John David Johnson - Publications

University of Colorado, Boulder, Boulder, CO, United States 
cytokines, brain, behavior

26 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2016 Lowrance SA, Ionadi A, McKay E, Douglas X, Johnson JD. Sympathetic nervous system contributes to enhanced corticosterone levels following chronic stress. Psychoneuroendocrinology. 68: 163-170. PMID 26974501 DOI: 10.1016/j.psyneuen.2016.02.027  0.8
2015 Remus JL, Stewart LT, Camp RM, Novak CM, Johnson JD. Interaction of metabolic stress with chronic mild stress in altering brain cytokines and sucrose preference. Behavioral Neuroscience. 129: 321-30. PMID 25914924 DOI: 10.1037/bne0000056  0.8
2015 Camp RM, Johnson JD. Repeated stressor exposure enhances contextual fear memory in a beta-adrenergic receptor-dependent process and increases impulsivity in a non-beta receptor-dependent fashion. Physiology & Behavior. PMID 25747320 DOI: 10.1016/j.physbeh.2015.03.008  0.8
2013 Johnson JD, Zimomra ZR, Stewart LT. Beta-adrenergic receptor activation primes microglia cytokine production. Journal of Neuroimmunology. 254: 161-4. PMID 22944319 DOI: 10.1016/j.jneuroim.2012.08.007  0.8
2012 Porterfield VM, Gabella KM, Simmons MA, Johnson JD. Repeated stressor exposure regionally enhances beta-adrenergic receptor-mediated brain IL-1β production. Brain, Behavior, and Immunity. 26: 1249-55. PMID 22902349 DOI: 10.1016/j.bbi.2012.08.001  0.8
2012 Camp RM, Remus JL, Kalburgi SN, Porterfield VM, Johnson JD. Fear conditioning can contribute to behavioral changes observed in a repeated stress model. Behavioural Brain Research. 233: 536-44. PMID 22664265 DOI: 10.1016/j.bbr.2012.05.040  0.8
2012 Campisi J, Sharkey C, Johnson JD, Asea A, Maslanik T, Bernstein-Hanley I, Fleshner M. Stress-induced facilitation of host response to bacterial challenge in F344 rats is dependent on extracellular heat shock protein 72 and independent of alpha beta T cells. Stress (Amsterdam, Netherlands). 15: 637-46. PMID 22217161 DOI: 10.3109/10253890.2011.653596  0.8
2011 Zimomra ZR, Porterfield VM, Camp RM, Johnson JD. Time-dependent mediators of HPA axis activation following live Escherichia coli. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 301: R1648-57. PMID 21917906 DOI: 10.1152/ajpregu.00301.2011  0.8
2011 Hains LE, Loram LC, Taylor FR, Strand KA, Wieseler JL, Barrientos RM, Young JJ, Frank MG, Sobesky J, Martin TJ, Eisenach JC, Maier SF, Johnson JD, Fleshner M, Watkins LR. Prior laparotomy or corticosterone potentiates lipopolysaccharide-induced fever and sickness behaviors. Journal of Neuroimmunology. 239: 53-60. PMID 21907418 DOI: 10.1016/j.jneuroim.2011.08.011  0.8
2011 Porterfield VM, Zimomra ZR, Caldwell EA, Camp RM, Gabella KM, Johnson JD. Rat strain differences in restraint stress-induced brain cytokines. Neuroscience. 188: 48-54. PMID 21605631 DOI: 10.1016/j.neuroscience.2011.05.023  0.8
2008 Johnson JD, Cortez V, Kennedy SL, Foley TE, Hanson H, Fleshner M. Role of central beta-adrenergic receptors in regulating proinflammatory cytokine responses to a peripheral bacterial challenge. Brain, Behavior, and Immunity. 22: 1078-86. PMID 18468841 DOI: 10.1016/j.bbi.2008.03.007  0.8
2006 Nickerson M, Kennedy SL, Johnson JD, Fleshner M. Sexual dimorphism of the intracellular heat shock protein 72 response. Journal of Applied Physiology (Bethesda, Md. : 1985). 101: 566-75. PMID 16690792 DOI: 10.1152/japplphysiol.00259.2006  0.8
2006 Johnson JD, Fleshner M. Releasing signals, secretory pathways, and immune function of endogenous extracellular heat shock protein 72. Journal of Leukocyte Biology. 79: 425-34. PMID 16387837 DOI: 10.1189/jlb.0905523  0.8
2005 Johnson JD, Campisi J, Sharkey CM, Kennedy SL, Nickerson M, Greenwood BN, Fleshner M. Catecholamines mediate stress-induced increases in peripheral and central inflammatory cytokines. Neuroscience. 135: 1295-307. PMID 16165282 DOI: 10.1016/j.neuroscience.2005.06.090  0.8
2005 Fleshner M, Johnson JD. Endogenous extra-cellular heat shock protein 72: releasing signal(s) and function. International Journal of Hyperthermia : the Official Journal of European Society For Hyperthermic Oncology, North American Hyperthermia Group. 21: 457-71. PMID 16048842 DOI: 10.1080/02656730500088211  0.8
2005 Johnson JD, Campisi J, Sharkey CM, Kennedy SL, Nickerson M, Fleshner M. Adrenergic receptors mediate stress-induced elevations in extracellular Hsp72. Journal of Applied Physiology (Bethesda, Md. : 1985). 99: 1789-95. PMID 16037404 DOI: 10.1152/japplphysiol.00390.2005  0.8
2005 Kennedy SL, Nickerson M, Campisi J, Johnson JD, Smith TP, Sharkey C, Fleshner M. Splenic norepinephrine depletion following acute stress suppresses in vivo antibody response. Journal of Neuroimmunology. 165: 150-60. PMID 15978673 DOI: 10.1016/j.jneuroim.2005.05.001  0.8
2004 O'Connor KA, Ginsberg AB, Maksimova E, Wieseler Frank JL, Johnson JD, Spencer RL, Campeau S, Watkins LR, Maier SF. Stress-induced sensitization of the hypothalamic-pituitary adrenal axis is associated with alterations of hypothalamic and pituitary gene expression. Neuroendocrinology. 80: 252-63. PMID 15627803 DOI: 10.1159/000082876  0.8
2004 Johnson JD, O'Connor KA, Watkins LR, Maier SF. The role of IL-1beta in stress-induced sensitization of proinflammatory cytokine and corticosterone responses. Neuroscience. 127: 569-77. PMID 15283957 DOI: 10.1016/j.neuroscience.2004.05.046  0.8
2003 Fleshner M, Campisi J, Johnson JD. Can exercise stress facilitate innate immunity? A functional role for stress-induced extracellular Hsp72. Exercise Immunology Review. 9: 6-24. PMID 14686090  0.8
2003 O'Connor KA, Hansen MK, Rachal Pugh C, Deak MM, Biedenkapp JC, Milligan ED, Johnson JD, Wang H, Maier SF, Tracey KJ, Watkins LR. Further characterization of high mobility group box 1 (HMGB1) as a proinflammatory cytokine: central nervous system effects. Cytokine. 24: 254-65. PMID 14609567 DOI: 10.1016/j.cyto.2003.08.001  0.8
2003 O'Connor KA, Johnson JD, Hansen MK, Wieseler Frank JL, Maksimova E, Watkins LR, Maier SF. Peripheral and central proinflammatory cytokine response to a severe acute stressor. Brain Research. 991: 123-32. PMID 14575884 DOI: 10.1016/j.brainres.2003.08.006  0.8
2003 O'Connor KA, Johnson JD, Hammack SE, Brooks LM, Spencer RL, Watkins LR, Maier SF. Inescapable shock induces resistance to the effects of dexamethasone. Psychoneuroendocrinology. 28: 481-500. PMID 12689607 DOI: 10.1016/S0306-4530(02)00035-5  0.8
2003 Johnson JD, O'Connor KA, Hansen MK, Watkins LR, Maier SF. Effects of prior stress on LPS-induced cytokine and sickness responses. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 284: R422-32. PMID 12399247 DOI: 10.1152/ajpregu.00230.2002  0.8
2002 Johnson JD, O'Connor KA, Deak T, Stark M, Watkins LR, Maier SF. Prior stressor exposure sensitizes LPS-induced cytokine production. Brain, Behavior, and Immunity. 16: 461-76. PMID 12096891 DOI: 10.1006/brbi.2001.0638  0.8
2002 Johnson JD, O'Connor KA, Deak T, Spencer RL, Watkins LR, Maier SF. Prior stressor exposure primes the HPA axis. Psychoneuroendocrinology. 27: 353-65. PMID 11818171 DOI: 10.1016/S0306-4530(01)00057-9  0.8
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