| Year |
Citation |
Score |
| 2024 |
Lin D, Gold A, Kaye S, Atkinson JR, Tol M, Sas A, Segal B, Tontonoz P, Zhu J, Gao J. Arachidonic acid mobilization and peroxidation promote microglial dysfunction in Aβ pathology. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 38866484 DOI: 10.1523/JNEUROSCI.0202-24.2024 |
0.481 |
|
| 2024 |
Jerome AD, Sas AR, Wang Y, Hammond LA, Wen J, Atkinson JR, Webb A, Liu T, Segal BM. Cytokine polarized, alternatively activated bone marrow neutrophils drive axon regeneration. Nature Immunology. PMID 38811815 DOI: 10.1038/s41590-024-01836-7 |
0.57 |
|
| 2023 |
Segal B, Jerome A, Sas A, Wang Y, Wen J, Atkinson J, Webb A, Liu T. Cytokine polarized, alternatively activated bone marrow neutrophils drive axon regeneration. Research Square. PMID 37961609 DOI: 10.21203/rs.3.rs-3491540/v1 |
0.576 |
|
| 2023 |
Zhang Y, Atkinson J, Burd CE, Graves J, Segal BM. Biological aging in multiple sclerosis. Multiple Sclerosis (Houndmills, Basingstoke, England). 13524585231204122. PMID 37877740 DOI: 10.1177/13524585231204122 |
0.513 |
|
| 2023 |
Mannion JM, Segal BM, McLoughlin RM, Lalor SJ. Respiratory tract Moraxella catarrhalis and Klebsiella pneumoniae can promote pathogenicity of myelin-reactive Th17 cells. Mucosal Immunology. PMID 37088262 DOI: 10.1016/j.mucimm.2023.04.003 |
0.442 |
|
| 2023 |
Gardner AM, Atkinson JR, Wilkinson NM, Jerome AD, Bellinger CE, Sas AR, Segal BM. TAM receptor signaling dictates lesion location and clinical phenotype during experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 375: 578016. PMID 36708633 DOI: 10.1016/j.jneuroim.2023.578016 |
0.595 |
|
| 2022 |
Jerome AD, Atkinson JR, McVey Moffatt AL, Sepeda JA, Segal BM, Sas AR. Characterization of Zymosan-Modulated Neutrophils With Neuroregenerative Properties. Frontiers in Immunology. 13: 912193. PMID 35711408 DOI: 10.3389/fimmu.2022.912193 |
0.571 |
|
| 2022 |
Atkinson JR, Jerome AD, Sas AR, Munie A, Wang C, Ma A, Arnold WD, Segal BM. Biological aging of CNS-resident cells alters the clinical course and immunopathology of autoimmune demyelinating disease. Jci Insight. PMID 35511417 DOI: 10.1172/jci.insight.158153 |
0.618 |
|
| 2021 |
Mockus TE, Munie A, Atkinson JR, Segal BM. Encephalitogenic and Regulatory CD8 T Cells in Multiple Sclerosis and Its Animal Models. Journal of Immunology (Baltimore, Md. : 1950). 206: 3-10. PMID 33443060 DOI: 10.4049/jimmunol.2000797 |
0.571 |
|
| 2020 |
Kalinski AL, Yoon C, Huffman LD, Duncker PC, Kohen R, Passino R, Hafner H, Johnson C, Kawaguchi R, Carbajal KS, Jara JS, Hollis ER, Geschwind DH, Segal BM, Giger R. Analysis of the immune response to sciatic nerve injury identifies efferocytosis as a key mechanism of nerve debridement. Elife. 9. PMID 33263277 DOI: 10.7554/eLife.60223 |
0.714 |
|
| 2020 |
Sas AR, Carbajal KS, Jerome AD, Menon R, Yoon C, Kalinski AL, Giger RJ, Segal BM. A new neutrophil subset promotes CNS neuron survival and axon regeneration. Nature Immunology. PMID 33106668 DOI: 10.1038/s41590-020-00813-0 |
0.319 |
|
| 2020 |
Xu J, Neal LM, Ganguly A, Kolbe JL, Hargarten JC, Elsegeiny W, Hollingsworth C, He X, Ivey M, Lopez R, Zhao J, Segal B, Williamson PR, Olszewski MA. Chemokine receptor CXCR3 is required for lethal brain pathology but not pathogen clearance during cryptococcal meningoencephalitis. Science Advances. 6: eaba2502. PMID 32596454 DOI: 10.1126/Sciadv.Aba2502 |
0.341 |
|
| 2019 |
Parker Harp CR, Archambault AS, Cheung M, Williams JW, Czepielewski RS, Duncker PC, Kilgore AJ, Miller AT, Segal BM, Kim AHJ, Randolph GJ, Wu GF. Neutrophils promote VLA-4-dependent B cell antigen presentation and accumulation within the meninges during neuroinflammation. Proceedings of the National Academy of Sciences of the United States of America. PMID 31699814 DOI: 10.1073/pnas.1909098116 |
0.727 |
|
| 2019 |
Kuhbandner K, Hammer A, Haase S, Terbrack E, Hoffmann A, Schippers A, Wagner N, Hussain RZ, Miller-Little WA, Koh AY, Stoolman JS, Segal BM, Linker RA, Stüve O. MAdCAM-1-Mediated Intestinal Lymphocyte Homing Is Critical for the Development of Active Experimental Autoimmune Encephalomyelitis. Frontiers in Immunology. 10: 903. PMID 31114574 DOI: 10.3389/fimmu.2019.00903 |
0.402 |
|
| 2019 |
Segal BM. The Diversity of Encephalitogenic CD4+ T Cells in Multiple Sclerosis and Its Animal Models. Journal of Clinical Medicine. 8. PMID 30669462 DOI: 10.3390/jcm8010120 |
0.378 |
|
| 2018 |
Segal BM. Enhancing natural killer cells is beneficial in multiple sclerosis - Commentary. Multiple Sclerosis (Houndmills, Basingstoke, England). 1352458518809296. PMID 30421650 DOI: 10.1177/1352458518809296 |
0.327 |
|
| 2018 |
Giles DA, Duncker PC, Wilkinson NM, Washnock-Schmid JM, Segal BM. CNS resident classical DCs play a critical role in CNS autoimmune disease. The Journal of Clinical Investigation. PMID 30226829 DOI: 10.1172/JCI123708 |
0.769 |
|
| 2018 |
Stoolman JS, Duncker PC, Huber AK, Giles DA, Washnock-Schmid JM, Soulika AM, Segal BM. An IFNγ/CXCL2 regulatory pathway determines lesion localization during EAE. Journal of Neuroinflammation. 15: 208. PMID 30012158 DOI: 10.1186/s12974-018-1237-y |
0.753 |
|
| 2017 |
Duncker PC, Stoolman JS, Huber AK, Segal BM. GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease Induction. Journal of Immunology (Baltimore, Md. : 1950). PMID 29288202 DOI: 10.4049/jimmunol.1701484 |
0.762 |
|
| 2017 |
Giles DA, Washnock-Schmid JM, Duncker PC, Dahlawi S, Ponath G, Pitt D, Segal BM. Myeloid cell plasticity in the evolution of central nervous system autoimmunity. Annals of Neurology. PMID 29283442 DOI: 10.1002/Ana.25128 |
0.754 |
|
| 2017 |
Neal LM, Xing E, Xu J, Kolbe JL, Osterholzer JJ, Segal BM, Williamson PR, Olszewski MA. CD4(+) T Cells Orchestrate Lethal Immune Pathology despite Fungal Clearance during Cryptococcus neoformans Meningoencephalitis. Mbio. 8. PMID 29162707 DOI: 10.1128/Mbio.01415-17 |
0.326 |
|
| 2016 |
Grifka-Walk HM, Segal BM. T-bet promotes the accumulation of encephalitogenic Th17 cells in the CNS. Journal of Neuroimmunology. PMID 27242075 DOI: 10.1016/j.jneuroim.2016.05.007 |
0.391 |
|
| 2016 |
Segal BM, Giger RJ. Stable biomarker for plastic microglia. Proceedings of the National Academy of Sciences of the United States of America. PMID 26966229 DOI: 10.1073/pnas.1601669113 |
0.369 |
|
| 2016 |
Phares TW, DiSano KD, Stohlman SA, Segal BM, Bergmann CC. CXCL13 promotes isotype-switched B cell accumulation to the central nervous system during viral encephalomyelitis. Brain, Behavior, and Immunity. PMID 26795429 DOI: 10.1016/J.Bbi.2016.01.016 |
0.344 |
|
| 2015 |
Carbajal KS, Mironova Y, Ulrich-Lewis JT, Kulkarni D, Grifka-Walk HM, Huber AK, Shrager P, Giger RJ, Segal BM. Th Cell Diversity in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis. Journal of Immunology (Baltimore, Md. : 1950). 195: 2552-9. PMID 26238492 DOI: 10.4049/Jimmunol.1501097 |
0.312 |
|
| 2015 |
Grifka-Walk HM, Giles DA, Segal BM. IL-12-polarized Th1 cells produce GM-CSF and induce EAE independent of IL-23. European Journal of Immunology. 45: 2780-6. PMID 26220255 DOI: 10.1002/eji.201545800 |
0.33 |
|
| 2015 |
Baldwin KT, Carbajal KS, Segal BM, Giger RJ. Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration. Proceedings of the National Academy of Sciences of the United States of America. 112: 2581-6. PMID 25675510 DOI: 10.1073/pnas.1423221112 |
0.307 |
|
| 2015 |
Rumble JM, Huber AK, Krishnamoorthy G, Srinivasan A, Giles DA, Zhang X, Wang L, Segal BM. Neutrophil-related factors as biomarkers in EAE and MS. The Journal of Experimental Medicine. 212: 23-35. PMID 25559893 DOI: 10.1084/Jem.20141015 |
0.408 |
|
| 2014 |
Rumble J, Segal BM. In vitro polarization of T-helper cells. Methods in Molecular Biology (Clifton, N.J.). 1193: 105-13. PMID 25151001 DOI: 10.1007/978-1-4939-1212-4_11 |
0.301 |
|
| 2014 |
Segal BM. Stage-specific immune dysregulation in multiple sclerosis. Journal of Interferon & Cytokine Research : the Official Journal of the International Society For Interferon and Cytokine Research. 34: 633-40. PMID 25084180 DOI: 10.1089/jir.2014.0025 |
0.35 |
|
| 2014 |
Stoolman JS, Duncker PC, Huber AK, Segal BM. Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950). 193: 564-70. PMID 24928987 DOI: 10.4049/jimmunol.1400825 |
0.746 |
|
| 2013 |
Grifka-Walk HM, Lalor SJ, Segal BM. Highly polarized Th17 cells induce EAE via a T-bet independent mechanism. European Journal of Immunology. 43: 2824-31. PMID 23878008 DOI: 10.1002/eji.201343723 |
0.334 |
|
| 2013 |
Lalor SJ, Segal BM. Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent. European Journal of Immunology. 43: 2866-74. PMID 23873018 DOI: 10.1002/eji.201343499 |
0.45 |
|
| 2013 |
Sandy AR, Stoolman J, Malott K, Pongtornpipat P, Segal BM, Maillard I. Notch signaling regulates T cell accumulation and function in the central nervous system during experimental autoimmune encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950). 191: 1606-13. PMID 23825310 DOI: 10.4049/Jimmunol.1301116 |
0.361 |
|
| 2013 |
Braley TJ, Segal BM. B-cell targeting agents in the treatment of multiple sclerosis. Current Treatment Options in Neurology. 15: 259-69. PMID 23609780 DOI: 10.1007/s11940-013-0232-y |
0.334 |
|
| 2012 |
Rao P, Segal BM. Experimental autoimmune encephalomyelitis. Methods in Molecular Biology (Clifton, N.J.). 900: 363-80. PMID 22933079 DOI: 10.1007/978-1-60761-720-4_18 |
0.388 |
|
| 2012 |
Segal BM. The unwavering commitment of regulatory T cells in the suppression of autoimmune encephalomyelitis: another aspect of immune privilege in the CNS. European Journal of Immunology. 42: 1102-5. PMID 22539285 DOI: 10.1002/eji.201242567 |
0.405 |
|
| 2011 |
Rainey-Barger EK, Rumble JM, Lalor SJ, Esen N, Segal BM, Irani DN. The lymphoid chemokine, CXCL13, is dispensable for the initial recruitment of B cells to the acutely inflamed central nervous system. Brain, Behavior, and Immunity. 25: 922-31. PMID 20933590 DOI: 10.1016/J.Bbi.2010.10.002 |
0.42 |
|
| 2010 |
Kroenke MA, Chensue SW, Segal BM. EAE mediated by a non-IFN-γ/non-IL-17 pathway. European Journal of Immunology. 40: 2340-8. PMID 20540117 DOI: 10.1002/Eji.201040489 |
0.317 |
|
| 2010 |
King IL, Kroenke MA, Segal BM. GM-CSF-dependent, CD103+ dermal dendritic cells play a critical role in Th effector cell differentiation after subcutaneous immunization. The Journal of Experimental Medicine. 207: 953-61. PMID 20421390 DOI: 10.1084/jem.20091844 |
0.339 |
|
| 2010 |
Segal BM. Th17 cells in autoimmune demyelinating disease. Seminars in Immunopathology. 32: 71-7. PMID 20195867 DOI: 10.1007/s00281-009-0186-z |
0.346 |
|
| 2009 |
King IL, Dickendesher TL, Segal BM. Circulating Ly-6C+ myeloid precursors migrate to the CNS and play a pathogenic role during autoimmune demyelinating disease. Blood. 113: 3190-7. PMID 19196868 DOI: 10.1182/blood-2008-07-168575 |
0.424 |
|
| 2008 |
Carlson T, Kroenke M, Rao P, Lane TE, Segal B. The Th17-ELR+ CXC chemokine pathway is essential for the development of central nervous system autoimmune disease. The Journal of Experimental Medicine. 205: 811-23. PMID 18347102 DOI: 10.1084/jem.20072404 |
0.437 |
|
| 2007 |
Segal BM. The role of natural killer cells in curbing neuroinflammation. Journal of Neuroimmunology. 191: 2-7. PMID 17904646 DOI: 10.1016/j.jneuroim.2007.09.006 |
0.343 |
|
| 2007 |
Deshpande P, King IL, Segal BM. Cutting edge: CNS CD11c+ cells from mice with encephalomyelitis polarize Th17 cells and support CD25+CD4+ T cell-mediated immunosuppression, suggesting dual roles in the disease process. Journal of Immunology (Baltimore, Md. : 1950). 178: 6695-9. PMID 17513712 |
0.371 |
|
| 2007 |
Kroenke MA, Segal BM. Th17 and Th1 responses directed against the immunizing epitope, as opposed to secondary epitopes, dominate the autoimmune repertoire during relapses of experimental autoimmune encephalomyelitis. Journal of Neuroscience Research. 85: 1685-93. PMID 17465017 DOI: 10.1002/jnr.21291 |
0.359 |
|
| 2006 |
Bagaeva LV, Rao P, Powers JM, Segal BM. CXC chemokine ligand 13 plays a role in experimental autoimmune encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950). 176: 7676-85. PMID 16751415 |
0.377 |
|
| 2006 |
Deshpande P, King IL, Segal BM. IL-12 driven upregulation of P-selectin ligand on myelin-specific T cells is a critical step in an animal model of autoimmune demyelination. Journal of Neuroimmunology. 173: 35-44. PMID 16413063 DOI: 10.1016/j.jneuroim.2005.11.016 |
0.323 |
|
| 2005 |
Segal BM. CNS chemokines, cytokines, and dendritic cells in autoimmune demyelination. Journal of the Neurological Sciences. 228: 210-4. PMID 15694210 DOI: 10.1016/j.jns.2004.10.014 |
0.426 |
|
| 2004 |
Rao P, Segal BM. Experimental autoimmune encephalomyelitis. Methods in Molecular Medicine. 102: 363-75. PMID 15286395 DOI: 10.1385/1-59259-805-6:363 |
0.394 |
|
| 2003 |
Bagaeva LV, Williams LP, Segal BM. IL-12 dependent/IFN gamma independent expression of CCR5 by myelin-reactive T cells correlates with encephalitogenicity. Journal of Neuroimmunology. 137: 109-16. PMID 12667654 DOI: 10.1016/S0165-5728(03)00079-1 |
0.377 |
|
| 2003 |
Segal BM. Experimental autoimmune encephalomyelitis: cytokines, effector T cells, and antigen-presenting cells in a prototypical Th1-mediated autoimmune disease. Current Allergy and Asthma Reports. 3: 86-93. PMID 12543000 |
0.4 |
|
| 2002 |
Ichikawa HT, Williams LP, Segal BM. Activation of APCs through CD40 or Toll-like receptor 9 overcomes tolerance and precipitates autoimmune disease. Journal of Immunology (Baltimore, Md. : 1950). 169: 2781-7. PMID 12193753 |
0.333 |
|
| 2000 |
Hilchey SP, Newman CL, Segal BM, Rosenblatt JD. Enhanctd t cell meduted anti-tumorimmunity is observed in b cell deficient mice Blood. 96: 27a. |
0.302 |
|
| 1994 |
Voskuhl RR, Robinson ED, Segal BM, Tranquill L, Camphausen K, Albert PS, Richert JR, McFarland HF. HLA restriction and TCR usage of T lymphocytes specific for a novel candidate autoantigen, X2 MBP, in multiple sclerosis. Journal of Immunology (Baltimore, Md. : 1950). 153: 4834-44. PMID 7525716 |
0.309 |
|
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