Year |
Citation |
Score |
2014 |
Kassas A, Moura IC, Yamashita Y, Scheffel J, Guérin-Marchand C, Blank U, Sims PJ, Wiedmer T, Monteiro RC, Rivera J, Charles N, Benhamou M. Regulation of the tyrosine phosphorylation of Phospholipid Scramblase 1 in mast cells that are stimulated through the high-affinity IgE receptor. Plos One. 9: e109800. PMID 25289695 DOI: 10.1371/Journal.Pone.0109800 |
0.327 |
|
2011 |
Chen CW, Sowden M, Zhao Q, Wiedmer T, Sims PJ. Nuclear phospholipid scramblase 1 prolongs the mitotic expansion of granulocyte precursors during G-CSF-induced granulopoiesis. Journal of Leukocyte Biology. 90: 221-33. PMID 21447647 DOI: 10.1189/Jlb.0111006 |
0.352 |
|
2009 |
Bateman A, Finn RD, Sims PJ, Wiedmer T, Biegert A, Söding J. Phospholipid scramblases and Tubby-like proteins belong to a new superfamily of membrane tethered transcription factors. Bioinformatics (Oxford, England). 25: 159-62. PMID 19010806 DOI: 10.1093/Bioinformatics/Btn595 |
0.464 |
|
2005 |
Zhou Q, Ben-Efraim I, Bigcas JL, Junqueira D, Wiedmer T, Sims PJ. Phospholipid scramblase 1 binds to the promoter region of the inositol 1,4,5-triphosphate receptor type 1 gene to enhance its expression. The Journal of Biological Chemistry. 280: 35062-8. PMID 16091359 DOI: 10.1074/Jbc.M504821200 |
0.483 |
|
2004 |
Wiedmer T, Zhao J, Li L, Zhou Q, Hevener A, Olefsky JM, Curtiss LK, Sims PJ. Adiposity, dyslipidemia, and insulin resistance in mice with targeted deletion of phospholipid scramblase 3 (PLSCR3). Proceedings of the National Academy of Sciences of the United States of America. 101: 13296-301. PMID 15328404 DOI: 10.1073/Pnas.0405354101 |
0.368 |
|
2004 |
Dong B, Zhou Q, Zhao J, Zhou A, Harty RN, Bose S, Banerjee A, Slee R, Guenther J, Williams BR, Wiedmer T, Sims PJ, Silverman RH. Phospholipid scramblase 1 potentiates the antiviral activity of interferon. Journal of Virology. 78: 8983-93. PMID 15308695 DOI: 10.1128/Jvi.78.17.8983-8993.2004 |
0.406 |
|
2004 |
Zhao KW, Li X, Zhao Q, Huang Y, Li D, Peng ZG, Shen WZ, Zhao J, Zhou Q, Chen Z, Sims PJ, Wiedmer T, Chen GQ. Protein kinase Cdelta mediates retinoic acid and phorbol myristate acetate-induced phospholipid scramblase 1 gene expression: its role in leukemic cell differentiation. Blood. 104: 3731-8. PMID 15308560 DOI: 10.1182/Blood-2004-04-1630 |
0.394 |
|
2004 |
Ben-Efraim I, Zhou Q, Wiedmer T, Gerace L, Sims PJ. Phospholipid scramblase 1 is imported into the nucleus by a receptor-mediated pathway and interacts with DNA. Biochemistry. 43: 3518-26. PMID 15035622 DOI: 10.1021/Bi0356911 |
0.414 |
|
2003 |
Nanjundan M, Sun J, Zhao J, Zhou Q, Sims PJ, Wiedmer T. Plasma membrane phospholipid scramblase 1 promotes EGF-dependent activation of c-Src through the epidermal growth factor receptor. The Journal of Biological Chemistry. 278: 37413-8. PMID 12871937 DOI: 10.1074/Jbc.M306182200 |
0.486 |
|
2003 |
Vries KJd, Wiedmer T, Sims PJ, Gadella BM. Caspase-Independent Exposure of Aminophospholipids and Tyrosine Phosphorylation in Bicarbonate Responsive Human Sperm Cells Biology of Reproduction. 68: 2122-2134. PMID 12606386 DOI: 10.1095/Biolreprod.102.012500 |
0.451 |
|
2003 |
Wiedmer T, Zhao J, Nanjundan M, Sims PJ. Palmitoylation of phospholipid scramblase 1 controls its distribution between nucleus and plasma membrane. Biochemistry. 42: 1227-33. PMID 12564925 DOI: 10.1021/Bi026679W |
0.529 |
|
2002 |
Zhou Q, Zhao J, Wiedmer T, Sims PJ. Normal hemostasis but defective hematopoietic response to growth factors in mice deficient in phospholipid scramblase 1. Blood. 99: 4030-8. PMID 12010804 DOI: 10.1182/Blood-2001-12-0271 |
0.419 |
|
2002 |
Sun J, Nanjundan M, Pike LJ, Wiedmer T, Sims PJ. Plasma membrane phospholipid scramblase 1 is enriched in lipid rafts and interacts with the epidermal growth factor receptor. Biochemistry. 41: 6338-45. PMID 12009895 DOI: 10.1021/Bi025610L |
0.421 |
|
2002 |
Silverman RH, Halloum A, Zhou A, Dong B, Al-Zoghaibi F, Kushner D, Zhou Q, Zhao J, Wiedmer T, Sims PJ. Suppression of ovarian carcinoma cell growth in vivo by the interferon-inducible plasma membrane protein, phospholipid scramblase 1. Cancer Research. 62: 397-402. PMID 11809687 |
0.35 |
|
2001 |
Sun J, Zhao J, Schwartz MA, Wang JY, Wiedmer T, Sims PJ. c-Abl tyrosine kinase binds and phosphorylates phospholipid scramblase 1. The Journal of Biological Chemistry. 276: 28984-90. PMID 11390389 DOI: 10.1074/Jbc.M102505200 |
0.407 |
|
2001 |
Sims PJ, Wiedmer T. Unraveling the mysteries of phospholipid scrambling. Thrombosis and Haemostasis. 86: 266-275. DOI: 10.1055/S-0037-1616224 |
0.531 |
|
2000 |
Wiedmer T, Zhou Q, Kwoh DY, Sims PJ. Identification of three new members of the phospholipid scramblase gene family. Biochimica Et Biophysica Acta. 1467: 244-253. PMID 10930526 DOI: 10.1016/S0005-2736(00)00236-4 |
0.401 |
|
2000 |
Tepper AD, Ruurs P, Wiedmer T, Sims PJ, Borst J, van Blitterswijk WJ. Sphingomyelin hydrolysis to ceramide during the execution phase of apoptosis results from phospholipid scrambling and alters cell-surface morphology. The Journal of Cell Biology. 150: 155-64. PMID 10893264 DOI: 10.1083/Jcb.150.1.155 |
0.468 |
|
2000 |
Zhou Q, Zhao J, Al-Zoghaibi F, Zhou A, Wiedmer T, Silverman RH, Sims PJ. Transcriptional control of the human plasma membranephospholipid scramblase 1 gene is mediated by interferon-α Blood. 95: 2593-2599. DOI: 10.1182/Blood.V95.8.2593.008K32_2593_2599 |
0.415 |
|
1999 |
Fadeel B, Gleiss B, Högstrand K, Chandra J, Wiedmer T, Sims PJ, Henter JI, Orrenius S, Samali A. Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expression. Biochemical and Biophysical Research Communications. 266: 504-511. PMID 10600532 DOI: 10.1006/Bbrc.1999.1820 |
0.403 |
|
1998 |
Stout JG, Zhou Q, Wiedmer T, Sims PJ. Change in conformation of plasma membrane phospholipid scramblase induced by occupancy of its Ca2+ binding site. Biochemistry. 37: 14860-6. PMID 9778361 DOI: 10.1021/Bi9812930 |
0.41 |
|
1998 |
Zhao J, Zhou Q, Wiedmer T, Sims PJ. Palmitoylation of phospholipid scramblase is required for normal function in promoting Ca2+-activated transbilayer movement of membrane phospholipids. Biochemistry. 37: 6361-6366. PMID 9572851 DOI: 10.1021/Bi980218M |
0.476 |
|
1998 |
Zhao J, Zhou Q, Wiedmer T, Sims PJ. Level of Expression of Phospholipid Scramblase Regulates Induced Movement of Phosphatidylserine to the Cell Surface Journal of Biological Chemistry. 273: 6603-6606. PMID 9506954 DOI: 10.1074/Jbc.273.12.6603 |
0.51 |
|
1998 |
Zhou Q, Sims PJ, Wiedmer T. Identity Of A Conserved Motif In Phospholipid Scramblase That Is Required For Ca2+-Accelerated Transbilayer Movement Of Membrane Phospholipids Biochemistry. 37: 2356-2360. PMID 9485382 DOI: 10.1021/Bi972625O |
0.47 |
|
1998 |
Zhou Q, Sims PJ, Wiedmer T. Expression of Proteins Controlling Transbilayer Movement of Plasma Membrane Phospholipids in the B Lymphocytes From a Patient With Scott Syndrome Blood. 92: 1707-1712. DOI: 10.1182/Blood.V92.5.1707.417K15_1707_1712 |
0.523 |
|
1997 |
Zhou Q, Zhao J, Stout JG, Luhm RA, Wiedmer T, Sims PJ. Molecular cloning of human plasma membrane phospholipid scramblase. A protein mediating transbilayer movement of plasma membrane phospholipids. The Journal of Biological Chemistry. 272: 18240-4. PMID 9218461 DOI: 10.1074/Jbc.272.29.18240 |
0.555 |
|
1997 |
Zhao J, Sims PJ, Wiedmer T. Production and characterization of a mutant cell line defective in aminophospholipid translocase. Biochimica Et Biophysica Acta. 1357: 57-64. PMID 9202175 DOI: 10.1016/S0167-4889(97)00014-1 |
0.426 |
|
1997 |
Stout JG, Bassé F, Luhm RA, Weiss HJ, Wiedmer T, Sims PJ. Scott syndrome erythrocytes contain a membrane protein capable of mediating Ca2+-dependent transbilayer migration of membrane phospholipids. The Journal of Clinical Investigation. 99: 2232-8. PMID 9151796 DOI: 10.1172/Jci119397 |
0.499 |
|
1996 |
Bassé F, Stout JG, Sims PJ, Wiedmer T. Isolation of an erythrocyte membrane protein that mediates Ca2+-dependent transbilayer movement of phospholipid. The Journal of Biological Chemistry. 271: 17205-10. PMID 8663431 DOI: 10.1074/Jbc.271.29.17205 |
0.524 |
|
1995 |
Sims PJ, Wiedmer T. Induction of cellular procoagulant activity by the membrane attack complex of complement Seminars in Cell Biology. 6: 275-282. PMID 8562920 DOI: 10.1006/Scel.1995.0037 |
0.448 |
|
1995 |
Bevers EM, Wiedmer T, Comfurius P, Zhao J, Smeets EF, Schlegel RA, Schroit AJ, Weiss HJ, Williamson P, Zwaal RFA, Sims PJ. The complex of phosphatidylinositol 4,5-bisphosphate and calcium ions is not responsible for Ca2+-induced loss of phospholipid asymmetry in the human erythrocyte: a study in Scott syndrome, a disorder of calcium-induced phospholipid scrambling. Blood. 86: 1983-1991. DOI: 10.1182/Blood.V86.5.1983.Bloodjournal8651983 |
0.425 |
|
1994 |
Kojima H, Newton-Nash D, Weiss HJ, Zhao J, Sims PJ, Wiedmer T. Production and characterization of transformed B-lymphocytes expressing the membrane defect of Scott syndrome. Journal of Clinical Investigation. 94: 2237-2244. PMID 7989579 DOI: 10.1172/Jci117586 |
0.49 |
|
1993 |
Wiedmer T, Hall S, Ortel T, Kane W, Rosse W, Sims P. Complement-induced vesiculation and exposure of membrane prothrombinase sites in platelets of paroxysmal nocturnal hemoglobinuria. Blood. 82: 1192-1196. DOI: 10.1182/Blood.V82.4.1192.Bloodjournal8241192 |
0.486 |
|
1992 |
Braga LL, Ninomiya H, McCoy JJ, Eacker S, Wiedmer T, Pham C, Wood S, Sims PJ, Petri WA. Inhibition of the complement membrane attack complex by the galactose-specific adhesion of Entamoeba histolytica. The Journal of Clinical Investigation. 90: 1131-7. PMID 1381719 DOI: 10.1172/Jci115931 |
0.419 |
|
1992 |
Bevers EM, Wiedmer T, Comfurius P, Shattil SJ, Weiss HJ, Zwaal RFA, Sims PJ. Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome Blood. 79: 380-388. DOI: 10.1182/Blood.V79.2.380.Bloodjournal792380 |
0.459 |
|
1991 |
Sims PJ, Wiedmer T. The response of human platelets to activated components of the complement system. Immunology Today. 12: 338-342. PMID 1755945 DOI: 10.1016/0167-5699(91)90012-I |
0.33 |
|
1991 |
Wiedmer T, Sims P. Participation of protein kinases in complement C5b-9-induced shedding of platelet plasma membrane vesicles. Blood. 78: 2880-2886. DOI: 10.1182/Blood.V78.11.2880.2880 |
0.511 |
|
1991 |
Gerrard J, Lint D, Sims P, Wiedmer T, Fugate R, McMillan E, Robertson C, Israels S. Identification of a platelet dense granule membrane protein that is deficient in a patient with the Hermansky-Pudlak syndrome. Blood. 77: 101-112. DOI: 10.1182/Blood.V77.1.101.Bloodjournal771101 |
0.466 |
|
1990 |
Wiedmer T, Shattil SJ, Cunningham M, Sims PJ. Role of calcium and calpain in complement-induced vesiculation of the platelet plasma membrane and in the exposure of the platelet factor Va receptor. Biochemistry. 29: 623-632. PMID 2159784 DOI: 10.1021/Bi00455A005 |
0.476 |
|
1989 |
Ando B, Wiedmer T, Sims P. The secretory release reaction initiated by complement proteins C5b-9 occurs without platelet aggregation through glycoprotein IIb-IIIa. Blood. 73: 462-467. DOI: 10.1182/Blood.V73.2.462.Bloodjournal732462 |
0.388 |
|
1986 |
Benz R, Schmid A, Wiedmer T, Sims PJ. Single-channel analysis of the conductance fluctuations induced in lipid bilayer membranes by complement proteins C5b-9. The Journal of Membrane Biology. 94: 37-45. PMID 2433454 DOI: 10.1007/Bf01901011 |
0.384 |
|
1986 |
Wiedmer T, Esmon C, Sims P. Complement proteins C5b-9 stimulate procoagulant activity through platelet prothrombinase. Blood. 68: 875-880. DOI: 10.1182/Blood.V68.4.875.875 |
0.389 |
|
1986 |
Sims P, Wiedmer T. Repolarization of the membrane potential of blood platelets after complement damage: evidence for a Ca++ -dependent exocytotic elimination of C5b-9 pores Blood. 68: 556-561. DOI: 10.1182/Blood.V68.2.556.Bloodjournal682556 |
0.517 |
|
1985 |
Wiedmer T, Sims PJ. Cyanine dye fluorescence used to measure membrane potential changes due to the assembly of complement proteins C5b-9. The Journal of Membrane Biology. 84: 249-258. PMID 4032456 DOI: 10.1007/Bf01871388 |
0.465 |
|
1985 |
Parker CJ, Wiedmer T, Sims PJ, Rosse WF. Characterization of the complement sensitivity of paroxysmal nocturnal hemoglobinuria erythrocytes. Journal of Clinical Investigation. 75: 2074-2084. PMID 4008653 DOI: 10.1172/Jci111927 |
0.382 |
|
1984 |
Sims PJ, Wiedmer T. The influence of electrochemical gradients of Na+ and K+ upon the membrane binding and pore forming activity of the terminal complement proteins. The Journal of Membrane Biology. 78: 169-176. PMID 6716452 DOI: 10.1007/Bf01869204 |
0.418 |
|
1984 |
Sims PJ, Wiedmer T. Kinetics of polymerization of a fluoresceinated derivative of complement protein C9 by the membrane-bound complex of complement proteins C5b-8. Biochemistry. 23: 3260-3267. PMID 6432040 DOI: 10.1021/Bi00309A021 |
0.444 |
|
1984 |
Pink DA, Chapman D, Laidlaw DJ, Wiedmer T. Electron spin resonance and steady-state fluorescence polarization studies of lipid bilayers containing integral proteins Biochemistry. 23: 4051-4058. PMID 6091736 DOI: 10.1021/Bi00313A007 |
0.36 |
|
1979 |
Wiedmer T, Francesco CD, Brodbeck U. Effects of Amphiphiles on Structure and Activity of Human Erythrocyte Membrane Acetylcholinesterase Febs Journal. 102: 59-64. PMID 520324 DOI: 10.1111/J.1432-1033.1979.Tb06262.X |
0.419 |
|
1978 |
Wiedmer T, Brodbeck U, Zahler P, Fulpius BW. Interactions of acetylcholine receptor and acetylcholinesterase with lipid monolayers Biochimica Et Biophysica Acta. 506: 161-172. PMID 620025 DOI: 10.1016/0005-2736(78)90387-5 |
0.324 |
|
Show low-probability matches. |