Year |
Citation |
Score |
2019 |
Lee S, Eyer J, Letournel F, Boumil E, Hall G, Shea TB. Neurofilaments form flexible bundles during neuritogenesis in culture and in mature axons in situ. Journal of Neuroscience Research. PMID 31304612 DOI: 10.1002/Jnr.24482 |
0.621 |
|
2017 |
Boumil EF, Vohnoutka R, Lee S, Pant H, Shea TB. Assembly and turnover of neurofilaments in growing axonal neurites. Biology Open. PMID 29158321 DOI: 10.1242/Bio.028795 |
0.382 |
|
2015 |
Lee S, Sohal IS, Therrien MA, Pal AK, Bello D, Shea TB. Additive Impairment of Synaptic Signaling in Cultured Cortical Neurons by Exogenously-Applied Oligomerized Amyloid-β and Airborne Nanoparticles Generated during Photocopying. Journal of Alzheimer's Disease : Jad. 47: 49-54. PMID 26402753 DOI: 10.3233/Jad-150099 |
0.305 |
|
2015 |
Boumil E, Vohnoutka R, Lee S, Shea TB. Early expression of the high molecular weight neurofilament subunit attenuates axonal neurite outgrowth. Neuroscience Letters. PMID 26225928 DOI: 10.1016/J.Neulet.2015.07.031 |
0.367 |
|
2014 |
Lee S, Shea TB. The high molecular weight neurofilament subunit plays an essential role in axonal outgrowth and stabilization. Biology Open. 3: 974-81. PMID 25260918 DOI: 10.1242/Bio.20149779 |
0.343 |
|
2014 |
Lee S, Pant HC, Shea TB. Divergent and convergent roles for kinases and phosphatases in neurofilament dynamics. Journal of Cell Science. 127: 4064-77. PMID 25015294 DOI: 10.1242/Jcs.153346 |
0.366 |
|
2014 |
Taddeo MA, Lee S, Shea TB. Synergistic inhibition of synaptic signaling in cortical cultures by subcytotoxic levels of oligomerized amyloid-β and iron: alleviation by zinc. Journal of Alzheimer's Disease : Jad. 41: 365-9. PMID 24625798 DOI: 10.3233/Jad-132696 |
0.307 |
|
2013 |
Lee S, Zemianek J, Shea TB. Rapid, reversible impairment of synaptic signaling in cultured cortical neurons by exogenously-applied amyloid-β. Journal of Alzheimer's Disease : Jad. 35: 395-402. PMID 23403533 DOI: 10.3233/Jad-122452 |
0.397 |
|
2013 |
Lee S, Shea TB. Regulation of tau proteolysis by phosphatases. Brain Research. 1495: 30-6. PMID 23159717 DOI: 10.1016/J.Brainres.2012.10.023 |
0.474 |
|
2013 |
Shea TB, Lee S. The discontinuous nature of neurofilament transport accommodates both establishment and repair of the axonal neurofilament array. Cytoskeleton (Hoboken, N.J.). 70: 67-73. PMID 23124969 DOI: 10.1002/Cm.21087 |
0.351 |
|
2012 |
Le MN, Kim W, Lee S, McKee AC, Hall GF. Multiple mechanisms of extracellular tau spreading in a non-transgenic tauopathy model. American Journal of Neurodegenerative Disease. 1: 316-33. PMID 23383401 |
0.672 |
|
2012 |
Lee S, Shea TB. Caspase-mediated truncation of tau potentiates aggregation. International Journal of Alzheimer's Disease. 2012: 731063. PMID 22988541 DOI: 10.1155/2012/731063 |
0.344 |
|
2012 |
Sunil N, Lee S, Shea TB. Interference with kinesin-based anterograde neurofilament axonal transport increases neurofilament-neurofilament bundling. Cytoskeleton (Hoboken, N.J.). 69: 371-9. PMID 22434685 DOI: 10.1002/Cm.21030 |
0.357 |
|
2012 |
Lee S, Kim W, Li Z, Hall GF. Accumulation of vesicle-associated human tau in distal dendrites drives degeneration and tau secretion in an in situ cellular tauopathy model. International Journal of Alzheimer's Disease. 2012: 172837. PMID 22315694 DOI: 10.1155/2012/172837 |
0.693 |
|
2012 |
Lee S, Lemere CA, Frost JL, Shea TB. Dietary supplementation with S-adenosyl methionine delayed amyloid-β and tau pathology in 3xTg-AD mice. Journal of Alzheimer's Disease : Jad. 28: 423-31. PMID 22045486 DOI: 10.3233/Jad-2011-111025 |
0.383 |
|
2011 |
Shea TB, Lee S. Neurofilament phosphorylation regulates axonal transport by an indirect mechanism: a merging of opposing hypotheses. Cytoskeleton (Hoboken, N.J.). 68: 589-95. PMID 21990272 DOI: 10.1002/Cm.20535 |
0.378 |
|
2011 |
Lee S, Hall GF, Shea TB. Potentiation of tau aggregation by cdk5 and GSK3β. Journal of Alzheimer's Disease : Jad. 26: 355-64. PMID 21677377 DOI: 10.3233/Jad-2011-102016 |
0.661 |
|
2011 |
Lee S, Sunil N, Tejada JM, Shea TB. Differential roles of kinesin and dynein in translocation of neurofilaments into axonal neurites. Journal of Cell Science. 124: 1022-31. PMID 21363889 DOI: 10.1242/Jcs.079046 |
0.36 |
|
2011 |
Lee S, Sunil N, Shea TB. C-terminal neurofilament phosphorylation fosters neurofilament-neurofilament associations that compete with axonal transport. Cytoskeleton (Hoboken, N.J.). 68: 8-17. PMID 20862740 DOI: 10.1002/Cm.20488 |
0.396 |
|
2011 |
Hall G, Saman S, Lee S. O4-04-05: Tau misprocessing leads to nonclassical tau secretion via vesicle release: implications for the spreading of tau lesions in Alzheimer's disease Alzheimer's & Dementia. 7: S690-S690. DOI: 10.1016/J.Jalz.2011.05.1993 |
0.711 |
|
2010 |
Kim W, Lee S, Hall GF. Secretion of human tau fragments resembling CSF-tau in Alzheimer's disease is modulated by the presence of the exon 2 insert. Febs Letters. 584: 3085-8. PMID 20553717 DOI: 10.1016/J.Febslet.2010.05.042 |
0.685 |
|
2010 |
Kushkuley J, Metkar S, Chan WK, Lee S, Shea TB. Aluminum induces neurofilament aggregation by stabilizing cross-bridging of phosphorylated c-terminal sidearms. Brain Research. 1322: 118-23. PMID 20132798 DOI: 10.1016/J.Brainres.2010.01.075 |
0.39 |
|
2010 |
Kim W, Lee S, Jung C, Ahmed A, Lee G, Hall GF. Interneuronal transfer of human tau between Lamprey central neurons in situ. Journal of Alzheimer's Disease : Jad. 19: 647-64. PMID 20110609 DOI: 10.3233/Jad-2010-1273 |
0.707 |
|
2010 |
Suchy J, Lee S, Ahmed A, Shea TB. Dietary supplementation with S-adenosyl methionine delays the onset of motor neuron pathology in a murine model of amyotrophic lateral sclerosis. Neuromolecular Medicine. 12: 86-97. PMID 19757209 DOI: 10.1007/S12017-009-8089-7 |
0.318 |
|
2010 |
Hall GF, Kim W, Lee S. Tau secretion to the extracellular space is modulated by the presence of the exon 2 alternatively spliced insert in cell culture and in situ tauopathy models Alzheimers & Dementia. 6. DOI: 10.1016/J.Jalz.2010.05.1756 |
0.612 |
|
2009 |
Kushkuley J, Chan WK, Lee S, Eyer J, Leterrier JF, Letournel F, Shea TB. Neurofilament cross-bridging competes with kinesin-dependent association of neurofilaments with microtubules. Journal of Cell Science. 122: 3579-86. PMID 19737816 DOI: 10.1242/Jcs.051318 |
0.383 |
|
2009 |
Shea TB, Chan WK, Kushkuley J, Lee S. Organizational dynamics, functions, and pathobiological dysfunctions of neurofilaments. Results and Problems in Cell Differentiation. 48: 29-45. PMID 19554281 DOI: 10.1007/400_2009_8 |
0.396 |
|
2009 |
Lee S, Jung C, Lee G, Hall GF. Exonic point mutations of human tau enhance its toxicity and cause characteristic changes in neuronal morphology, tau distribution and tau phosphorylation in the lamprey cellular model of tauopathy. Journal of Alzheimer's Disease : Jad. 16: 99-111. PMID 19158426 DOI: 10.3233/Jad-2009-0954 |
0.649 |
|
2009 |
Shea TB, Lee S, Kushkuley J, Dubey M, Chan WKH. Neurofilament dynamics: A tug of war by microtubule motors Future Neurology. 4: 351-362. DOI: 10.2217/Fnl.09.4 |
0.382 |
|
2008 |
Lee S, Chu B, Yao J, Shea TB, Hall GF. The glutamate-rich region of the larger lamprey neurofilament sidearm is essential for proper neurofilament architecture. Brain Research. 1231: 1-5. PMID 18675794 DOI: 10.1016/J.Brainres.2008.07.050 |
0.589 |
|
2008 |
Hall GF, Lee S, Ferreira A, Lee G. O4-01-04: Mitochondrial clumping is a central feature in the Tau neurodegeneration mechanism in an in situ tauopathy model Alzheimer's & Dementia. 4: T184-T184. DOI: 10.1016/J.Jalz.2008.05.503 |
0.608 |
|
2007 |
Zhang F, Ström AL, Fukada K, Lee S, Hayward LJ, Zhu H. Interaction between familial amyotrophic lateral sclerosis (ALS)-linked SOD1 mutants and the dynein complex. The Journal of Biological Chemistry. 282: 16691-9. PMID 17403682 DOI: 10.1074/Jbc.M609743200 |
0.347 |
|
2005 |
Jung C, Lee S, Ortiz D, Zhu Q, Julien JP, Shea TB. The high and middle molecular weight neurofilament subunits regulate the association of neurofilaments with kinesin: inhibition by phosphorylation of the high molecular weight subunit. Brain Research. Molecular Brain Research. 141: 151-5. PMID 16246456 DOI: 10.1016/J.Molbrainres.2005.08.009 |
0.348 |
|
2002 |
Hall GF, Lee S, Yao J. Neurofibrillary degeneration can be arrested in an in vivo cellular model of human tauopathy by application of a compound which inhibits tau filament formation in vitro. Journal of Molecular Neuroscience : Mn. 19: 253-60. PMID 12540050 DOI: 10.1385/Jmn:19:3:251 |
0.692 |
|
2001 |
Yabe JT, Chan WK, Chylinski TM, Lee S, Pimenta AF, Shea TB. The predominant form in which neurofilament subunits undergo axonal transport varies during axonal initiation, elongation, and maturation. Cell Motility and the Cytoskeleton. 48: 61-83. PMID 11124711 DOI: 10.1002/1097-0169(200101)48:1<61::Aid-Cm6>3.0.Co;2-S |
0.393 |
|
2000 |
Jung C, Yabe JT, Lee S, Shea TB. Hypophosphorylated neurofilament subunits undergo axonal transport more rapidly than more extensively phosphorylated subunits in situ. Cell Motility and the Cytoskeleton. 47: 120-9. PMID 11013392 DOI: 10.1002/1097-0169(200010)47:2<120::Aid-Cm3>3.0.Co;2-6 |
0.363 |
|
2000 |
Hall GF, Chu B, Lee S, Liu Y, Yao J. The single neurofilament subunit of the lamprey forms filaments and regulates axonal caliber and neuronal size in vivo. Cell Motility and the Cytoskeleton. 46: 166-82. PMID 10913964 DOI: 10.1002/1097-0169(200007)46:3<166::Aid-Cm2>3.0.Co;2-8 |
0.63 |
|
2000 |
Hall GF, Reinhold L, Lee S, Yao J. Staging of neurofibrillary degeneration in a unique cellular model of the role of human tau filament formation in neurofibrillary degenerative disease Neurobiology of Aging. 21: 20. DOI: 10.1016/S0197-4580(00)82767-0 |
0.651 |
|
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