| Year |
Citation |
Score |
| 2023 |
Becerra-Calixto A, Mukherjee A, Ramirez S, Sepulveda S, Sinha T, Al-Lahham R, De Gregorio N, Gherardelli C, Soto C. Lewy Body-like Pathology and Loss of Dopaminergic Neurons in Midbrain Organoids Derived from Familial Parkinson's Disease Patient. Cells. 12. PMID 36831291 DOI: 10.3390/cells12040625 |
0.488 |
|
| 2023 |
Cuanalo-Contreras K, Schulz J, Mukherjee A, Park KW, Armijo E, Soto C. Extensive accumulation of misfolded protein aggregates during natural aging and senescence. Frontiers in Aging Neuroscience. 14: 1090109. PMID 36778589 DOI: 10.3389/fnagi.2022.1090109 |
0.571 |
|
| 2022 |
Mukherjee A, Cuanalo-Contreras K, Sood A, Soto C. Development of a novel pharmacophore model to screen specific inhibitors for the serine-threonine protein phosphatase calcineurin. Biochemistry and Biophysics Reports. 31: 101311. PMID 36032402 DOI: 10.1016/j.bbrep.2022.101311 |
0.475 |
|
| 2022 |
Mukherjee A, Al-Lahham R, Corkins ME, Samanta S, Schmeichel AM, Singer W, Low PA, Govindaraju T, Soto C. Identification of Multicolor Fluorescent Probes for Heterogeneous Aβ Deposits in Alzheimer's Disease. Frontiers in Aging Neuroscience. 13: 802614. PMID 35185519 DOI: 10.3389/fnagi.2021.802614 |
0.486 |
|
| 2021 |
Ramirez S, Mukherjee A, Sepulveda SE, Gherardelli C, Becerra-Calixto A, Bravo-Vasquez N, Soto C. Protocol for controlled cortical impact in human cerebral organoids to model traumatic brain injury. Star Protocols. 2: 100987. PMID 34927096 DOI: 10.1016/j.xpro.2021.100987 |
0.433 |
|
| 2021 |
Ramirez S, Mukherjee A, Sepulveda S, Becerra-Calixto A, Bravo-Vasquez N, Gherardelli C, Chavez M, Soto C. Modeling Traumatic Brain Injury in Human Cerebral Organoids. Cells. 10. PMID 34685663 DOI: 10.3390/cells10102683 |
0.477 |
|
| 2021 |
Diao X, Wang F, Becerra-Calixto A, Soto C, Mukherjee A. Induced Pluripotent Stem Cell-Derived Dopaminergic Neurons from Familial Parkinson's Disease Patients Display α-Synuclein Pathology and Abnormal Mitochondrial Morphology. Cells. 10. PMID 34572052 DOI: 10.3390/cells10092402 |
0.488 |
|
| 2021 |
Mukherjee A, Becerra Calixto AD, Chavez M, Delgado JP, Soto C. Mitochondrial transplant to replenish damaged mitochondria: A novel therapeutic strategy for neurodegenerative diseases? Progress in Molecular Biology and Translational Science. 177: 49-63. PMID 33453942 DOI: 10.1016/bs.pmbts.2020.10.001 |
0.521 |
|
| 2020 |
Shahnawaz M, Mukherjee A, Pritzkow S, Mendez N, Rabadia P, Liu X, Hu B, Schmeichel A, Singer W, Wu G, Tsai AL, Shirani H, Nilsson KPR, Low PA, Soto C. Discriminating α-synuclein strains in Parkinson's disease and multiple system atrophy. Nature. PMID 32025029 DOI: 10.1038/S41586-020-1984-7 |
0.573 |
|
| 2017 |
Mukherjee A, Morales-Scheihing D, Salvadores N, Moreno-Gonzalez I, Gonzalez C, Taylor-Presse K, Mendez N, Shahnawaz M, Gaber AO, Sabek OM, Fraga DW, Soto C. Induction of IAPP amyloid deposition and associated diabetic abnormalities by a prion-like mechanism. The Journal of Experimental Medicine. PMID 28765400 DOI: 10.1084/Jem.20161134 |
0.584 |
|
| 2017 |
Shahnawaz M, Park KW, Mukherjee A, Diaz-Espinoza R, Soto C. Prion-like characteristics of the bacterial protein Microcin E492. Scientific Reports. 7: 45720. PMID 28361921 DOI: 10.1038/Srep45720 |
0.581 |
|
| 2017 |
Mukherjee A, Soto C. Prion-Like Protein Aggregates and Type 2 Diabetes. Cold Spring Harbor Perspectives in Medicine. PMID 28159831 DOI: 10.1101/Cshperspect.A024315 |
0.639 |
|
| 2016 |
Cuanalo-Contreras K, Park KW, Mukherjee A, Millán-Pérez Peña L, Soto C. Delaying aging in Caenorhabditis elegans with protein aggregation inhibitors. Biochemical and Biophysical Research Communications. PMID 27810360 DOI: 10.1016/J.Bbrc.2016.10.143 |
0.582 |
|
| 2015 |
Mukherjee A, Syeb K, Concannon J, Callegari K, Soto C, Glicksman MA. Development of a Fluorescent Quenching Based High Throughput Assay to Screen for Calcineurin Inhibitors. Plos One. 10: e0131297. PMID 26176772 DOI: 10.1371/Journal.Pone.0131297 |
0.592 |
|
| 2015 |
Mukherjee A, Morales-Scheihing D, Butler PC, Soto C. Type 2 diabetes as a protein misfolding disease. Trends in Molecular Medicine. 21: 439-49. PMID 25998900 DOI: 10.1016/J.Molmed.2015.04.005 |
0.624 |
|
| 2013 |
Cuanalo-Contreras K, Mukherjee A, Soto C. Role of protein misfolding and proteostasis deficiency in protein misfolding diseases and aging. International Journal of Cell Biology. 2013: 638083. PMID 24348562 DOI: 10.1155/2013/638083 |
0.604 |
|
| 2013 |
Barria MA, Mukherjee A, Gonzalez-Romero D, Morales R, Soto C. Correction: De Novo Generation of Infectious Prions In Vitro Produces a New Disease Phenotype Plos Pathogens. 9. DOI: 10.1371/Annotation/4B55946A-Edb5-4Feb-Aeb5-2Ee160394D17 |
0.478 |
|
| 2012 |
Diaz-Espinoza R, Mukherjee A, Soto C. Kosmotropic anions promote conversion of recombinant prion protein into a PrPSc-like misfolded form. Plos One. 7: e31678. PMID 22347503 DOI: 10.1371/Journal.Pone.0031678 |
0.558 |
|
| 2011 |
Mukherjee A, Soto C. Role of calcineurin in neurodegeneration produced by misfolded proteins and endoplasmic reticulum stress. Current Opinion in Cell Biology. 23: 223-30. PMID 21295458 DOI: 10.1016/J.Ceb.2010.12.006 |
0.588 |
|
| 2010 |
Mukherjee A, Morales-Scheihing D, Gonzalez-Romero D, Green K, Taglialatela G, Soto C. Calcineurin inhibition at the clinical phase of prion disease reduces neurodegeneration, improves behavioral alterations and increases animal survival. Plos Pathogens. 6: e1001138. PMID 20949081 DOI: 10.1371/Journal.Ppat.1001138 |
0.555 |
|
| 2009 |
Barria MA, Mukherjee A, Gonzalez-Romero D, Morales R, Soto C. De novo generation of infectious prions in vitro produces a new disease phenotype. Plos Pathogens. 5: e1000421. PMID 19436715 DOI: 10.1371/Journal.Ppat.1000421 |
0.561 |
|
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