Neha Shrestha
Affiliations: | 2011-2015 | NUS, Singapore, Singapore |
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"Neha Shrestha"Mean distance: 16.04 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentorWeiping Han | grad student | 2011-2015 | NUS |
Ling Qi | post-doc | 2016- | University of Michigan Medical School (Cell Biology Tree) |
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Publications
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Abdon B, Liang Y, da Luz Scheffer D, et al. (2023) Muscle specific ER-associated degradation maintains postnatal muscle hypertrophy and systemic energy metabolism. Jci Insight |
Shrestha N, Torres M, Zhang J, et al. (2022) Integration of ER protein quality control mechanisms defines β-cell function and ER architecture. The Journal of Clinical Investigation |
Shrestha N, De Franco E, Arvan P, et al. (2021) Pathological β-Cell Endoplasmic Reticulum Stress in Type 2 Diabetes: Current Evidence. Frontiers in Endocrinology. 12: 650158 |
Shrestha N, Reinert RB, Qi L. (2020) Endoplasmic Reticulum Protein Quality Control in β Cells. Seminars in Cell & Developmental Biology |
Shrestha N, Liu T, Ji Y, et al. (2020) Sel1L-Hrd1 ER-associated degradation maintains β-cell identity via TGFβ signaling. The Journal of Clinical Investigation |
Shrestha N, Reinert RB, Arvan P, et al. (2020) 49-OR: Synergistic Effect of ERAD and Autophagy in Preventing Proinsulin Aggregation and Promoting Beta-Cell Survival Diabetes. 69 |
Reinert RB, Cui X, Shrestha N, et al. (2020) 323-OR: Synergism between Protein Quality Control Pathways to Maintain Alpha-Cell Mass and Function Diabetes. 69 |
Ji Y, Sun S, Shrestha N, et al. (2019) Toll-like receptors TLR2 and TLR4 block the replication of pancreatic β cells in diet-induced obesity. Nature Immunology |
Reinert RB, Shrestha N, Ji Y, et al. (2019) 2197-P: SEL1L-HRD1 Endoplasmic Reticulum-Associated Degradation Is Required for Maintaining Glucagon Production in Pancreatic Alpha Cells Diabetes. 68 |
SHRESTHA N, LI X, JI Y, et al. (2019) 2166-P: Sel1L-Hrd1 ERAD Is a Key Regulator of Beta-Cell Function and Identity Diabetes. 68: 2166-P |