LaShauna C. Evans, Ph.D. - Publications

Affiliations: 
2012 Physiology University of Maryland School of Medicine, Baltimore, MD, United States 
Area:
Physiology Biology, Molecular Biology, Biochemistry

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2013 Al-Hasan YM, Evans LC, Pinkas GA, Dabkowski ER, Stanley WC, Thompson LP. Chronic hypoxia impairs cytochrome oxidase activity via oxidative stress in selected fetal Guinea pig organs. Reproductive Sciences (Thousand Oaks, Calif.). 20: 299-307. PMID 22923417 DOI: 10.1177/1933719112453509  0.63
2012 Hashimoto K, Pinkas G, Evans L, Liu H, Al-Hasan Y, Thompson LP. Protective effect of N-acetylcysteine on liver damage during chronic intrauterine hypoxia in fetal guinea pig. Reproductive Sciences (Thousand Oaks, Calif.). 19: 1001-9. PMID 22534333 DOI: 10.1177/1933719112440052  0.585
2012 Evans LC, Liu H, Thompson LP. Differential effect of intrauterine hypoxia on caspase 3 and DNA fragmentation in fetal guinea pig hearts and brains. Reproductive Sciences (Thousand Oaks, Calif.). 19: 298-305. PMID 22383778 DOI: 10.1177/1933719111420883  0.608
2012 Evans LC, Liu H, Pinkas GA, Thompson LP. Chronic hypoxia increases peroxynitrite, MMP9 expression, and collagen accumulation in fetal guinea pig hearts. Pediatric Research. 71: 25-31. PMID 22289847 DOI: 10.1038/Pr.2011.10  0.631
2011 Thompson LP, Liu H, Evans L, Mong JA. Prenatal nicotine increases matrix metalloproteinase 2 (MMP-2) expression in fetal guinea pig hearts. Reproductive Sciences (Thousand Oaks, Calif.). 18: 1103-10. PMID 21775771 DOI: 10.1177/1933719111404605  0.586
2009 Thompson L, Dong Y, Evans L. Chronic hypoxia increases inducible NOS-derived nitric oxide in fetal guinea pig hearts. Pediatric Research. 65: 188-92. PMID 19047955 DOI: 10.1203/Pdr.0B013E31818D6Ad0  0.601
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