Kambiz Nassirpour Alavian

Affiliations: 
Harvard Medical School, Boston, MA, United States 
Area:
Parkinson's
Google:
"Kambiz Alavian"
Mean distance: 17.7 (cluster 6)
 
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Publications

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Fang Y, Liu C, Lin J, et al. (2019) PhySpeTree: an automated pipeline for reconstructing phylogenetic species trees. Bmc Evolutionary Biology. 19: 219
Chen R, Park HA, Mnatsakanyan N, et al. (2019) Parkinson's disease protein DJ-1 regulates ATP synthase protein components to increase neuronal process outgrowth. Cell Death & Disease. 10: 469
Weinert M, Millet A, Jonas EA, et al. (2019) The mitochondrial metabolic function of is modulated by . Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201802754R
Mallach A, Weinert M, Arthur J, et al. (2019) Post mortem examination of Parkinson's disease brains suggests decline in mitochondrial biomass, reversed by deep brain stimulation of subthalamic nucleus. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201802628R
Niu Y, Moghimyfiroozabad S, Safaie S, et al. (2017) Phylogenetic Profiling of Mitochondrial Proteins and Integration Analysis of Bacterial Transcription Units Suggest Evolution of F1Fo ATP Synthase from Multiple Modules. Journal of Molecular Evolution. 85: 219-233
Niu Y, Liu C, Moghimyfiroozabad S, et al. (2017) PrePhyloPro: phylogenetic profile-based prediction of whole proteome linkages. Peerj. 5: e3712
Park HA, Licznerski P, Mnatsakanyan N, et al. (2017) Inhibition of Bcl-xL prevents pro-death actions of ΔN-Bcl-xL at the mitochondrial inner membrane during glutamate excitotoxicity. Cell Death and Differentiation
Beutner G, Alavian KN, Jonas EA, et al. (2016) Erratum to: The Mitochondrial Permeability Transition Pore and ATP Synthase. Handbook of Experimental Pharmacology
Mnatsakanyan N, Beutner G, Porter GA, et al. (2016) Physiological roles of the mitochondrial permeability transition pore. Journal of Bioenergetics and Biomembranes
Licznerski P, Duric V, Banasr M, et al. (2015) Decreased SGK1 Expression and Function Contributes to Behavioral Deficits Induced by Traumatic Stress. Plos Biology. 13: e1002282
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