| Year |
Citation |
Score |
| 2018 |
Weir MC, Shu ST, Patel RK, Hellwig S, Chen L, Tan L, Gray NS, Smithgall TE. Selective inhibition of the myeloid Src-family kinase Fgr potently suppresses AML cell growth in vitro and in vivo. Acs Chemical Biology. PMID 29763550 DOI: 10.1021/Acschembio.8B00154 |
0.348 |
|
| 2017 |
Weir MC, Hellwig S, Tan L, Liu Y, Gray NS, Smithgall TE. Dual inhibition of Fes and Flt3 tyrosine kinases potently inhibits Flt3-ITD+ AML cell growth. Plos One. 12: e0181178. PMID 28727840 DOI: 10.1371/Journal.Pone.0181178 |
0.354 |
|
| 2017 |
Weir MC, Shu S, Hellwig S, Patel R, Chen L, Tan L, Gray N, Smithgall T. A Unique N-Phenylbenzamide Inhibitor Targeting Non-Receptor Tyrosine Kinases Potently Suppresses AML Cell Growth In Vitro and In Vivo Blood. 130: 3813-3813. DOI: 10.1182/Blood.V130.Suppl_1.3813.3813 |
0.315 |
|
| 2015 |
Weir M, Hellwig S, Gray NS, Smithgall TE. Abstract A25: Dual inhibition of Flt3 and Fes tyrosine kinases potently blocks proliferation of AML cells expressing an active Flt3 mutant. Clinical Cancer Research. 21. DOI: 10.1158/1557-3265.Hemmal14-A25 |
0.368 |
|
| 2015 |
Weir MC, Hellwig S, Patel R, Gray NS, Smithgall TE. Abstract B180: Multi-targeted tyrosine kinase inhibitors potently suppress FLT3-ITD+ AML cell growth Molecular Cancer Therapeutics. 14. DOI: 10.1158/1535-7163.Targ-15-B180 |
0.368 |
|
| 2013 |
Weir M, Hellwig S, Gray NS, Smithgall TE. Abstract 2086: Dual inhibitors of Flt3 and c-Fest tyrosine kinases potently inhibit proliferation of AML cells expressing an active Flt3 mutant. Cancer Research. 73: 2086-2086. DOI: 10.1158/1538-7445.Am2013-2086 |
0.351 |
|
| 2012 |
Hellwig S, Miduturu CV, Kanda S, Zhang J, Filippakopoulos P, Salah E, Deng X, Choi HG, Zhou W, Hur W, Knapp S, Gray NS, Smithgall TE. Small-molecule inhibitors of the c-Fes protein-tyrosine kinase. Chemistry & Biology. 19: 529-40. PMID 22520759 DOI: 10.1016/J.Chembiol.2012.01.020 |
0.365 |
|
| 2011 |
Hellwig S, Smithgall TE. Structure and regulation of the c-Fes protein-tyrosine kinase. Frontiers in Bioscience (Landmark Edition). 16: 3146-55. PMID 21622225 DOI: 10.2741/3902 |
0.342 |
|
| 2011 |
Hellwig S, Gray NS, Smithgall TE. Abstract A234: Identification and characterization of inhibitor-resistant mutants of the protein-tyrosine kinase c-Fes. Molecular Cancer Therapeutics. 10. DOI: 10.1158/1535-7163.Targ-11-A234 |
0.369 |
|
| 2010 |
Aruscavage PJ, Hellwig S, Bass BL. Small DNA pieces in C. elegans are intermediates of DNA fragmentation during apoptosis. Plos One. 5: e11217. PMID 20585459 DOI: 10.1371/Journal.Pone.0011217 |
0.487 |
|
| 2010 |
Hellwig S, Smithgall TE. Abstract 4171: The role of the Src kinase family in determining three-dimensional epithelial cell morphology of colorectal cancer cell lines Cancer Research. 70: 4171-4171. DOI: 10.1158/1538-7445.Am10-4171 |
0.304 |
|
| 2010 |
Hellwig S, Zhang J, Filippakopoulos P, Salah E, Knapp S, Gray NS, Smithgall TE. Abstract 3683: Small molecule inhibitors of the Fes non-receptor tyrosine kinase Cancer Research. 70: 3683-3683. DOI: 10.1158/1538-7445.Am10-3683 |
0.322 |
|
| 2009 |
Shaffer JM, Hellwig S, Smithgall TE. Bimolecular fluorescence complementation demonstrates that the c-Fes protein-tyrosine kinase forms constitutive oligomers in living cells. Biochemistry. 48: 4780-8. PMID 19382747 DOI: 10.1021/Bi900238F |
0.344 |
|
| 2008 |
Hellwig S, Bass BL. A starvation-induced noncoding RNA modulates expression of Dicer-regulated genes. Proceedings of the National Academy of Sciences of the United States of America. 105: 12897-902. PMID 18723671 DOI: 10.1073/Pnas.0805118105 |
0.546 |
|
| 2005 |
Bass BL, Hellwig S, Hundley HA. A nuclear RNA is cut out for translation. Cell. 123: 181-3. PMID 16239133 DOI: 10.1016/J.Cell.2005.10.009 |
0.542 |
|
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