| Year |
Citation |
Score |
| 2022 |
Grimaud B, Frétaud M, Terras F, Bénassy A, Duroure K, Bercier V, Trippé-Allard G, Mohammedi R, Gacoin T, Del Bene F, Marquier F, Langevin C, Treussart F. Fast Nonlinear Microscopy Reveals Impairment of Fast Axonal Transport Induced by Molecular Motor Imbalances in the Brain of Zebrafish Larvae. Acs Nano. PMID 36459488 DOI: 10.1021/acsnano.2c06799 |
0.724 |
|
| 2022 |
Baron DM, Fenton AR, Saez-Atienzar S, Giampetruzzi A, Sreeram A, Keagle PJ, Doocy VR, Smith NJ, Danielson EW, Andresano M, McCormack MC, Garcia J, Bercier V, Van Den Bosch L, Brent JR, et al. ALS-associated KIF5A mutations abolish autoinhibition resulting in a toxic gain of function. Cell Reports. 39: 110598. PMID 35385738 DOI: 10.1016/j.celrep.2022.110598 |
0.401 |
|
| 2019 |
Atkins M, Gasmi L, Bercier V, Revenu C, Del Bene F, Hazan J, Fassier C. FIGNL1 associates with KIF1Bβ and BICD1 to restrict dynein transport velocity during axon navigation. The Journal of Cell Biology. 218: 3290-3306. PMID 31541015 DOI: 10.1083/Jcb.201805128 |
0.724 |
|
| 2019 |
Bercier V, Hubbard JM, Fidelin K, Duroure K, Auer TO, Revenu C, Wyart C, Del Bene F. Dynactin1 depletion leads to neuromuscular synapse instability and functional abnormalities. Molecular Neurodegeneration. 14: 27. PMID 31291987 DOI: 10.1186/S13024-019-0327-3 |
0.663 |
|
| 2019 |
Bercier V, Rosello M, Del Bene F, Revenu C. Zebrafish as a Model for the Study of Live Processive Transport in Neurons. Frontiers in Cell and Developmental Biology. 7: 17. PMID 30838208 DOI: 10.3389/Fcell.2019.00017 |
0.61 |
|
| 2018 |
Alberio L, Locarno A, Saponaro A, Romano E, Bercier V, Albadri S, Simeoni F, Moleri S, Pelucchi S, Porro A, Marcello E, Barsotti N, Kukovetz K, Boender AJ, Contestabile A, et al. A light-gated potassium channel for sustained neuronal inhibition. Nature Methods. PMID 30377377 DOI: 10.1038/S41592-018-0186-9 |
0.69 |
|
| 2015 |
Auer TO, Xiao T, Bercier V, Gebhardt C, Duroure K, Concordet JP, Wyart C, Suster M, Kawakami K, Wittbrodt J, Baier H, Del Bene F. Deletion of a kinesin I motor unmasks a mechanism of homeostatic branching control by neurotrophin-3. Elife. 4. PMID 26076409 DOI: 10.7554/Elife.05061 |
0.718 |
|
| 2015 |
Auer TO, Xiao T, Bercier V, Gebhardt C, Duroure K, Concordet J, Wyart C, Suster M, Kawakami K, Wittbrodt J, Baier H, Bene FD. Author response: Deletion of a kinesin I motor unmasks a mechanism of homeostatic branching control by neurotrophin-3 Elife. DOI: 10.7554/Elife.05061.030 |
0.674 |
|
| 2013 |
Kabashi E, El Oussini H, Bercier V, Gros-Louis F, Valdmanis PN, McDearmid J, Mejier IA, Dion PA, Dupre N, Hollinger D, Sinniger J, Dirrig-Grosch S, Camu W, Meininger V, Loeffler JP, et al. Investigating the contribution of VAPB/ALS8 loss of function in amyotrophic lateral sclerosis. Human Molecular Genetics. 22: 2350-60. PMID 23446633 DOI: 10.1093/Hmg/Ddt080 |
0.362 |
|
| 2010 |
Kabashi E, Lin L, Tradewell ML, Dion PA, Bercier V, Bourgouin P, Rochefort D, Bel Hadj S, Durham HD, Vande Velde C, Rouleau GA, Drapeau P. Gain and loss of function of ALS-related mutations of TARDBP (TDP-43) cause motor deficits in vivo. Human Molecular Genetics. 19: 671-83. PMID 19959528 DOI: 10.1093/Hmg/Ddp534 |
0.404 |
|
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