W. Wade Kothmann - Publications
Affiliations: | Synaptic Physiology Unit | National Institute of Neurological Disorders and Stroke, Bethesda, MD, United States |
Area:
retina, gap junctionsYear | Citation | Score | |||
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2023 | Lee YH, Kothmann WW, Lin YP, Chuang AZ, Diamond JS, O'Brien J. Sources of calcium at Connexin 36 gap junctions in the retina. Eneuro. PMID 37527925 DOI: 10.1523/ENEURO.0493-22.2023 | 0.574 | |||
2012 | Kothmann WW, Trexler EB, Whitaker CM, Li W, Massey SC, O'Brien J. Nonsynaptic NMDA receptors mediate activity-dependent plasticity of gap junctional coupling in the AII amacrine cell network. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 6747-59. PMID 22593045 DOI: 10.1523/Jneurosci.5087-11.2012 | 0.558 | |||
2011 | Oesch NW, Kothmann WW, Diamond JS. Illuminating synapses and circuitry in the retina. Current Opinion in Neurobiology. 21: 238-44. PMID 21349699 DOI: 10.1016/j.conb.2011.01.008 | 0.388 | |||
2009 | Kothmann WW, Massey SC, O'Brien J. Dopamine-stimulated dephosphorylation of connexin 36 mediates AII amacrine cell uncoupling. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 14903-11. PMID 19940186 DOI: 10.1523/JNEUROSCI.3436-09.2009 | 0.516 | |||
2007 | Kothmann WW, Li X, Burr GS, O'Brien J. Connexin 35/36 is phosphorylated at regulatory sites in the retina. Visual Neuroscience. 24: 363-75. PMID 17640446 DOI: 10.1017/S095252380707037X | 0.543 | |||
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