Ting Miao - Publications

Affiliations: 
2018- Iowa State University, Ames, IA, United States 

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Miao T, Bai H. Identification of Acetylation Sites of Fatty Acid Synthase (FASN) by Mass Spectrometry and FASN Activity Assay. Bio-Protocol. 13: e4873. PMID 37969759 DOI: 10.21769/BioProtoc.4873  0.39
2023 Kang P, Liu P, Kim J, Bolton M, Kumar A, Miao T, Shimell M, O'Connor MB, Powell-Coffman J, Bai H. Ptth regulates lifespan through innate immunity pathway in . Biorxiv : the Preprint Server For Biology. PMID 37873203 DOI: 10.1101/2023.09.30.560323  0.585
2022 Miao T, Kim J, Kang P, Fujiwara H, Hsu FF, Bai H. Acetyl-CoA-mediated autoacetylation of fatty acid synthase as a metabolic switch of de novo lipogenesis in . Proceedings of the National Academy of Sciences of the United States of America. 119: e2212220119. PMID 36459649 DOI: 10.1073/pnas.2212220119  0.549
2021 Wu X, Niu K, Wang X, Zhao J, Wang H, Li D, Wang H, Miao T, Yang Y, Ma H, Zhang Y, Pan L, Liu R, Bai H, Liu N. microRNA-252 and FoxO repress inflammaging by a dual inhibitory mechanism on Dawdle-mediated TGF-β pathway in Drosophila. Genetics. PMID 35100390 DOI: 10.1093/genetics/iyab234  0.505
2020 Huang K, Miao T, Chang K, Kim J, Kang P, Jiang Q, Simmonds AJ, Di Cara F, Bai H. Impaired peroxisomal import in Drosophila oenocytes causes cardiac dysfunction by inducing upd3 as a peroxikine. Nature Communications. 11: 2943. PMID 32523050 DOI: 10.1038/S41467-020-16781-W  0.558
2019 Chang K, Kang P, Liu Y, Huang K, Miao T, Sagona AP, Nezis IP, Bodmer R, Ocorr K, Bai H. TGFB-INHB/activin signaling regulates age-dependent autophagy and cardiac health through inhibition of MTORC2. Autophagy. 1-16. PMID 31884871 DOI: 10.1080/15548627.2019.1704117  0.618
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