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Smrithi Prem - Publications

Affiliations: 
Neuroscience Rutgers-Robert Wood Johnson Medical School 
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6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2022 Connacher R, Williams M, Prem S, Yeung PL, Matteson P, Mehta M, Markov A, Peng C, Zhou X, McDermott CR, Pang ZP, Flax J, Brzustowicz L, Lu CW, Millonig JH, et al. Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17: 1786. PMID 35830838 DOI: 10.1016/j.stemcr.2022.06.007  0.629
2022 Connacher R, Williams M, Prem S, Yeung PL, Matteson P, Mehta M, Markov A, Peng C, Zhou X, McDermott CR, Pang ZP, Flax J, Brzustowicz L, Lu CW, Millonig JH, et al. Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. PMID 35623351 DOI: 10.1016/j.stemcr.2022.04.019  0.649
2020 Prem S, Millonig JH, DiCicco-Bloom E. Dysregulation of Neurite Outgrowth and Cell Migration in Autism and Other Neurodevelopmental Disorders. Advances in Neurobiology. 25: 109-153. PMID 32578146 DOI: 10.1007/978-3-030-45493-7_5  0.74
2020 Turkalj L, Mehta M, Matteson P, Prem S, Williams M, Connacher RJ, DiCicco-Bloom E, Millonig JH. Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in Neurobiology. 25: 79-107. PMID 32578145 DOI: 10.1007/978-3-030-45493-7_4  0.623
2018 Williams M, Prem S, Zhou X, Matteson P, Yeung PL, Lu CW, Pang Z, Brzustowicz L, Millonig JH, Dicicco-Bloom E. Rapid Detection of Neurodevelopmental Phenotypes in Human Neural Precursor Cells (NPCs). Journal of Visualized Experiments : Jove. PMID 29553565 DOI: 10.3791/56628  0.73
2015 Genestine M, Lin L, Durens M, Yan Y, Jiang Y, Prem S, Bailoor K, Kelly B, Sonsalla PK, Matteson PG, Silverman J, Crawley JN, Millonig JH, DiCicco-Bloom E. Engrailed-2 (En2) deletion produces multiple neurodevelopmental defects in monoamine systems, forebrain structures and neurogenesis and behavior. Human Molecular Genetics. PMID 26220976 DOI: 10.1093/Hmg/Ddv301  0.742
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