| Year |
Citation |
Score |
| 2024 |
Rifat A, Ossola B, Bürli RW, Dawson LA, Brice NL, Rowland A, Lizio M, Xu X, Page K, Fidzinski P, Onken J, Holtkamp M, Heppner FL, Geiger JRP, Madry C. Differential contribution of THIK-1 K channels and P2X7 receptors to ATP-mediated neuroinflammation by human microglia. Journal of Neuroinflammation. 21: 58. PMID 38409076 DOI: 10.1186/s12974-024-03042-6 |
0.534 |
|
| 2023 |
Izquierdo P, Jolivet RB, Attwell D, Madry C. Amyloid plaques and normal ageing have differential effects on microglial Ca activity in the mouse brain. Pflugers Archiv : European Journal of Physiology. PMID 37966547 DOI: 10.1007/s00424-023-02871-3 |
0.747 |
|
| 2022 |
Ossola B, Rifat A, Rowland A, Hunter H, Drinkall S, Bender C, Hamlischer M, Teall M, Burley R, Barker DF, Cadwalladr D, Dickson L, Lawrence JMK, Harvey JRM, Lizio M, ... ... Madry C, et al. Characterisation of C101248: A novel selective THIK-1 channel inhibitor for the modulation of microglial NLRP3-inflammasome. Neuropharmacology. 224: 109330. PMID 36375694 DOI: 10.1016/j.neuropharm.2022.109330 |
0.727 |
|
| 2019 |
Kyrargyri V, Madry C, Rifat A, Arancibia-Carcamo IL, Jones SP, Chan VTT, Xu Y, Robaye B, Attwell D. P2Y receptors regulate microglial morphology, surveillance, and resting levels of interleukin 1β release. Glia. PMID 31520551 DOI: 10.1002/glia.23719 |
0.748 |
|
| 2019 |
Kyrargyri V, Attwell D, Jolivet RB, Madry C. Analysis of Signaling Mechanisms Regulating Microglial Process Movement. Methods in Molecular Biology (Clifton, N.J.). 2034: 191-205. PMID 31392686 DOI: 10.1007/978-1-4939-9658-2_14 |
0.726 |
|
| 2019 |
Nortley R, Korte N, Izquierdo P, Hirunpattarasilp C, Mishra A, Jaunmuktane Z, Kyrargyri V, Pfeiffer T, Khennouf L, Madry C, Gong H, Richard-Loendt A, Huang W, Saito T, Saido TC, et al. Amyloid β oligomers constrict human capillaries in Alzheimer's disease via signaling to pericytes. Science (New York, N.Y.). PMID 31221773 DOI: 10.1126/Science.Aav9518 |
0.714 |
|
| 2019 |
Izquierdo P, Attwell D, Madry C. Ion Channels and Receptors as Determinants of Microglial Function. Trends in Neurosciences. PMID 30678990 DOI: 10.1016/j.tins.2018.12.007 |
0.573 |
|
| 2018 |
Madry C, Arancibia-Cárcamo IL, Kyrargyri V, Chan VTT, Hamilton NB, Attwell D. Effects of the ecto-ATPase apyrase on microglial ramification and surveillance reflect cell depolarization, not ATP depletion. Proceedings of the National Academy of Sciences of the United States of America. PMID 29382767 DOI: 10.1073/pnas.1715354115 |
0.631 |
|
| 2017 |
Madry C, Kyrargyri V, Arancibia-Cárcamo IL, Jolivet R, Kohsaka S, Bryan RM, Attwell D. Microglial Ramification, Surveillance, and Interleukin-1β Release Are Regulated by the Two-Pore Domain K+ Channel THIK-1. Neuron. PMID 29290552 DOI: 10.1016/J.Neuron.2017.12.002 |
0.764 |
|
| 2017 |
Bledsoe D, Tamer C, Mesic I, Madry C, Klein BG, Laube B, Costa BM. Positive Modulatory Interactions of NMDA Receptor GluN1/2B Ligand Binding Domains Attenuate Antagonists Activity. Frontiers in Pharmacology. 8: 229. PMID 28536523 DOI: 10.3389/fphar.2017.00229 |
0.722 |
|
| 2016 |
Mesic I, Madry C, Geider K, Bernhard M, Betz H, Laube B. The N-terminal Domain of the GluN3A Subunit determines the Efficacy of Glycine-activated NMDA Receptors. Neuropharmacology. PMID 26777280 DOI: 10.1016/j.neuropharm.2016.01.014 |
0.799 |
|
| 2015 |
Madry C, Attwell D. Receptors, ion channels, and signaling mechanisms underlying microglial dynamics. The Journal of Biological Chemistry. 290: 12443-50. PMID 25855789 DOI: 10.1074/jbc.R115.637157 |
0.569 |
|
| 2010 |
Madry C, Haglerød C, Attwell D. The role of pannexin hemichannels in the anoxic depolarization of hippocampal pyramidal cells. Brain : a Journal of Neurology. 133: 3755-63. PMID 20940167 DOI: 10.1093/brain/awq284 |
0.635 |
|
| 2010 |
Madry C, Betz H, Geiger JR, Laube B. Potentiation of Glycine-Gated NR1/NR3A NMDA Receptors Relieves Ca-Dependent Outward Rectification. Frontiers in Molecular Neuroscience. 3: 6. PMID 20407581 DOI: 10.3389/fnmol.2010.00006 |
0.816 |
|
| 2009 |
Hwang JY, Duncan RS, Madry C, Singh M, Koulen P. Progesterone potentiates calcium release through IP3 receptors by an Akt-mediated mechanism in hippocampal neurons. Cell Calcium. 45: 233-42. PMID 19081133 DOI: 10.1016/J.Ceca.2008.10.006 |
0.794 |
|
| 2008 |
Madry C, Betz H, Geiger JR, Laube B. Supralinear potentiation of NR1/NR3A excitatory glycine receptors by Zn2+ and NR1 antagonist. Proceedings of the National Academy of Sciences of the United States of America. 105: 12563-8. PMID 18711142 DOI: 10.1073/pnas.0805624105 |
0.803 |
|
| 2008 |
Koulen P, Madry C, Duncan RS, Hwang JY, Nixon E, McClung N, Gregg EV, Singh M. Progesterone potentiates IP(3)-mediated calcium signaling through Akt/PKB. Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. 21: 161-72. PMID 18209483 DOI: 10.1159/000113758 |
0.737 |
|
| 2008 |
Schüler T, Mesic I, Madry C, Bartholomäus I, Laube B. Formation of NR1/NR2 and NR1/NR3 heterodimers constitutes the initial step in N-methyl-D-aspartate receptor assembly. The Journal of Biological Chemistry. 283: 37-46. PMID 17959602 DOI: 10.1074/jbc.M703539200 |
0.722 |
|
| 2007 |
Madry C, Mesic I, Betz H, Laube B. The N-terminal domains of both NR1 and NR2 subunits determine allosteric Zn2+ inhibition and glycine affinity of N-methyl-D-aspartate receptors. Molecular Pharmacology. 72: 1535-44. PMID 17878266 DOI: 10.1124/mol.107.040071 |
0.792 |
|
| 2007 |
Madry C, Mesic I, Bartholomäus I, Nicke A, Betz H, Laube B. Principal role of NR3 subunits in NR1/NR3 excitatory glycine receptor function. Biochemical and Biophysical Research Communications. 354: 102-8. PMID 17214961 DOI: 10.1016/J.Bbrc.2006.12.153 |
0.793 |
|
| 2005 |
Koulen P, Wei J, Madry C, Liu J, Nixon E. Differentially distributed IP3 receptors and Ca2+ signaling in rod bipolar cells. Investigative Ophthalmology & Visual Science. 46: 292-8. PMID 15623787 DOI: 10.1167/Iovs.04-0939 |
0.757 |
|
| 2005 |
Koulen P, Liu J, Nixon E, Madry C. Interaction between mGluR8 and calcium channels in photoreceptors is sensitive to pertussis toxin and occurs via G protein betagamma subunit signaling. Investigative Ophthalmology & Visual Science. 46: 287-91. PMID 15623786 DOI: 10.1167/Iovs.04-0963 |
0.761 |
|
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