Sila Ultanir - Publications

Affiliations: 
The Francis Crick Institute, London, England, United Kingdom 
Area:
Excitatory synapses

12 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Castano A, Silvestre M, Wells CI, Sanderson JL, Ferrer CA, Ong HW, Lang Y, Richardson W, Silvaroli JA, Bashore FM, Smith JL, Genereux IM, Dempster K, Drewry DH, Pabla NS, ... ... Ultanir S, et al. Discovery and characterization of a specific inhibitor of serine-threonine kinase cyclin dependent kinase-like 5 (CDKL5) demonstrates role in hippocampal CA1 physiology. Elife. 12. PMID 37490324 DOI: 10.7554/eLife.88206  0.398
2023 Sampedro-Castañeda M, Ultanir SK. Activity-dependent membrane sculpting deficits in TAOK1-linked neurodevelopmental disease. Trends in Neurosciences. PMID 37230852 DOI: 10.1016/j.tins.2023.05.006  0.32
2023 Castano A, Silvestre M, Wells CI, Sanderson JL, Ferrer CA, Ong HW, Liang Y, Richardson W, Silvaroli JA, Bashore FM, Smith JL, Genereux IM, Dempster K, Drewry DH, Pabla NS, ... ... Ultanir SK, et al. Discovery and characterization of a specific inhibitor of serine-threonine kinase cyclin dependent kinase-like 5 (CDKL5) demonstrates role in hippocampal CA1 physiology. Biorxiv : the Preprint Server For Biology. PMID 37162893 DOI: 10.1101/2023.04.24.538049  0.398
2023 Ong HW, Liang Y, Richardson W, Lowry ER, Wells CI, Chen X, Silvestre M, Dempster K, Silvaroli JA, Smith JL, Wichterle H, Pabla NS, Ultanir SK, Bullock AN, Drewry DH, et al. Discovery of a Potent and Selective CDKL5/GSK3 Chemical Probe That Is Neuroprotective. Acs Chemical Neuroscience. PMID 37084253 DOI: 10.1021/acschemneuro.3c00135  0.301
2022 Baskaran P, Mihaylov SR, Vinsland E, Shah K, Granat L, Ultanir SK, Tee AR, Murn J, Bateman JM. Phosphorylation of the novel mTOR substrate Unkempt regulates cellular morphogenesis. The Journal of Biological Chemistry. 102788. PMID 36509146 DOI: 10.1016/j.jbc.2022.102788  0.363
2022 Roşianu F, Mihaylov SR, Eder N, Martiniuc A, Claxton S, Flynn HR, Jalal S, Domart MC, Collinson L, Skehel M, Snijders AP, Krause M, Tooze SA, Ultanir SK. Loss of NDR1/2 kinases impairs endomembrane trafficking and autophagy leading to neurodegeneration. Life Science Alliance. 6. PMID 36446521 DOI: 10.26508/lsa.202201712  0.382
2018 Lin AW, Gill KK, Castañeda MS, Matucci I, Eder N, Claxton S, Flynn H, Snijders AP, George R, Ultanir SK. Chemical genetic identification of GAK substrates reveals its role in regulating Na/K-ATPase. Life Science Alliance. 1: e201800118. PMID 30623173 DOI: 10.26508/lsa.201800118  0.457
2018 Baltussen LL, Negraes PD, Silvestre M, Claxton S, Moeskops M, Christodoulou E, Flynn HR, Snijders AP, Muotri AR, Ultanir SK. Chemical genetic identification of CDKL5 substrates reveals its role in neuronal microtubule dynamics. The Embo Journal. PMID 30266824 DOI: 10.15252/embj.201899763  0.387
2017 Baltussen L, Rosianu F, Ultanir S. Kinases in synaptic development and neurological diseases. Progress in Neuro-Psychopharmacology & Biological Psychiatry. PMID 29241837 DOI: 10.1016/j.pnpbp.2017.12.006  0.454
2014 Ultanir SK, Yadav S, Hertz NT, Oses-Prieto JA, Claxton S, Burlingame AL, Shokat KM, Jan LY, Jan YN. MST3 kinase phosphorylates TAO1/2 to enable Myosin Va function in promoting spine synapse development. Neuron. 84: 968-82. PMID 25456499 DOI: 10.1016/J.Neuron.2014.10.025  0.576
2012 Ultanir SK, Hertz NT, Li G, Ge WP, Burlingame AL, Pleasure SJ, Shokat KM, Jan LY, Jan YN. Chemical genetic identification of NDR1/2 kinase substrates AAK1 and Rabin8 Uncovers their roles in dendrite arborization and spine development. Neuron. 73: 1127-42. PMID 22445341 DOI: 10.1016/J.Neuron.2012.01.019  0.589
2007 Ultanir SK, Kim JE, Hall BJ, Deerinck T, Ellisman M, Ghosh A. Regulation of spine morphology and spine density by NMDA receptor signaling in vivo. Proceedings of the National Academy of Sciences of the United States of America. 104: 19553-8. PMID 18048342 DOI: 10.1073/pnas.0704031104  0.507
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