David Acton - Publications
Affiliations: | 2017- | Molecular Neuroscience Laboratories | Salk Institute for Biological Studies, La Jolla, CA, United States |
Area:
Glia, gliotransmission, spinal cord, electrophysiology, locomotionYear | Citation | Score | |||
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2023 | Ren X, Liu S, Virlogeux A, Kang SJ, Brusch J, Liu Y, Dymecki SM, Han S, Goulding M, Acton D. Identification of an essential spinoparabrachial pathway for mechanical itch. Neuron. PMID 37023756 DOI: 10.1016/j.neuron.2023.03.013 | 0.732 | |||
2019 | Acton D, Ren X, Di Costanzo S, Dalet A, Bourane S, Bertocchi I, Eva C, Goulding M. Spinal Neuropeptide Y1 Receptor-Expressing Neurons Form an Essential Excitatory Pathway for Mechanical Itch. Cell Reports. 28: 625-639.e6. PMID 31315043 DOI: 10.1016/J.Celrep.2019.06.033 | 0.642 | |||
2018 | Acton D, Broadhead MJ, Miles GB. Modulation of spinal motor networks by astrocyte-derived adenosine is dependent on D1-like dopamine receptor signalling. Journal of Neurophysiology. PMID 29790837 DOI: 10.1152/Jn.00783.2017 | 0.692 | |||
2017 | Koch SC, Acton D, Goulding M. Spinal Circuits for Touch, Pain, and Itch. Annual Review of Physiology. PMID 28961064 DOI: 10.1146/Annurev-Physiol-022516-034303 | 0.699 | |||
2017 | Acton D, Miles GB. Gliotransmission and adenosinergic modulation: insights from mammalian spinal motor networks. Journal of Neurophysiology. jn.00230.2017. PMID 28954893 DOI: 10.1152/Jn.00230.2017 | 0.743 | |||
2017 | Acton D, Miles GB. Differential regulation of NMDA receptors by D-serine and glycine in mammalian spinal locomotor networks. Journal of Neurophysiology. jn.00810.2016. PMID 28202572 DOI: 10.1152/Jn.00810.2016 | 0.682 | |||
2015 | Acton D, Miles GB. Stimulation of Glia Reveals Modulation of Mammalian Spinal Motor Networks by Adenosine. Plos One. 10: e0134488. PMID 26252389 DOI: 10.1371/Journal.Pone.0134488 | 0.738 | |||
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