Year |
Citation |
Score |
2023 |
de Zélicourt A, Fayssoil A, Mansart A, Zarrouki F, Karoui A, Piquereau J, Lefebvre F, Gerbaud P, Mika D, Dakouane-Giudicelli M, Lanchec E, Feng M, Leblais V, Bobe R, Launay JM, ... ... Cancela JM, et al. Two-pore channels (TPCs) acts as a hub for excitation-contraction coupling, metabolism and cardiac hypertrophy signalling. Cell Calcium. 117: 102839. PMID 38134531 DOI: 10.1016/j.ceca.2023.102839 |
0.344 |
|
2023 |
Martucci LL, Launay JM, Kawakami N, Sicard C, Desvignes N, Dakouane-Giudicelli M, Spix B, Têtu M, Gilmaire FO, Paulcan S, Callebert J, Vaillend C, Bracher F, Grimm C, Fossier P, ... ... Cancela JM, et al. Endolysosomal TPCs regulate social behavior by controlling oxytocin secretion. Proceedings of the National Academy of Sciences of the United States of America. 120: e2213682120. PMID 36745816 DOI: 10.1073/pnas.2213682120 |
0.495 |
|
2022 |
Martucci LL, Cancela JM. Neurophysiological functions and pharmacological tools of acidic and non-acidic Ca stores. Cell Calcium. 104: 102582. PMID 35462080 DOI: 10.1016/j.ceca.2022.102582 |
0.647 |
|
2022 |
de Zélicourt A, Fayssoil A, Dakouane-Giudicelli M, De Jesus I, Karoui A, Zarrouki F, Lefebvre F, Mansart A, Launay JM, Piquereau J, Tarragó MG, Bonay M, Forand A, Moog S, Piétri-Rouxel F, ... ... Cancela JM, et al. CD38-NADase is a new major contributor to Duchenne muscular dystrophic phenotype. Embo Molecular Medicine. e12860. PMID 35298089 DOI: 10.15252/emmm.202012860 |
0.417 |
|
2019 |
Martucci LL, Amar M, Chaussenot R, Benet G, Bauer O, de Zélicourt A, Nosjean A, Launay JM, Callebert J, Sebrié C, Galione A, Edeline JM, de la Porte S, Fossier P, Granon S, ... ... Cancela JM, et al. A multiscale analysis in CD38 mice unveils major prefrontal cortex dysfunctions. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201800489R. PMID 30844310 DOI: 10.1096/Fj.201800489R |
0.36 |
|
2010 |
Cosker F, Cheviron N, Yamasaki M, Menteyne A, Lund FE, Moutin MJ, Galione A, Cancela JM. The ecto-enzyme CD38 is a nicotinic acid adenine dinucleotide phosphate (NAADP) synthase that couples receptor activation to Ca2+ mobilization from lysosomes in pancreatic acinar cells. The Journal of Biological Chemistry. 285: 38251-9. PMID 20870729 DOI: 10.1074/Jbc.M110.125864 |
0.642 |
|
2008 |
Bezin S, Charpentier G, Lee HC, Baux G, Fossier P, Cancela JM. Regulation of nuclear Ca2+ signaling by translocation of the Ca2+ messenger synthesizing enzyme ADP-ribosyl cyclase during neuronal depolarization. The Journal of Biological Chemistry. 283: 27859-70. PMID 18632662 DOI: 10.1074/Jbc.M804701200 |
0.791 |
|
2008 |
Bezin S, Fossier P, Cancela JM. Nucleoplasmic reticulum is not essential in nuclear calcium signalling mediated by cyclic ADPribose in primary neurons Pflugers Archiv European Journal of Physiology. 456: 581-586. PMID 18197416 DOI: 10.1007/S00424-007-0435-8 |
0.763 |
|
2006 |
Menteyne A, Burdakov A, Charpentier G, Petersen OH, Cancela JM. Generation of specific Ca(2+) signals from Ca(2+) stores and endocytosis by differential coupling to messengers. Current Biology : Cb. 16: 1931-7. PMID 17027490 DOI: 10.1016/J.Cub.2006.07.070 |
0.787 |
|
2006 |
Jacquard C, Trioulier Y, Cosker F, Escartin C, Bizat N, Hantraye P, Cancela JM, Bonvento G, Brouillet E. Brain mitochondrial defects amplify intracellular [Ca2+] rise and neurodegeneration but not Ca2+ entry during NMDA receptor activation. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 20: 1021-3. PMID 16571773 DOI: 10.1096/Fj.05-5085Fje |
0.762 |
|
2006 |
Bezin S, Charpentier G, Fossier P, Cancela JM. The Ca2+-releasing messenger NAADP, a new player in the nervous system. Journal of Physiology, Paris. 99: 111-8. PMID 16458493 DOI: 10.1016/J.Jphysparis.2005.12.007 |
0.791 |
|
2005 |
Yamasaki M, Thomas JM, Churchill GC, Garnham C, Lewis AM, Cancela JM, Patel S, Galione A. Role of NAADP and cADPR in the induction and maintenance of agonist-evoked Ca2+ spiking in mouse pancreatic acinar cells. Current Biology : Cb. 15: 874-8. PMID 15886108 DOI: 10.1016/J.Cub.2005.04.033 |
0.768 |
|
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