| Year |
Citation |
Score |
| 2024 |
Regan CP, Morissette P, Kraus RL, Wang E, Arrington L, Vavrek M, de Hoon J, Depre M, Lodeweyck T, Demeyer I, Laethem T, Stoch A, Struyk A. Autonomic Dysfunction Linked to Inhibition of the Na1.7 Sodium Channel. Circulation. 149: 1394-1396. PMID 38648272 DOI: 10.1161/CIRCULATIONAHA.123.067331 |
0.324 |
|
| 2016 |
Wu F, Mi W, Fu Y, Struyk A, Cannon SC. Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis. Brain : a Journal of Neurology. PMID 27048647 DOI: 10.1093/Brain/Aww070 |
0.74 |
|
| 2012 |
Wu F, Mi W, Hernández-Ochoa EO, Burns DK, Fu Y, Gray HF, Struyk AF, Schneider MF, Cannon SC. A calcium channel mutant mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 122: 4580-91. PMID 23187123 DOI: 10.1172/Jci66091 |
0.75 |
|
| 2011 |
Wu F, Mi W, Burns DK, Fu Y, Gray HF, Struyk AF, Cannon SC. A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 121: 4082-94. PMID 21881211 DOI: 10.1172/Jci57398 |
0.76 |
|
| 2011 |
Kudryashova E, Struyk A, Mokhonova E, Cannon SC, Spencer MJ. The common missense mutation D489N in TRIM32 causing limb girdle muscular dystrophy 2H leads to loss of the mutated protein in knock-in mice resulting in a Trim32-null phenotype. Human Molecular Genetics. 20: 3925-32. PMID 21775502 DOI: 10.1093/Hmg/Ddr311 |
0.634 |
|
| 2011 |
Francis DG, Rybalchenko V, Struyk A, Cannon SC. Leaky sodium channels from voltage sensor mutations in periodic paralysis, but not paramyotonia. Neurology. 76: 1635-41. PMID 21490317 DOI: 10.1212/Wnl.0B013E318219Fb57 |
0.713 |
|
| 2011 |
Fu Y, Struyk A, Markin V, Cannon S. Gating behaviour of sodium currents in adult mouse muscle recorded with an improved two-electrode voltage clamp. The Journal of Physiology. 589: 525-46. PMID 21135045 DOI: 10.1113/Jphysiol.2010.199430 |
0.728 |
|
| 2010 |
Francis D, Struyk A, Cannon SC. Arginine Mutations in the S4 VSD of NaV1.4 Associated with Hypokalemic Periodic Paralysis, But Not with Paramyotonia, Create a Gating Pore Conductance Biophysical Journal. 98: 111a. DOI: 10.1016/J.Bpj.2009.12.4199 |
0.716 |
|
| 2009 |
Struyk A, Cannon SC. Gating Pore Currents from S4 Mutations of NaV1.4: A Common Pathomechanism in Hypokalemic Periodic Paralysis Biophysical Journal. 96: 203a. DOI: 10.1016/J.Bpj.2008.12.1095 |
0.709 |
|
| 2008 |
Struyk AF, Markin VS, Francis D, Cannon SC. Gating pore currents in DIIS4 mutations of NaV1.4 associated with periodic paralysis: saturation of ion flux and implications for disease pathogenesis. The Journal of General Physiology. 132: 447-64. PMID 18824591 DOI: 10.1085/Jgp.200809967 |
0.695 |
|
| 2008 |
Struyk AF, Cannon SC. Paradoxical depolarization of BA2+- treated muscle exposed to low extracellular K+: insights into resting potential abnormalities in hypokalemic paralysis. Muscle & Nerve. 37: 326-37. PMID 18041053 DOI: 10.1002/Mus.20928 |
0.631 |
|
| 2007 |
Struyk AF, Cannon SC. A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore. The Journal of General Physiology. 130: 11-20. PMID 17591984 DOI: 10.1085/Jgp.200709755 |
0.723 |
|
| 2002 |
Struyk AF, Cannon SC. Slow inactivation does not block the aqueous accessibility to the outer pore of voltage-gated Na channels. The Journal of General Physiology. 120: 509-16. PMID 12356853 DOI: 10.1085/Jgp.20028672 |
0.607 |
|
| 2000 |
Struyk AF, Scoggan KA, Bulman DE, Cannon SC. The human skeletal muscle Na channel mutation R669H associated with hypokalemic periodic paralysis enhances slow inactivation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 20: 8610-7. PMID 11102465 DOI: 10.1523/Jneurosci.20-23-08610.2000 |
0.714 |
|
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