Pablo Cisternas - Publications

Affiliations: 
Universidad Catolica de Chile 
Area:
Neurobiology

10 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2022 Cisternas P, Taylor X, Martinez P, Maldonado O, Jury N, Lasagna-Reeves CA. The reduction of astrocytic tau prevents amyloid-β-induced synaptotoxicity. Brain Communications. 4: fcac235. PMID 36196088 DOI: 10.1093/braincomms/fcac235  0.769
2022 Taylor X, Cisternas P, Jury N, Martinez P, Huang X, You Y, Redding-Ochoa J, Vidal R, Zhang J, Troncoso J, Lasagna-Reeves CA. Activated endothelial cells induce a distinct type of astrocytic reactivity. Communications Biology. 5: 282. PMID 35351973 DOI: 10.1038/s42003-022-03237-8  0.725
2020 Cisternas P, Taylor X, Perkins A, Maldonado O, Allman E, Cordova R, Marambio Y, Munoz B, Pennington T, Xiang S, Zhang J, Vidal R, Atwood B, Lasagna-Reeves CA. Vascular amyloid accumulation alters the gabaergic synapse and induces hyperactivity in a model of cerebral amyloid angiopathy. Aging Cell. e13233. PMID 32914559 DOI: 10.1111/Acel.13233  0.772
2020 Taylor X, Cisternas P, You Y, You Y, Xiang S, Marambio Y, Zhang J, Vidal R, Lasagna-Reeves CA. A1 reactive astrocytes and a loss of TREM2 are associated with an early stage of pathology in a mouse model of cerebral amyloid angiopathy. Journal of Neuroinflammation. 17: 223. PMID 32711525 DOI: 10.1186/S12974-020-01900-7  0.778
2019 Cisternas P, Taylor X, A Lasagna-Reeves C. The Amyloid-Tau-Neuroinflammation Axis in the Context of Cerebral Amyloid Angiopathy. International Journal of Molecular Sciences. 20. PMID 31847365 DOI: 10.3390/Ijms20246319  0.814
2019 You Y, Perkins A, Cisternas P, Muñoz B, Taylor X, You Y, Garringer HJ, Oblak AL, Atwood BK, Vidal R, Lasagna-Reeves CA. Tau as a mediator of neurotoxicity associated to cerebral amyloid angiopathy. Acta Neuropathologica Communications. 7: 26. PMID 30808415 DOI: 10.1186/S40478-019-0680-Z  0.778
2019 You Y, Perkins A, Cisternas P, Munoz B, Taylor X, You Y, Oblak AL, Atwood B, Vidal R, Reeves CAL. P4-131: Tau As A Mediator Of Neurotoxicity Associated To Cerebral Amyloid Angiopathy Alzheimers & Dementia. 15. DOI: 10.1016/J.Jalz.2019.06.3792  0.79
2017 Opazo MC, González PA, Flores BD, Venegas LF, Albornoz EA, Cisternas P, Bohmwald K, Nieto PA, Bueno SM, Kalergis AM, Riedel CA. Gestational Hypothyroxinemia Imprints a Switch in the Capacity of Astrocytes and Microglial Cells of the Offspring to React in Inflammation. Molecular Neurobiology. PMID 28656482 DOI: 10.1007/S12035-017-0627-Y  0.575
2015 Cisternas P, Louveau A, Bueno SM, Kalergis AM, Boudin H, Riedel CA. Gestational Hypothyroxinemia Affects Glutamatergic Synaptic Protein Distribution and Neuronal Plasticity Through Neuron-Astrocyte Interplay. Molecular Neurobiology. PMID 26687181 DOI: 10.1007/S12035-015-9609-0  0.585
2013 Albornoz EA, Carreño LJ, Cortes CM, Gonzalez PA, Cisternas PA, Cautivo KM, Catalán TP, Opazo MC, Eugenin EA, Berman JW, Bueno SM, Kalergis AM, Riedel CA. Gestational hypothyroidism increases the severity of experimental autoimmune encephalomyelitis in adult offspring. Thyroid : Official Journal of the American Thyroid Association. 23: 1627-37. PMID 23777566 DOI: 10.1089/Thy.2012.0401  0.496
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