Tom Van Lee, Ph.D. - Publications

Affiliations: 
Molecular and Human Genetics Baylor College of Medicine, Houston, TX 
Area:
Notch signaling, Developmental Glycobiology

22 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2022 Shields A, Amcheslavsky A, Brown E, Lee TV, Nie Y, Tanji T, Ip YT, Bergmann A. Toll-9 interacts with Toll-1 to mediate a feedback loop during apoptosis-induced proliferation in Drosophila. Cell Reports. 39: 110817. PMID 35584678 DOI: 10.1016/j.celrep.2022.110817  0.506
2019 Ojelade SA, Lee TV, Giagtzoglou N, Yu L, Ugur B, Li Y, Duraine L, Zuo Z, Petyuk V, De Jager PL, Bennett DA, Arenkiel BR, Bellen HJ, Shulman JM. cindr, the Drosophila Homolog of the CD2AP Alzheimer's Disease Risk Gene, Is Required for Synaptic Transmission and Proteostasis. Cell Reports. 28: 1799-1813.e5. PMID 31412248 DOI: 10.1016/J.Celrep.2019.07.041  0.487
2018 Pandey A, Li-Kroeger D, Sethi MK, Lee TV, Buettner FFR, Bakker H, Jafar-Nejad H. Sensitized genetic backgrounds reveal differential roles for EGF repeat xylosyltransferases in Drosophila Notch signaling. Glycobiology. PMID 30169771 DOI: 10.1093/Glycob/Cwy080  0.746
2017 Lee TV, Pandey A, Jafar-Nejad H. Xylosylation of the Notch receptor preserves the balance between its activation by trans-Delta and inhibition by cis-ligands in Drosophila. Plos Genetics. 13: e1006723. PMID 28394891 DOI: 10.1371/Journal.Pgen.1006723  0.717
2017 Ojelade SA, Lee TV, Giagtzoglou N, Shulman JM. [O2-18-04]: THE ALZHEIMER's DISEASE SUSCEPTIBILITY GENE CD2AP REGULATES PRESYNAPTIC FUNCTION Alzheimer's & Dementia. 13: P603-P603. DOI: 10.1016/J.Jalz.2017.07.250  0.325
2016 Servián-Morilla E, Takeuchi H, Lee TV, Clarimon J, Mavillard F, Area-Gómez E, Rivas E, Nieto-González JL, Rivero MC, Cabrera-Serrano M, Gómez-Sánchez L, Martínez-López JA, Estrada B, Márquez C, Morgado Y, et al. A POGLUT1 mutation causes a muscular dystrophy with reduced Notch signaling and satellite cell loss. Embo Molecular Medicine. 8: 1289-1309. PMID 27807076 DOI: 10.15252/Emmm.201505815  0.659
2016 Lee TV, Kamber Kaya HE, Simin R, Baehrecke EH, Bergmann A. The initiator caspase Dronc is subject of enhanced autophagy upon proteasome impairment in Drosophila. Cell Death and Differentiation. PMID 27104928 DOI: 10.1038/Cdd.2016.40  0.56
2015 Lee TV, Jafar-Nejad H. O-Glucose Glycans in Drosophila Notch Signaling Glycoscience: Biology and Medicine. 849-856. DOI: 10.1007/978-4-431-54841-6_164  0.659
2014 Haltom AR, Lee TV, Harvey BM, Leonardi J, Chen YJ, Hong Y, Haltiwanger RS, Jafar-Nejad H. The protein O-glucosyltransferase Rumi modifies eyes shut to promote rhabdomere separation in Drosophila. Plos Genetics. 10: e1004795. PMID 25412384 DOI: 10.1371/Journal.Pgen.1004795  0.682
2014 LeBon L, Lee TV, Sprinzak D, Jafar-Nejad H, Elowitz MB. Fringe proteins modulate Notch-ligand cis and trans interactions to specify signaling states. Elife. 3: e02950. PMID 25255098 DOI: 10.7554/Elife.02950  0.655
2014 LeBon L, Lee TV, Sprinzak D, Jafar-Nejad H, Elowitz MB. Correction: Fringe proteins modulate Notch-ligand cis and trans interactions to specify signaling states Elife. 3. DOI: 10.7554/Elife.04998  0.657
2014 Haltom AR, Lee TV, Harvey BM, Leonardi J, Chen Y, Hong Y, Haltiwanger RS, Jafar-Nejad H. Loss of O -glucose on Notch cannot explain the rumi −/− rhabdomere attachment phenotype. Plos Genetics. DOI: 10.1371/Journal.Pgen.1004795.G002  0.64
2013 Lee TV, Sethi MK, Leonardi J, Rana NA, Buettner FF, Haltiwanger RS, Bakker H, Jafar-Nejad H. Negative regulation of notch signaling by xylose. Plos Genetics. 9: e1003547. PMID 23754965 DOI: 10.1371/Journal.Pgen.1003547  0.711
2011 Lee TV, Fan Y, Wang S, Srivastava M, Broemer M, Meier P, Bergmann A. Drosophila IAP1-mediated ubiquitylation controls activation of the initiator caspase DRONC independent of protein degradation. Plos Genetics. 7: e1002261. PMID 21909282 DOI: 10.1371/Journal.Pgen.1002261  0.647
2010 Lee TV, Takeuchi H, Jafar-Nejad H. Regulation of notch signaling via O-glucosylation insights from Drosophila studies. Methods in Enzymology. 480: 375-98. PMID 20816218 DOI: 10.1016/S0076-6879(10)80017-5  0.715
2010 Fan Y, Lee TV, Xu D, Chen Z, Lamblin AF, Steller H, Bergmann A. Dual roles of Drosophila p53 in cell death and cell differentiation. Cell Death and Differentiation. 17: 912-21. PMID 19960025 DOI: 10.1038/Cdd.2009.182  0.709
2009 Xu D, Woodfield SE, Lee TV, Fan Y, Antonio C, Bergmann A. Genetic control of programmed cell death (apoptosis) in Drosophila. Fly. 3: 78-90. PMID 19182545 DOI: 10.4161/Fly.3.1.7800  0.683
2008 Ditzel M, Broemer M, Tenev T, Bolduc C, Lee TV, Rigbolt KT, Elliott R, Zvelebil M, Blagoev B, Bergmann A, Meier P. Inactivation of effector caspases through nondegradative polyubiquitylation. Molecular Cell. 32: 540-53. PMID 19026784 DOI: 10.1016/J.Molcel.2008.09.025  0.521
2008 Lee TV, Ding T, Chen Z, Rajendran V, Scherr H, Lackey M, Bolduc C, Bergmann A. The E1 ubiquitin-activating enzyme Uba1 in Drosophila controls apoptosis autonomously and tissue growth non-autonomously. Development (Cambridge, England). 135: 43-52. PMID 18045837 DOI: 10.1242/Dev.011288  0.558
2006 Lee TV, Allison KS, Ojelade SA, Giagtzoglou N, Lah JJ, Levey AI, Seyfried NT, Shulman JM. O4-01-05: FUNCTIONAL GENETIC DISSECTION OF AN ALZHEIMER'S DISEASE SUSCEPTIBILITY NETWORK Alzheimer's & Dementia. 14: P1401-P1401. DOI: 10.1016/J.Jalz.2018.06.2913  0.301
2005 Werz C, Lee TV, Lee PL, Lackey M, Bolduc C, Stein DS, Bergmann A. Mis-specified cells die by an active gene-directed process, and inhibition of this death results in cell fate transformation in Drosophila. Development (Cambridge, England). 132: 5343-52. PMID 16280349 DOI: 10.1242/Dev.02150  0.546
2005 Cashio P, Lee TV, Bergmann A. Genetic control of programmed cell death in Drosophila melanogaster. Seminars in Cell & Developmental Biology. 16: 225-35. PMID 15797833 DOI: 10.1016/J.Semcdb.2005.01.002  0.518
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