987956 DOI: 10.1091/Mbc.E06-07-0622 | 0.805 | |||
2006 | Loewen CA, Royer SM, Reist NE. Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations but calcium-binding motif mutants do not. The Journal of Comparative Neurology. 496: 1-12. PMID 16528727 DOI: 10.1002/cne.20868 | 0.805 | |||
2005 | Loewen CJ, Levine TP. A highly conserved binding site in vesicle-associated membrane protein-associated protein (VAP) for the FFAT motif of lipid-binding proteins. The Journal of Biological Chemistry. 280: 14097-104. PMID 15668246 DOI: 10.1074/jbc.M500147200 | 0.309 | |||
2002 | Mackler JM, Drummond JA, Loewen CA, Robinson IM, Reist NE. The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivo. Nature. 418: 340-4. PMID 12110842 DOI: 10.1038/Nature00846 | 0.54 | |||
2001 | Loewen CA, Mackler JM, Reist NE. Drosophila synaptotagmin I null mutants survive to early adulthood. Genesis (New York, N.Y. : 2000). 31: 30-6. PMID 11668675 DOI: 10.1002/Gene.10002 | 0.531 | |||
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