Year |
Citation |
Score |
2023 |
Feng W, Lopez JR, Antrobus S, Zheng J, Uryash A, Dong Y, Beqollari D, Bannister RA, Hopkins PM, Beam KG, Allen PD, Pessah IN. Putative malignant hyperthermia mutation Ca1.1-R174W is insufficient to trigger a fulminant response to halothane or confer heat stress intolerance. The Journal of Biological Chemistry. 104992. PMID 37392848 DOI: 10.1016/j.jbc.2023.104992 |
0.717 |
|
2022 |
Peters CH, Rickert C, Morotti S, Grandi E, Aronow KA, Beam KG, Proenza C. The funny current If is essential for the fight-or-flight response in cardiac pacemaker cells. The Journal of General Physiology. 154. PMID 36305844 DOI: 10.1085/jgp.202213193 |
0.768 |
|
2022 |
Dayal A, Perni S, Franzini-Armstrong C, Beam KG, Grabner M. The distal C terminus of the dihydropyridine receptor β subunit is essential for tetrad formation in skeletal muscle. Proceedings of the National Academy of Sciences of the United States of America. 119: e2201136119. PMID 35507876 DOI: 10.1073/pnas.2201136119 |
0.821 |
|
2022 |
Perni S, Beam K. Junctophilins 1, 2, and 3 all support voltage-induced Ca2+ release despite considerable divergence. The Journal of General Physiology. 154. PMID 35089322 DOI: 10.1085/jgp.202113024 |
0.668 |
|
2022 |
Perni S, Beam KG. Voltage-induced Ca2+ release is supported by junctophilins 1, 2 and 3, and not by junctophilin 4. The Journal of General Physiology. 154. PMID 34766994 DOI: 10.1085/jgp.2021ecc22 |
0.653 |
|
2021 |
Perni S, Beam K. Neuronal junctophilins recruit specific Ca and RyR isoforms to ER-PM junctions and functionally alter Ca2.1 and Ca2.2. Elife. 10. PMID 33769283 DOI: 10.7554/eLife.64249 |
0.701 |
|
2020 |
Perni S, Beam KG. Neuronal Junctophilin 3 Can Replace Muscle Junctophilin 2 in Voltage-induced Calcium Release Biophysical Journal. 118. DOI: 10.1016/J.Bpj.2019.11.702 |
0.663 |
|
2019 |
Nelson MT, Beam K. Decision letter: A potent voltage-gated calcium channel inhibitor engineered from a nanobody targeted to auxiliary CaVβ subunits Elife. DOI: 10.7554/Elife.49253.016 |
0.365 |
|
2019 |
Perni S, Beam KG. Interactions of Junctophilins and STIM1 with ER Calcium-Releasing Channels Biophysical Journal. 116. DOI: 10.1016/J.Bpj.2018.11.2164 |
0.652 |
|
2018 |
Polster A, Dittmer PJ, Perni S, Bichraoui H, Sather WA, Beam KG. Stac Proteins Suppress Ca-Dependent Inactivation of Neuronal L-type Ca Channels. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 30201773 DOI: 10.1523/Jneurosci.0695-18.2018 |
0.728 |
|
2018 |
Polster A, Nelson BR, Papadopoulos S, Olson EN, Beam KG. Stac proteins associate with the critical domain for excitation-contraction coupling in the II-III loop of Ca1.1. The Journal of General Physiology. PMID 29467163 DOI: 10.1085/Jgp.201711917 |
0.503 |
|
2018 |
Perni S, Lavorato M, Beam KG. De Novo Reconstitution of Skeletal Muscle Voltage-Induced Calcium Release Biophysical Journal. 114: 40a. DOI: 10.1016/J.Bpj.2017.11.271 |
0.677 |
|
2018 |
Polster A, Nelson BR, Papadopoulos S, Olson EN, Beam KG. Stac Proteins Associate with the Domain of the Ca V 1.1 II-III Loop Critical for EC Coupling Biophysical Journal. 114. DOI: 10.1016/J.Bpj.2017.11.269 |
0.415 |
|
2018 |
Perni S, Beam KG. Role of Neuronal Junctophilins in Recruitment and Modulation of Voltage-Gated Calcium Channels in PM-ER Junctions Biophysical Journal. 114: 306a. DOI: 10.1016/J.Bpj.2017.11.1736 |
0.655 |
|
2017 |
Polster A, Perni S, Filipova D, Moua O, Ohrtman JD, Bichraoui H, Beam KG, Papadopoulos S. Junctional trafficking and restoration of retrograde signaling by the cytoplasmic RyR1 domain. The Journal of General Physiology. PMID 29284662 DOI: 10.1085/Jgp.201711879 |
0.827 |
|
2017 |
Perni S, Lavorato M, Beam KG. De novo reconstitution reveals the proteins required for skeletal muscle voltage-induced Ca2+ release. Proceedings of the National Academy of Sciences of the United States of America. PMID 29229815 DOI: 10.1073/Pnas.1716461115 |
0.749 |
|
2017 |
Perni S, Beam KG. Interactions between Neuronal Junctophilins and Voltage Gated Ion Channels Biophysical Journal. 112: 255a. DOI: 10.1016/J.Bpj.2016.11.1391 |
0.63 |
|
2016 |
Polster A, Nelson BR, Olson EN, Beam KG. Stac3 has a direct role in skeletal muscle-type excitation-contraction coupling that is disrupted by a myopathy-causing mutation. Proceedings of the National Academy of Sciences of the United States of America. PMID 27621462 DOI: 10.1073/Pnas.1612441113 |
0.559 |
|
2016 |
Bannister RA, Sheridan DC, Beam KG. Distinct Components of Retrograde CaV1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1. Biophysical Journal. 110: 912-21. PMID 26910427 DOI: 10.1016/J.Bpj.2015.12.031 |
0.819 |
|
2016 |
Meza U, Romberg CF, Moua O, Beam KG, Bannister RA. Functional Analysis of CaV1.1 L-Type Calcium Channels Expressed in tsA201 Cells Biophysical Journal. 110. DOI: 10.1016/J.Bpj.2015.11.2374 |
0.754 |
|
2015 |
Ohrtman JD, Romberg CF, Moua O, Bannister RA, Levinson SR, Beam KG. Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus. The Journal of General Physiology. 145: 303-14. PMID 25779869 DOI: 10.1085/Jgp.201411292 |
0.839 |
|
2015 |
Polster A, Perni S, Bichraoui H, Beam KG. Stac adaptor proteins regulate trafficking and function of muscle and neuronal L-type Ca2+ channels. Proceedings of the National Academy of Sciences of the United States of America. 112: 602-6. PMID 25548159 DOI: 10.1073/Pnas.1423113112 |
0.762 |
|
2015 |
Beqollari D, Romberg CF, Feng W, Lopez JR, Lavorato M, Perni S, Hopkins PM, Franzini-Armstrong C, Pessah IN, Allen PD, Beam KG, Bannister RA. Calcium Channel Dysfunction in a Mutant Mouse Model of Malignant Hyperthermia(CaV1.1 R174W) Biophysical Journal. 108: 504a. DOI: 10.1016/J.Bpj.2014.11.2760 |
0.84 |
|
2015 |
Feng W, Dong Y, Bannister R, Hopkins P, Franzini-Armstrong C, Beam K, Allen PD, Pessah I. Malignant Hyperthermia Susceptibility Mutation Cav1.1 R174W Dramatically Alters RyR1 Single Channel Function Biophysical Journal. 108: 270a. DOI: 10.1016/J.Bpj.2014.11.1485 |
0.814 |
|
2014 |
Polster A, Tanabe T, Moua O, Beam KG. Multiple Regions Inhibit Expression of Cav1.1 Ca2+ Channels in Non-Muscle Cells Biophysical Journal. 106. DOI: 10.1016/J.Bpj.2013.11.795 |
0.504 |
|
2014 |
Bichraoui H, Moua O, Polster A, Tanabe T, Papadopoulos S, Beam KG. Testing for Direct Interactions Between the DHPR and the RyR1 Cytoplasmic Foot Biophysical Journal. 106: 1-5. DOI: 10.1016/J.Bpj.2013.11.794 |
0.489 |
|
2013 |
Bannister RA, Beam KG. Impaired gating of an L-Type Ca(2+) channel carrying a mutation linked to malignant hyperthermia. Biophysical Journal. 104: 1917-22. PMID 23663834 DOI: 10.1016/J.Bpj.2013.03.035 |
0.755 |
|
2013 |
Bannister RA, Beam KG. Ca(V)1.1: The atypical prototypical voltage-gated Ca²⺠channel. Biochimica Et Biophysica Acta. 1828: 1587-97. PMID 22982493 DOI: 10.1016/J.Bbamem.2012.09.007 |
0.755 |
|
2013 |
Perni S, Polster A, Papadopoulos S, Beam KG, Franzini-Armstrong C. The Cytoplasmic Domain of the RyR1 Foot is Sufficient for DHPR (Cav1.1) Organization into Tetrads Biophysical Journal. 104: 446a. DOI: 10.1016/J.Bpj.2012.11.2472 |
0.726 |
|
2013 |
Bichraoui H, Polster A, Papadopoulos S, Beam KG. The Cytoplasmic Foot of RyR1 forms a Stable Homotetrameric Structure Despite Lacking the Membrane-Spanning C-Terminal Domains Biophysical Journal. 104: 4-5. DOI: 10.1016/J.Bpj.2012.11.2471 |
0.516 |
|
2013 |
Polster A, Ohrtman JD, Beam KG, Papadopoulos S. The Cytoplasmic Foot of RyR1 without the Membrane Spanning Domain Targets Junctionally and Retrogradely Enhances DHPR L-Type Ca2+ Currents Biophysical Journal. 104: 445a. DOI: 10.1016/J.Bpj.2012.11.2470 |
0.845 |
|
2013 |
Bannister RA, Moua O, Beam KG. Effects of Substituting Tryptophan for Basic Residues in the S4 Voltage-Sensing Helices of CaV1.1 Biophysical Journal. 104. DOI: 10.1016/J.Bpj.2012.11.1633 |
0.768 |
|
2012 |
Szpyt J, Lorenzon N, Perez CF, Norris E, Allen PD, Beam KG, Samsó M. Three-dimensional localization of the α and β subunits and of the II-III loop in the skeletal muscle L-type Ca2+ channel. The Journal of Biological Chemistry. 287: 43853-61. PMID 23118233 DOI: 10.1074/Jbc.M112.419283 |
0.552 |
|
2012 |
Polster A, Ohrtman JD, Beam KG, Papadopoulos S. Fluorescence resonance energy transfer (FRET) indicates that association with the type I ryanodine receptor (RyR1) causes reorientation of multiple cytoplasmic domains of the dihydropyridine receptor (DHPR) α(1S) subunit. The Journal of Biological Chemistry. 287: 41560-8. PMID 23071115 DOI: 10.1074/Jbc.M112.404194 |
0.819 |
|
2012 |
Cherednichenko G, Zhang R, Bannister RA, Timofeyev V, Li N, Fritsch EB, Feng W, Barrientos GC, Schebb NH, Hammock BD, Beam KG, Chiamvimonvat N, Pessah IN. Triclosan impairs excitation-contraction coupling and Ca2+ dynamics in striated muscle. Proceedings of the National Academy of Sciences of the United States of America. 109: 14158-63. PMID 22891308 DOI: 10.1073/Pnas.1211314109 |
0.761 |
|
2012 |
Eltit JM, Bannister RA, Moua O, Altamirano F, Hopkins PM, Pessah IN, Molinski TF, López JR, Beam KG, Allen PD. Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptor. Proceedings of the National Academy of Sciences of the United States of America. 109: 7923-8. PMID 22547813 DOI: 10.1073/Pnas.1119207109 |
0.769 |
|
2012 |
Sheridan DC, Moua O, Lorenzon NM, Beam KG. Bimolecular fluorescence complementation and targeted biotinylation provide insight into the topology of the skeletal muscle Ca ( 2+) channel β1a subunit. Channels (Austin, Tex.). 6: 26-40. PMID 22522946 DOI: 10.4161/Chan.18916 |
0.62 |
|
2012 |
Cherednichenko G, Zhang R, Bannister RA, Timofeyev V, Li N, Fritsch EB, Feng W, Barrientos GC, Schebb NH, Hammock BD, Beam KG, Chiamvimonvat N, Pessah IN. Triclosan impairs excitation-contraction coupling and Ca2+ dynamics in striated muscle (Proceedings of the National Academy of Sciences of the United States of America (2012) 109, 35, (14158 -14163) DOI:10.1073/pnas. 1211314109) Proceedings of the National Academy of Sciences of the United States of America. 109: 16393. DOI: 10.1073/Pnas.1215071109 |
0.709 |
|
2012 |
Polster A, Ohrtman J, Beam KG, Papadopoulos S. FRET Reveals Substantial Reorientation of the Cytoplasmic Interface of the Skeletal Muscle DHPR in the Presence of RyR1 Biophysical Journal. 102: 362a. DOI: 10.1016/J.Bpj.2011.11.1975 |
0.838 |
|
2011 |
Bannister RA, Beam KG. Properties of Na+ currents conducted by a skeletal muscle L-type Ca2+ channel pore mutant (SkEIIIK). Channels (Austin, Tex.). 5: 262-8. PMID 21406961 DOI: 10.4161/Chan.5.3.15269 |
0.758 |
|
2011 |
Bannister RA, Moua O, Eltit JM, Allen PD, Beam KG. Electrophysiological Analysis of Two Malignant Hyperthermia-Linked Mutations in the 1,4-Dihydropyridine Receptor α1S Subunit Biophysical Journal. 100. DOI: 10.1016/J.Bpj.2010.12.3409 |
0.738 |
|
2010 |
Beam KG, Bannister RA. Looking for answers to EC coupling's persistent questions. The Journal of General Physiology. 136: 7-12. PMID 20584887 DOI: 10.1085/Jgp.201010461 |
0.627 |
|
2010 |
Estève E, Eltit JM, Bannister RA, Liu K, Pessah IN, Beam KG, Allen PD, López JR. A malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR. The Journal of General Physiology. 135: 619-28. PMID 20479110 DOI: 10.1085/Jgp.200910328 |
0.762 |
|
2010 |
Bannister RA, Estève E, Eltit JM, Pessah IN, Allen PD, López JR, Beam KG. A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channels. The Journal of General Physiology. 135: 629-40. PMID 20479108 DOI: 10.1085/Jgp.200910329 |
0.765 |
|
2010 |
Bannister RA, Moua O, Ahern CA, Beam KG. Effects of Tryptophan Substitutions for Highly-Conserved Phenylalanines in the Skeletal Muscle L-Type Calcium Channel Biophysical Journal. 98: 5-6. DOI: 10.1016/J.Bpj.2011.11.2365 |
0.765 |
|
2010 |
Ohrtman JD, Beam KG, Allen PD. Both Voltage- and Ca2+-Dependent Inactivation of Cav1.2 Ca2+ Channels are Suppresed in Myotubes, Likely Because of Triad Junction Proteins other than RyR1 Biophysical Journal. 98: 516a. DOI: 10.1016/J.Bpj.2009.12.2807 |
0.853 |
|
2010 |
Cherednichenko G, Bannister RA, Beam KG, Pessah IN. Triclosan Uncouples Excitation-Contraction Coupling in Skeletal Myotubes Without Blocking RyR1 Biophysical Journal. 98: 304a. DOI: 10.1016/J.Bpj.2009.12.1652 |
0.742 |
|
2009 |
Bannister RA, Beam KG. Ryanodine modification of RyR1 retrogradely affects L-type Ca(2+) channel gating in skeletal muscle. Journal of Muscle Research and Cell Motility. 30: 217-23. PMID 19802526 DOI: 10.1007/S10974-009-9190-0 |
0.775 |
|
2009 |
Bannister RA, Beam KG. The cardiac alpha(1C) subunit can support excitation-triggered Ca2+ entry in dysgenic and dyspedic myotubes. Channels (Austin, Tex.). 3: 268-73. PMID 19625771 DOI: 10.4161/Chan.3.4.9342 |
0.792 |
|
2009 |
Bannister RA, Papadopoulos S, Haarmann CS, Beam KG. Effects of inserting fluorescent proteins into the alpha1S II-III loop: insights into excitation-contraction coupling. The Journal of General Physiology. 134: 35-51. PMID 19564426 DOI: 10.1085/Jgp.200910241 |
0.733 |
|
2009 |
Bannister RA, Pessah IN, Beam KG. The skeletal L-type Ca(2+) current is a major contributor to excitation-coupled Ca(2+) entry. The Journal of General Physiology. 133: 79-91. PMID 19114636 DOI: 10.1085/Jgp.200810105 |
0.765 |
|
2009 |
Ohrtman JD, Beam KG. Depolarization-induced Potentiation Of Cav1.1 Does Not Require The Distal C-terminus Biophysical Journal. 96: 184a-185a. DOI: 10.1016/J.Bpj.2008.12.865 |
0.832 |
|
2009 |
Bannister RA, Lopez JR, Allen PD, Beam KG. Does Altered Retrograde Coupling Between and RyR1 and the DHPR Contribute to Malignant Hyperthermia Biophysical Journal. 96: 1-9. DOI: 10.1016/J.Bpj.2008.12.473 |
0.76 |
|
2009 |
Bannister RA, Beam KG. The cardiac α1C subunit can support excitation-triggered Ca2+ entry in dysgenic and dyspedic myotubes Channels. 3. |
0.678 |
|
2008 |
Lorenzon NM, Beam KG. Disease causing mutations of calcium channels. Channels (Austin, Tex.). 2: 163-79. PMID 18836296 DOI: 10.4161/Chan.2.3.5950 |
0.483 |
|
2008 |
Ohrtman J, Ritter B, Polster A, Beam KG, Papadopoulos S. Sequence differences in the IQ motifs of CaV1.1 and CaV1.2 strongly impact calmodulin binding and calcium-dependent inactivation. The Journal of Biological Chemistry. 283: 29301-11. PMID 18718913 DOI: 10.1074/Jbc.M805152200 |
0.831 |
|
2008 |
Bannister RA, Grabner M, Beam KG. The alpha(1S) III-IV loop influences 1,4-dihydropyridine receptor gating but is not directly involved in excitation-contraction coupling interactions with the type 1 ryanodine receptor. The Journal of Biological Chemistry. 283: 23217-23. PMID 18556650 DOI: 10.1074/Jbc.M804312200 |
0.765 |
|
2008 |
Bannister RA, Colecraft HM, Beam KG. Rem inhibits skeletal muscle EC coupling by reducing the number of functional L-type Ca2+ channels. Biophysical Journal. 94: 2631-8. PMID 18192376 DOI: 10.1529/Biophysj.107.116467 |
0.788 |
|
2008 |
Gach MP, Cherednichenko G, Haarmann C, Lopez JR, Beam KG, Pessah IN, Franzini-Armstrong C, Allen PD. Alpha2delta1 dihydropyridine receptor subunit is a critical element for excitation-coupled calcium entry but not for formation of tetrads in skeletal myotubes. Biophysical Journal. 94: 3023-34. PMID 18192372 DOI: 10.1529/Biophysj.107.118893 |
0.652 |
|
2007 |
Lorenzon NM, Beam KG. Accessibility of targeted DHPR sites to streptavidin and functional effects of binding on EC coupling. The Journal of General Physiology. 130: 379-88. PMID 17893191 DOI: 10.1083/Jcb1791Oia3 |
0.504 |
|
2006 |
Sheridan DC, Takekura H, Franzini-Armstrong C, Beam KG, Allen PD, Perez CF. Bidirectional signaling between calcium channels of skeletal muscle requires multiple direct and indirect interactions. Proceedings of the National Academy of Sciences of the United States of America. 103: 19760-5. PMID 17172444 DOI: 10.1073/Pnas.0609473103 |
0.772 |
|
2006 |
Leuranguer V, Papadopoulos S, Beam KG. Organization of calcium channel beta1a subunits in triad junctions in skeletal muscle. The Journal of Biological Chemistry. 281: 3521-7. PMID 16317008 DOI: 10.1074/Jbc.M509566200 |
0.441 |
|
2005 |
Bannister RA, Beam KG. The alpha1S N-terminus is not essential for bi-directional coupling with RyR1. Biochemical and Biophysical Research Communications. 336: 134-41. PMID 16139246 DOI: 10.1016/J.Bbrc.2005.08.047 |
0.731 |
|
2005 |
Hurne AM, O'Brien JJ, Wingrove D, Cherednichenko G, Allen PD, Beam KG, Pessah IN. Ryanodine receptor type 1 (RyR1) mutations C4958S and C4961S reveal excitation-coupled calcium entry (ECCE) is independent of sarcoplasmic reticulum store depletion. The Journal of Biological Chemistry. 280: 36994-7004. PMID 16120606 DOI: 10.1074/Jbc.M506441200 |
0.713 |
|
2005 |
Cherednichenko G, Hurne AM, Fessenden JD, Eun HL, Allen PD, Beam KG, Pessah IN. Erratum: Conformational activation of Ca2+ entry by depolarization of skeletal myotubes (Proceedings of the National Academy of Sciences of the United States of America (November 2, 2004) 101 (15793-15798)) Proceedings of the National Academy of Sciences of the United States of America. 102. DOI: 10.1073/Pnas.0500805102 |
0.403 |
|
2004 |
Cherednichenko G, Hurne AM, Fessenden JD, Lee EH, Allen PD, Beam KG, Pessah IN. Conformational activation of Ca2+ entry by depolarization of skeletal myotubes. Proceedings of the National Academy of Sciences of the United States of America. 101: 15793-8. PMID 15505226 DOI: 10.1073/Pnas.0403485101 |
0.541 |
|
2004 |
Papadopoulos S, Leuranguer V, Bannister RA, Beam KG. Mapping sites of potential proximity between the dihydropyridine receptor and RyR1 in muscle using a cyan fluorescent protein-yellow fluorescent protein tandem as a fluorescence resonance energy transfer probe. The Journal of Biological Chemistry. 279: 44046-56. PMID 15280389 DOI: 10.1074/Jbc.M405317200 |
0.725 |
|
2004 |
Lorenzon NM, Haarmann CS, Norris EE, Papadopoulos S, Beam KG. Metabolic biotinylation as a probe of supramolecular structure of the triad junction in skeletal muscle. The Journal of Biological Chemistry. 279: 44057-64. PMID 15280388 DOI: 10.1074/Jbc.M405318200 |
0.443 |
|
2003 |
Wilkens CM, Beam KG. Insertion of alpha1S II-III loop and C terminal sequences into alpha1H fails to restore excitation-contraction coupling in dysgenic myotubes. Journal of Muscle Research and Cell Motility. 24: 99-109. PMID 12953840 DOI: 10.1023/A:1024830132118 |
0.821 |
|
2003 |
Leuranguer V, Dirksen RT, Beam KG. Potentiated L-type Ca2+ channels rectify. The Journal of General Physiology. 121: 541-50. PMID 12743165 DOI: 10.1085/Jgp.200308833 |
0.669 |
|
2002 |
Protasi F, Paolini C, Nakai J, Beam KG, Franzini-Armstrong C, Allen PD. Multiple regions of RyR1 mediate functional and structural interactions with alpha(1S)-dihydropyridine receptors in skeletal muscle. Biophysical Journal. 83: 3230-44. PMID 12496092 DOI: 10.1016/S0006-3495(02)75325-3 |
0.64 |
|
2002 |
O'Brien JJ, Feng W, Allen PD, Chen SR, Pessah IN, Beam KG. Ca2+ activation of RyR1 is not necessary for the initiation of skeletal-type excitation-contraction coupling. Biophysical Journal. 82: 2428-35. PMID 11964231 DOI: 10.1016/S0006-3495(02)75586-0 |
0.689 |
|
2002 |
Proenza C, O'Brien J, Nakai J, Mukherjee S, Allen PD, Beam KG. Identification of a region of RyR1 that participates in allosteric coupling with the alpha(1S) (Ca(V)1.1) II-III loop. The Journal of Biological Chemistry. 277: 6530-5. PMID 11726651 DOI: 10.1074/Jbc.M106471200 |
0.787 |
|
2001 |
Wilkens CM, Grabner M, Beam KG. Potentiation of the cardiac L-type Ca(2+) channel (alpha(1C)) by dihydropyridine agonist and strong depolarization occur via distinct mechanisms. The Journal of General Physiology. 118: 495-508. PMID 11696608 DOI: 10.1085/Jgp.118.5.495 |
0.817 |
|
2001 |
Stroffekova K, Proenza C, Beam KG. The protein-labeling reagent FLASH-EDT2 binds not only to CCXXCC motifs but also non-specifically to endogenous cysteine-rich proteins. Pflã¼Gers Archiv : European Journal of Physiology. 442: 859-66. PMID 11680618 DOI: 10.1007/S004240100619 |
0.712 |
|
2001 |
Lorenzon NM, Grabner M, Suda N, Beam KG. Structure and targeting of RyR1: implications from fusion of green fluorescent protein at the amino-terminal. Archives of Biochemistry and Biophysics. 388: 13-7. PMID 11361129 DOI: 10.1006/Abbi.2000.2263 |
0.627 |
|
2001 |
Wilkens CM, Kasielke N, Flucher BE, Beam KG, Grabner M. Excitation-contraction coupling is unaffected by drastic alteration of the sequence surrounding residues L720-L764 of the alpha 1S II-III loop. Proceedings of the National Academy of Sciences of the United States of America. 98: 5892-7. PMID 11320225 DOI: 10.1073/Pnas.101618098 |
0.815 |
|
2000 |
Proenza C, Wilkens CM, Beam KG. Excitation-contraction coupling is not affected by scrambled sequence in residues 681-690 of the dihydropyridine receptor II-III loop. The Journal of Biological Chemistry. 275: 29935-7. PMID 10915779 DOI: 10.1074/Jbc.C000464200 |
0.809 |
|
2000 |
Proenza C, Wilkens C, Lorenzon NM, Beam KG. A carboxyl-terminal region important for the expression and targeting of the skeletal muscle dihydropyridine receptor. The Journal of Biological Chemistry. 275: 23169-74. PMID 10801875 DOI: 10.1074/Jbc.M003389200 |
0.798 |
|
2000 |
Suda N, Dirksen RT, Gonzalez A, Beam KG. Low serum promotes maturation of excitation-contraction coupling in myotubes. Pflã¼Gers Archiv : European Journal of Physiology. 439: 555-8. PMID 10764214 DOI: 10.1007/S004249900210 |
0.674 |
|
2000 |
Lorenzon NM, Beam KG. Calcium channelopathies. Kidney International. 57: 794-802. PMID 10720931 DOI: 10.1046/J.1523-1755.2000.00917.X |
0.39 |
|
1999 |
Pan F, Beam KG. The absence of resurgent sodium current in mouse spinal neurons. Brain Research. 849: 162-8. PMID 10592298 DOI: 10.1016/S0006-8993(99)02060-0 |
0.348 |
|
1999 |
Dirksen RT, Beam KG. Role of calcium permeation in dihydropyridine receptor function. Insights into channel gating and excitation-contraction coupling. The Journal of General Physiology. 114: 393-403. PMID 10469729 DOI: 10.1085/Jgp.114.3.393 |
0.724 |
|
1999 |
Grabner M, Dirksen RT, Suda N, Beam KG. The II-III loop of the skeletal muscle dihydropyridine receptor is responsible for the Bi-directional coupling with the ryanodine receptor. The Journal of Biological Chemistry. 274: 21913-9. PMID 10419512 DOI: 10.1074/Jbc.274.31.21913 |
0.799 |
|
1999 |
Beam K. Calcium channel splicing: Mind your Ps and Qs Nature Neuroscience. 2: 393-394. PMID 10321237 DOI: 10.1038/8052 |
0.377 |
|
1998 |
Nakai J, Tanabe T, Konno T, Adams B, Beam KG. Localization in the II-III loop of the dihydropyridine receptor of a sequence critical for excitation-contraction coupling. The Journal of Biological Chemistry. 273: 24983-6. PMID 9737952 DOI: 10.1074/Jbc.273.39.24983 |
0.499 |
|
1998 |
GarcÃa KD, Sprunger LK, Meisler MH, Beam KG. The sodium channel Scn8a is the major contributor to the postnatal developmental increase of sodium current density in spinal motoneurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 18: 5234-9. PMID 9651206 DOI: 10.1523/Jneurosci.18-14-05234.1998 |
0.323 |
|
1998 |
Lorenzon NM, Lutz CM, Frankel WN, Beam KG. Altered calcium channel currents in Purkinje cells of the neurological mutant mouse leaner. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 18: 4482-9. PMID 9614225 DOI: 10.1523/Jneurosci.18-12-04482.1998 |
0.397 |
|
1998 |
Nakai J, Sekiguchi N, Rando TA, Allen PD, Beam KG. Two regions of the ryanodine receptor involved in coupling with L-type Ca2+ channels. The Journal of Biological Chemistry. 273: 13403-6. PMID 9593671 DOI: 10.1074/Jbc.273.22.13403 |
0.584 |
|
1998 |
Grabner M, Dirksen RT, Beam KG. Tagging with green fluorescent protein reveals a distinct subcellular distribution of L-type and non-L-type Ca2+ channels expressed in dysgenic myotubes. Proceedings of the National Academy of Sciences of the United States of America. 95: 1903-8. PMID 9465115 DOI: 10.1073/Pnas.95.4.1903 |
0.787 |
|
1997 |
Beam KG, Franzini-Armstrong C. Functional and structural approaches to the study of excitation-contraction coupling. Methods in Cell Biology. 52: 283-306. PMID 9379955 DOI: 10.1016/S0091-679X(08)60384-2 |
0.618 |
|
1997 |
Dirksen RT, Nakai J, Gonzalez A, Imoto K, Beam KG. The S5-S6 linker of repeat I is a critical determinant of L-type Ca2+ channel conductance. Biophysical Journal. 73: 1402-9. PMID 9284307 DOI: 10.1016/S0006-3495(97)78172-4 |
0.655 |
|
1997 |
GarcÃa J, Nakai J, Imoto K, Beam KG. Role of S4 segments and the leucine heptad motif in the activation of an L-type calcium channel. Biophysical Journal. 72: 2515-23. PMID 9168028 DOI: 10.1016/S0006-3495(97)78896-9 |
0.43 |
|
1997 |
Nakai J, Ogura T, Protasi F, Franzini-Armstrong C, Allen PD, Beam KG. Functional nonequality of the cardiac and skeletal ryanodine receptors. Proceedings of the National Academy of Sciences of the United States of America. 94: 1019-22. PMID 9023375 DOI: 10.1073/Pnas.94.3.1019 |
0.728 |
|
1996 |
Adams BA, Tanabe T, Beam KG. Ca2+ current activation rate correlates with alpha 1 subunit density. Biophysical Journal. 71: 156-62. PMID 8804599 DOI: 10.1016/S0006-3495(96)79212-3 |
0.502 |
|
1996 |
GarcÃa KD, Mynlieff M, Sanders DB, Beam KG, Walrond JP. Lambert-Eaton sera reduce low-voltage and high-voltage activated Ca2+ currents in murine dorsal root ganglion neurons. Proceedings of the National Academy of Sciences of the United States of America. 93: 9264-9. PMID 8799189 DOI: 10.1073/Pnas.93.17.9264 |
0.713 |
|
1996 |
Dirksen RT, Beam KG. Unitary behavior of skeletal, cardiac, and chimeric L-type Ca2+ channels expressed in dysgenic myotubes. The Journal of General Physiology. 107: 731-42. PMID 8783073 DOI: 10.1085/Jgp.107.6.731 |
0.657 |
|
1996 |
GarcÃa KD, Beam KG. Reduction of calcium currents by Lambert-Eaton syndrome sera: motoneurons are preferentially affected, and L-type currents are spared. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 16: 4903-13. PMID 8756422 DOI: 10.1523/Jneurosci.16-16-04903.1996 |
0.421 |
|
1996 |
Gallant EM, Balog EM, Beam KG. Slow calcium current is not reduced in malignant hyperthermic porcine myotubes. Muscle & Nerve. 19: 450-5. PMID 8622723 DOI: 10.1002/(Sici)1097-4598(199604)19:4<450::Aid-Mus4>3.0.Co;2-B |
0.547 |
|
1996 |
Nakai J, Dirksen RT, Nguyen HT, Pessah IN, Beam KG, Allen PD. Enhanced dihydropyridine receptor channel activity in the presence of ryanodine receptor. Nature. 380: 72-5. PMID 8598910 DOI: 10.1038/380072A0 |
0.737 |
|
1995 |
Dirksen RT, Beam KG. Single calcium channel behavior in native skeletal muscle. The Journal of General Physiology. 105: 227-47. PMID 7539048 DOI: 10.1085/Jgp.105.2.227 |
0.653 |
|
1994 |
Nakai J, Adams BA, Imoto K, Beam KG. Critical roles of the S3 segment and S3-S4 linker of repeat I in activation of L-type calcium channels. Proceedings of the National Academy of Sciences of the United States of America. 91: 1014-8. PMID 8302825 DOI: 10.1073/Pnas.91.3.1014 |
0.433 |
|
1994 |
Mynlieff M, Beam KG. Adenosine acting at an A1 receptor decreases N-type calcium current in mouse motoneurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 14: 3628-34. PMID 8207477 DOI: 10.1523/Jneurosci.14-06-03628.1994 |
0.666 |
|
1994 |
GarcÃa J, Tanabe T, Beam KG. Relationship of calcium transients to calcium currents and charge movements in myotubes expressing skeletal and cardiac dihydropyridine receptors. The Journal of General Physiology. 103: 125-47. PMID 8169595 DOI: 10.1085/Jgp.103.1.125 |
0.45 |
|
1994 |
GarcÃa J, Beam KG. Measurement of calcium transients and slow calcium current in myotubes. The Journal of General Physiology. 103: 107-23. PMID 8169594 DOI: 10.1085/Jgp.103.1.107 |
0.413 |
|
1994 |
Takekura H, Bennett L, Tanabe T, Beam KG, Franzini-Armstrong C. Restoration of junctional tetrads in dysgenic myotubes by dihydropyridine receptor cDNA. Biophysical Journal. 67: 793-803. PMID 7948692 DOI: 10.1016/S0006-3495(94)80539-9 |
0.587 |
|
1994 |
Adams BA, Mori Y, Kim MS, Tanabe T, Beam KG. Heterologous expression of BI Ca2+ channels in dysgenic skeletal muscle. The Journal of General Physiology. 104: 985-96. PMID 7876830 DOI: 10.1085/Jgp.104.5.985 |
0.569 |
|
1994 |
GarcÃa J, Beam KG. Calcium transients associated with the T type calcium current in myotubes. The Journal of General Physiology. 104: 1113-28. PMID 7699366 DOI: 10.1085/Jgp.104.6.1113 |
0.399 |
|
1993 |
Mori Y, Niidome T, Fujita Y, Mynlieff M, Dirksen RT, Beam KG, Iwabe N, Miyata T, Furutama D, Furuichi T. Molecular diversity of voltage-dependent calcium channel. Annals of the New York Academy of Sciences. 707: 87-108. PMID 9137545 DOI: 10.1111/J.1749-6632.1993.Tb38045.X |
0.733 |
|
1993 |
Tanabe T, Mikami A, Niidome T, Numa S, Adams BA, Beam KG. Structure and function of voltage-dependent calcium channels from muscle. Annals of the New York Academy of Sciences. 707: 81-6. PMID 9137544 DOI: 10.1111/J.1749-6632.1993.Tb38044.X |
0.46 |
|
1993 |
Chaudhari N, Beam KG. mRNA for cardiac calcium channel is expressed during development of skeletal muscle. Developmental Biology. 155: 507-15. PMID 8381753 DOI: 10.1006/Dbio.1993.1048 |
0.638 |
|
1992 |
Beam KG, Adams BA, Niidome T, Numa S, Tanabe T. Function of a truncated dihydropyridine receptor as both voltage sensor and calcium channel. Nature. 360: 169-71. PMID 1331811 DOI: 10.1038/360169A0 |
0.494 |
|
1992 |
Mynlieff M, Beam KG. Characterization of voltage-dependent calcium currents in mouse motoneurons. Journal of Neurophysiology. 68: 85-92. PMID 1325548 DOI: 10.1152/Jn.1992.68.1.85 |
0.676 |
|
1992 |
Mynlieff M, Beam KG. Developmental expression of voltage-dependent calcium currents in identified mouse motoneurons. Developmental Biology. 152: 407-10. PMID 1322848 DOI: 10.1016/0012-1606(92)90148-A |
0.63 |
|
1991 |
Adams BA, Beam KG. Contractions of dysgenic skeletal muscle triggered by a potentiated, endogenous calcium current. The Journal of General Physiology. 97: 687-96. PMID 1711572 DOI: 10.1085/Jgp.97.4.687 |
0.488 |
|
1991 |
Tanabe T, Adams BA, Numa S, Beam KG. Repeat I of the dihydropyridine receptor is critical in determining calcium channel activation kinetics. Nature. 352: 800-3. PMID 1652692 DOI: 10.1038/352800A0 |
0.504 |
|
1991 |
McCobb DP, Beam KG. Action potential waveform voltage-clamp commands reveal striking differences in calcium entry via low and high voltage-activated calcium channels. Neuron. 7: 119-27. PMID 1648936 DOI: 10.1016/0896-6273(91)90080-J |
0.42 |
|
1990 |
Chaudhari N, Beam KG. Fibroblasts fuse with myotubes developing in culture. Advances in Experimental Medicine and Biology. 280: 131-6; discussion 13. PMID 2248133 DOI: 10.1007/978-1-4684-5865-7_15 |
0.607 |
|
1990 |
McCobb DP, Best PM, Beam KG. The differentiation of excitability in embryonic chick limb motoneurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 10: 2974-84. PMID 2168941 DOI: 10.1523/Jneurosci.10-09-02974.1990 |
0.414 |
|
1990 |
Adams BA, Tanabe T, Mikami A, Numa S, Beam KG. Intramembrane charge movement restored in dysgenic skeletal muscle by injection of dihydropyridine receptor cDNAs. Nature. 346: 569-72. PMID 2165571 DOI: 10.1038/346569A0 |
0.438 |
|
1990 |
Tanabe T, Beam KG, Adams BA, Niidome T, Numa S. Regions of the skeletal muscle dihydropyridine receptor critical for excitation-contraction coupling. Nature. 346: 567-9. PMID 2165570 DOI: 10.1038/346567A0 |
0.543 |
|
1990 |
Adams BA, Beam KG. Muscular dysgenesis in mice: a model system for studying excitation-contraction coupling. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 4: 2809-16. PMID 2165014 DOI: 10.1096/Fasebj.4.10.2165014 |
0.508 |
|
1990 |
Tanabe T, Mikami A, Numa S, Beam KG. Cardiac-type excitation-contraction coupling in dysgenic skeletal muscle injected with cardiac dihydropyridine receptor cDNA. Nature. 344: 451-3. PMID 2157159 DOI: 10.1038/344451A0 |
0.499 |
|
1990 |
Numa S, Tanabe T, Takeshima H, Mikami A, Niidome T, Nishimura S, Adams BA, Beam KG. Molecular insights into excitation-contraction coupling. Cold Spring Harbor Symposia On Quantitative Biology. 55: 1-7. PMID 1966760 DOI: 10.1101/Sqb.1990.055.01.003 |
0.307 |
|
1989 |
Chaudhari N, Beam KG. The muscular dysgenesis mutation in mice leads to arrest of the genetic program for muscle differentiation. Developmental Biology. 133: 456-67. PMID 2731637 DOI: 10.1016/0012-1606(89)90049-3 |
0.616 |
|
1989 |
McCobb DP, Best PM, Beam KG. Development alters the expression of calcium currents in chick limb motoneurons. Neuron. 2: 1633-43. PMID 2560650 DOI: 10.1016/0896-6273(89)90052-4 |
0.379 |
|
1989 |
Courbin P, Koenig J, Ressouches A, Beam KG, Powell JA. Rescue of excitation-contraction coupling in dysgenic muscle by addition of fibroblasts in vitro. Neuron. 2: 1341-50. PMID 2560638 DOI: 10.1016/0896-6273(89)90072-X |
0.487 |
|
1989 |
Adams BA, Beam KG. A novel calcium current in dysgenic skeletal muscle. The Journal of General Physiology. 94: 429-44. PMID 2558151 DOI: 10.1085/Jgp.94.3.429 |
0.514 |
|
1989 |
Beam KG, Tanabe T, Numa S. Structure, function, and regulation of the skeletal muscle dihydropyridine receptor. Annals of the New York Academy of Sciences. 560: 127-37. PMID 2545129 DOI: 10.1111/J.1749-6632.1989.Tb24090.X |
0.374 |
|
1989 |
Knudson CM, Chaudhari N, Sharp AH, Powell JA, Beam KG, Campbell KP. Specific absence of the alpha 1 subunit of the dihydropyridine receptor in mice with muscular dysgenesis. The Journal of Biological Chemistry. 264: 1345-8. PMID 2536362 |
0.646 |
|
1989 |
Chaudhari N, Delay R, Beam KG. Restoration of normal function in genetically defective myotubes by spontaneous fusion with fibroblasts. Nature. 341: 445-7. PMID 2507925 DOI: 10.1038/341445A0 |
0.629 |
|
1989 |
Kinnamon SC, Cummings TA, Roper SD, Beam KG. Calcium currents in isolated taste receptor cells of the mudpuppy Annals of the New York Academy of Sciences. 560: 112-115. DOI: 10.1111/J.1749-6632.1989.Tb24085.X |
0.33 |
|
1988 |
Tanabe T, Beam KG, Powell JA, Numa S. Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA. Nature. 336: 134-9. PMID 2903448 DOI: 10.1038/336134A0 |
0.491 |
|
1988 |
Beam KG, Knudson CM. Effect of postnatal development on calcium currents and slow charge movement in mammalian skeletal muscle. The Journal of General Physiology. 91: 799-815. PMID 2458430 DOI: 10.1085/Jgp.91.6.799 |
0.324 |
|
1988 |
Beam KG, Knudson CM. Calcium currents in embryonic and neonatal mammalian skeletal muscle. The Journal of General Physiology. 91: 781-98. PMID 2458429 DOI: 10.1085/Jgp.91.6.781 |
0.413 |
|
1988 |
Kinnamon SC, Dionne VE, Beam KG. Apical localization of K+ channels in taste cells provides the basis for sour taste transduction. Proceedings of the National Academy of Sciences of the United States of America. 85: 7023-7. PMID 2457924 DOI: 10.1073/Pnas.85.18.7023 |
0.684 |
|
1986 |
Caldwell JH, Campbell DT, Beam KG. Na channel distribution in vertebrate skeletal muscle. The Journal of General Physiology. 87: 907-32. PMID 2425042 DOI: 10.1085/Jgp.87.6.907 |
0.369 |
|
1986 |
Beam KG, Knudson CM, Powell JA. A lethal mutation in mice eliminates the slow calcium current in skeletal muscle cells. Nature. 320: 168-70. PMID 2419767 DOI: 10.1038/320168a0 |
0.348 |
|
1985 |
Beam KG, Caldwell JH, Campbell DT. Na channels in skeletal muscle concentrated near the neuromuscular junction. Nature. 313: 588-90. PMID 2578630 DOI: 10.1038/313588A0 |
0.449 |
|
1983 |
Simon BJ, Beam KG. Charge movement in a fast twitch skeletal muscle from rat. Biophysical Journal. 41: 223-6. PMID 6838965 DOI: 10.1016/S0006-3495(83)84423-3 |
0.314 |
|
1983 |
Donaldson PL, Beam KG. Calcium currents in a fast-twitch skeletal muscle of the rat. The Journal of General Physiology. 82: 449-68. PMID 6315856 DOI: 10.1085/Jgp.82.4.449 |
0.406 |
|
1983 |
Beam KG, Donaldson PL. Slow components of potassium tail currents in rat skeletal muscle. The Journal of General Physiology. 81: 513-30. PMID 6304232 DOI: 10.1085/Jgp.81.4.513 |
0.391 |
|
1983 |
Beam KG, Donaldson PL. A quantitative study of potassium channel kinetics in rat skeletal muscle from 1 to 37 degrees C. The Journal of General Physiology. 81: 485-512. PMID 6304231 DOI: 10.1085/Jgp.81.4.485 |
0.345 |
|
1979 |
Beam KG, Alper SL, Palade GE, Greengard P. Hormonally regulated phosphoprotein of turkey erythrocytes: localization to plasma membrane. The Journal of Cell Biology. 83: 1-15. PMID 229109 DOI: 10.1083/jcb.83.1.1 |
0.311 |
|
1978 |
Nestler EJ, Beam KG, Greengard P. Nicotinic cholinergic stimulation increases cyclic GMP levels in vertebrate skeletal muscle. Nature. 275: 451-3. PMID 211436 DOI: 10.1038/275451A0 |
0.606 |
|
1977 |
Beam KG, Nestler EJ, Greengard P. Increased cyclic GMP levels associated with contraction in muscle fibres of the giant barnacle. Nature. 267: 534-6. PMID 195221 DOI: 10.1038/267534A0 |
0.65 |
|
1976 |
Beam KG. A voltage-clamp study of the effect of two lidocaine derivatives on the time course of end-plate currents. The Journal of Physiology. 258: 279-300. PMID 1085354 DOI: 10.1113/Jphysiol.1976.Sp011420 |
0.323 |
|
1976 |
Beam KG, Greengard P. Cyclic nucleotides, protein phosphorylation and synaptic function. Cold Spring Harbor Symposia On Quantitative Biology. 40: 157-68. PMID 7379 DOI: 10.1101/Sqb.1976.040.01.017 |
0.39 |
|
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