Arjun Krishnaswamy, PhD - Publications

MCB Harvard University, Cambridge, MA, United States 
retinal circuit development

14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.


Year Citation  Score
2018 Duan X, Krishnaswamy A, Laboulaye MA, Liu J, Peng YR, Yamagata M, Toma K, Sanes JR. Cadherin Combinations Recruit Dendrites of Distinct Retinal Neurons to a Shared Interneuronal Scaffold. Neuron. PMID 30197236 DOI: 10.1016/j.neuron.2018.08.019  1
2018 Liu J, Reggiani JDS, Laboulaye MA, Pandey S, Chen B, Rubenstein JLR, Krishnaswamy A, Sanes JR. Tbr1 instructs laminar patterning of retinal ganglion cell dendrites. Nature Neuroscience. PMID 29632360 DOI: 10.1038/s41593-018-0127-z  1
2017 Peng YR, Tran NM, Krishnaswamy A, Kostadinov D, Martersteck EM, Sanes JR. Satb1 Regulates Contactin 5 to Pattern Dendrites of a Mammalian Retinal Ganglion Cell. Neuron. PMID 28781169 DOI: 10.1016/j.neuron.2017.07.019  1
2015 Krishnaswamy A, Yamagata M, Duan X, Hong YK, Sanes JR. Sidekick 2 directs formation of a retinal circuit that detects differential motion. Nature. 524: 466-70. PMID 26287463 DOI: 10.1038/nature14682  1
2015 Krishnaswamy A, Yamagata M, Duan X, Hong YK, Sanes JR. Sidekick 2 directs formation of a retinal circuit that detects differential motion Nature. 524: 466-470. DOI: 10.1038/nature14682  1
2014 Duan X, Krishnaswamy A, De la Huerta I, Sanes JR. Type II cadherins guide assembly of a direction-selective retinal circuit. Cell. 158: 793-807. PMID 25126785 DOI: 10.1016/j.cell.2014.06.047  1
2014 Lilley BN, Krishnaswamy A, Wang Z, Kishi M, Frank E, Sanes JR. SAD kinases control the maturation of nerve terminals in the mammalian peripheral and central nervous systems. Proceedings of the National Academy of Sciences of the United States of America. 111: 1138-43. PMID 24395778 DOI: 10.1073/pnas.1321990111  1
2012 Krishnaswamy A, Cooper E. Reactive oxygen species inactivate neuronal nicotinic acetylcholine receptors through a highly conserved cysteine near the intracellular mouth of the channel: implications for diseases that involve oxidative stress. The Journal of Physiology. 590: 39-47. PMID 21969449 DOI: 10.1113/jphysiol.2011.214007  1
2011 Davisson MT, Bronson RT, Tadenev AL, Motley WW, Krishnaswamy A, Seburn KL, Burgess RW. A spontaneous mutation in contactin 1 in the mouse. Plos One. 6: e29538. PMID 22242131 DOI: 10.1371/journal.pone.0029538  1
2010 Campanucci V, Krishnaswamy A, Cooper E. Diabetes depresses synaptic transmission in sympathetic ganglia by inactivating nAChRs through a conserved intracellular cysteine residue. Neuron. 66: 827-34. PMID 20620869 DOI: 10.1016/j.neuron.2010.06.010  1
2009 Caffery PM, Krishnaswamy A, Sanders T, Liu J, Hartlaub H, Klysik J, Cooper E, Hawrot E. Engineering neuronal nicotinic acetylcholine receptors with functional sensitivity to alpha-bungarotoxin: a novel alpha3-knock-in mouse. The European Journal of Neuroscience. 30: 2064-76. PMID 20128845 DOI: 10.1111/j.1460-9568.2009.07016.x  1
2009 Krishnaswamy A, Cooper E. An activity-dependent retrograde signal induces the expression of the high-affinity choline transporter in cholinergic neurons. Neuron. 61: 272-86. PMID 19186169 DOI: 10.1016/j.neuron.2008.11.025  1
2008 Campanucci VA, Krishnaswamy A, Cooper E. Mitochondrial reactive oxygen species inactivate neuronal nicotinic acetylcholine receptors and induce long-term depression of fast nicotinic synaptic transmission. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 1733-44. PMID 18272694 DOI: 10.1523/JNEUROSCI.5130-07.2008  1
2005 Rassadi S, Krishnaswamy A, Pié B, McConnell R, Jacob MH, Cooper E. A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 8555-66. PMID 16162937 DOI: 10.1523/JNEUROSCI.1983-05.2005  1
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