Jane E. Hartung, BS - Publications
Affiliations: | Biology | Furman University, Greenville, SC, United States |
Area:
Neurodegeneration, molecular neurobiologyYear | Citation | Score | |||
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2018 | Zhang X, Hartung JE, Bortsov AV, Kim S, O'Buckley SC, Kozlowski J, Nackley AG. Sustained Stimulation of β- and β-adrenergic Receptors Leads to Persistent Functional Pain and Neuroinflammation. Brain, Behavior, and Immunity. PMID 29935309 DOI: 10.1016/J.Bbi.2018.06.017 | 0.733 | |||
2016 | Harmon JB, Sanders AE, Wilder RS, Essick GK, Slade GD, Hartung JE, Nackley AG. Circulating Omentin-1 and Chronic Painful Temporomandibular Disorders. Journal of Oral & Facial Pain and Headache. 30: 203-209. PMID 27472522 DOI: 10.11607/Ofph.1608 | 0.715 | |||
2016 | Hartung J, Ciszek B, O'Buckley S, Nackley A. (374) Distinct mechanisms underlie the development and maintenance of COMT-dependent pain The Journal of Pain. 17: S68. DOI: 10.1016/J.Jpain.2016.01.281 | 0.713 | |||
2015 | Hartung JE, Eskew O, Wong T, Tchivileva IE, Oladosu FA, O'Buckley SC, Nackley AG. Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation. Brain, Behavior, and Immunity. PMID 26187567 DOI: 10.1016/J.Bbi.2015.07.014 | 0.599 | |||
2014 | Hartung JE, Ciszek BP, Nackley AG. β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines. Pain. 155: 1346-55. PMID 24727346 DOI: 10.1016/J.Pain.2014.04.011 | 0.712 | |||
2014 | Hartung J, Nackley A. 127. B2-and B3-adrenergic receptors drive the development of COMT-dependent pain by increasing release of NO and innate immune cytokines Brain, Behavior, and Immunity. 40: e37. DOI: 10.1016/J.Bbi.2014.06.147 | 0.757 | |||
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