Year |
Citation |
Score |
2009 |
Roessler E, Pei W, Ouspenskaia MV, Karkera JD, Veléz JI, Banerjee-Basu S, Gibney G, Lupo PJ, Mitchell LE, Towbin JA, Bowers P, Belmont JW, Goldmuntz E, Baxevanis AD, Feldman B, et al. Cumulative ligand activity of NODAL mutations and modifiers are linked to human heart defects and holoprosencephaly. Molecular Genetics and Metabolism. 98: 225-34. PMID 19553149 DOI: 10.1016/J.Ymgme.2009.05.005 |
0.32 |
|
2008 |
Roessler E, Ouspenskaia MV, Karkera JD, Vélez JI, Kantipong A, Lacbawan F, Bowers P, Belmont JW, Towbin JA, Goldmuntz E, Feldman B, Muenke M. Reduced NODAL signaling strength via mutation of several pathway members including FOXH1 is linked to human heart defects and holoprosencephaly. American Journal of Human Genetics. 83: 18-29. PMID 18538293 DOI: 10.1016/J.Ajhg.2008.05.012 |
0.331 |
|
2003 |
Grossman HA, Goon B, Bowers P, Leitz G. Once-weekly epoetin alfa dosing is as effective as three times-weekly dosing in increasing hemoglobin levels and is associated with improved quality of life in anemic HIV-infected patients. Journal of Acquired Immune Deficiency Syndromes (1999). 34: 368-78. PMID 14615654 DOI: 10.1097/00126334-200312010-00003 |
0.301 |
|
2000 |
Bamford RN, Roessler E, Burdine RD, SaplakoÄŸlu U, dela Cruz J, Splitt M, Goodship JA, Towbin J, Bowers P, Ferrero GB, Marino B, Schier AF, Shen MM, Muenke M, Casey B. Loss-of-function mutations in the EGF-CFC gene CFC1 are associated with human left-right laterality defects. Nature Genetics. 26: 365-9. PMID 11062482 DOI: 10.1038/81695 |
0.302 |
|
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