ZaoDung Ling - Publications

Affiliations: 
Rush University, Chicago, IL, United States 
Area:
Neuroscience Biology, Pharmacology

21 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2009 Wang S, Yan JY, Lo YK, Carvey PM, Ling Z. Dopaminergic and serotoninergic deficiencies in young adult rats prenatally exposed to the bacterial lipopolysaccharide. Brain Research. 1265: 196-204. PMID 19236855 DOI: 10.1016/J.Brainres.2009.02.022  0.485
2009 Ling Z, Zhu Y, Tong CW, Snyder JA, Lipton JW, Carvey PM. Prenatal lipopolysaccharide does not accelerate progressive dopamine neuron loss in the rat as a result of normal aging. Experimental Neurology. 216: 312-20. PMID 19133261 DOI: 10.1016/J.Expneurol.2008.12.004  0.597
2008 Aroutcheva A, Ling Z, Faro S. Prevotella bivia as a source of lipopolysaccharide in the vagina. Anaerobe. 14: 256-60. PMID 18849004 DOI: 10.1016/j.anaerobe.2008.08.002  0.364
2007 Zhu Y, Carvey PM, Ling Z. Altered glutathione homeostasis in animals prenatally exposed to lipopolysaccharide. Neurochemistry International. 50: 671-80. PMID 17291629 DOI: 10.1016/J.Neuint.2006.12.013  0.527
2007 Zhao C, Ling Z, Newman MB, Bhatia A, Carvey PM. TNF-alpha knockout and minocycline treatment attenuates blood-brain barrier leakage in MPTP-treated mice. Neurobiology of Disease. 26: 36-46. PMID 17234424 DOI: 10.1016/J.Nbd.2006.11.012  0.491
2006 Ling Z, Zhu Y, Tong Cw, Snyder JA, Lipton JW, Carvey PM. Progressive dopamine neuron loss following supra-nigral lipopolysaccharide (LPS) infusion into rats exposed to LPS prenatally. Experimental Neurology. 199: 499-512. PMID 16504177 DOI: 10.1016/J.Expneurol.2006.01.010  0.604
2005 Carvey PM, Chen EY, Lipton JW, Tong CW, Chang QA, Ling ZD. Intra-parenchymal injection of tumor necrosis factor-alpha and interleukin 1-beta produces dopamine neuron loss in the rat. Journal of Neural Transmission (Vienna, Austria : 1996). 112: 601-12. PMID 15583962 DOI: 10.1007/S00702-004-0222-Z  0.687
2004 Ling Z, Chang QA, Tong CW, Leurgans SE, Lipton JW, Carvey PM. Rotenone potentiates dopamine neuron loss in animals exposed to lipopolysaccharide prenatally. Experimental Neurology. 190: 373-83. PMID 15530876 DOI: 10.1016/J.Expneurol.2004.08.006  0.711
2004 Ling ZD, Chang Q, Lipton JW, Tong CW, Landers TM, Carvey PM. Combined toxicity of prenatal bacterial endotoxin exposure and postnatal 6-hydroxydopamine in the adult rat midbrain. Neuroscience. 124: 619-28. PMID 14980732 DOI: 10.1016/J.Neuroscience.2003.12.017  0.703
2003 Carvey PM, Chang Q, Lipton JW, Ling Z. Prenatal exposure to the bacteriotoxin lipopolysaccharide leads to long-term losses of dopamine neurons in offspring: a potential, new model of Parkinson's disease. Frontiers in Bioscience : a Journal and Virtual Library. 8: s826-37. PMID 12957870 DOI: 10.2741/1158  0.689
2002 Lipton JW, Vu TQ, Ling Z, Gyawali S, Mayer JR, Carvey PM. Prenatal cocaine exposure induces an attenuation of uterine blood flow in the rat. Neurotoxicology and Teratology. 24: 143-8. PMID 11943502 DOI: 10.1016/S0892-0362(01)00209-4  0.345
2002 Gayle DA, Ling Z, Tong C, Landers T, Lipton JW, Carvey PM. Lipopolysaccharide (LPS)-induced dopamine cell loss in culture: roles of tumor necrosis factor-alpha, interleukin-1beta, and nitric oxide. Brain Research. Developmental Brain Research. 133: 27-35. PMID 11850061 DOI: 10.1016/S0165-3806(01)00315-7  0.521
2002 Ling Z, Gayle DA, Ma SY, Lipton JW, Tong CW, Hong JS, Carvey PM. In utero bacterial endotoxin exposure causes loss of tyrosine hydroxylase neurons in the postnatal rat midbrain. Movement Disorders : Official Journal of the Movement Disorder Society. 17: 116-24. PMID 11835448 DOI: 10.1002/Mds.10078  0.553
2000 Kordower JH, Emborg ME, Bloch J, Ma SY, Chu Y, Leventhal L, McBride J, Chen EY, Palfi S, Roitberg BZ, Brown WD, Holden JE, Pyzalski R, Taylor MD, Carvey P, ... Ling Z, et al. Neurodegeneration prevented by lentiviral vector delivery of GDNF in primate models of Parkinson's disease. Science (New York, N.Y.). 290: 767-73. PMID 11052933 DOI: 10.1126/Science.290.5492.767  0.427
1999 Lipton JW, Ling Z, Vu TQ, Robie HC, Mangan KP, Weese-Mayer DE, Carvey PM. Prenatal cocaine exposure reduces glial cell line-derived neurotrophic factor (GDNF) in the striatum and the carotid body of the rat: implications for DA neurodevelopment. Brain Research. Developmental Brain Research. 118: 231-5. PMID 10611525 DOI: 10.1016/S0165-3806(99)00127-3  0.433
1999 Lipton JW, Mayer JR, Vu TQ, Ling Z, Carvey PM, Weese-Mayer DE. Cocaine-Induced Reductions in Fetal Dopamine (DA) and Glial Cell Line-Derived Neurotrophic Factor (GDNF) Are a Result of Attenuated Uterine Blood Flow Pediatric Research. 45: 68A-68A. DOI: 10.1203/00006450-199904020-00408  0.41
1998 Lipton JW, Robie HS, Ling Z, Weese-Mayer DE, Carvey PM. Uterine Position Determines the Extent of Dopamine Reduction after Chronic Prenatal Cocaine Exposure. Annals of the New York Academy of Sciences. 844: 314-323. PMID 29090821 DOI: 10.1111/J.1749-6632.1998.Tb08246.X  0.445
1998 Lipton JW, Robie HC, Ling Z, Weese-Mayer DE, Carvey PM. The magnitude of brain dopamine depletion from prenatal cocaine exposure is a function of uterine position. Neurotoxicology and Teratology. 20: 373-82. PMID 9697963 DOI: 10.1016/S0892-0362(97)00143-8  0.358
1998 Lipton JW, Robie HS, Ling Z, Weese-Mayer DE, Carvey PM. The Magnitude of Whole Brain Dopamine Depletion from Prenatal Cocaine Exposure is Inversely Related to Brain Cocaine Levels and is a Function of Uterine Position 282 Pediatric Research. 43: 51-51. DOI: 10.1203/00006450-199804001-00303  0.352
1997 Carvey PM, Ling ZD. The case for neuroprotection with dopamine agonists Clinical Neuropharmacology. 20: S8-S21. DOI: 10.1097/00002826-199720001-00003  0.367
1996 Ling ZD, Pieri SC, Carvey PM. Comparison of the neurotoxicity of dihydroxyphenylalanine stereoisomers in cultured dopamine neurons. Clinical Neuropharmacology. 19: 360-5. PMID 8829000 DOI: 10.1097/00002826-199619040-00010  0.463
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