Heather A. Mitchell, Ph.D.

Emory University, Atlanta, GA
"Heather Mitchell"
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David Weinshenker grad student 2010 Emory
 (Catecholamines and Arousal.)
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Rigby MJ, Ding Y, Farrugia MA, et al. (2020) The endoplasmic reticulum acetyltransferases ATase1/NAT8B and ATase2/NAT8 are differentially regulated to adjust engagement of the secretory pathway. Journal of Neurochemistry. e14958
Porter-Stransky KA, Centanni SW, Karne SL, et al. (2018) Noradrenergic Transmission at Alpha1-Adrenergic Receptors in the Ventral Periaqueductal Gray Modulates Arousal. Biological Psychiatry
Hullinger R, Li M, Wang J, et al. (2016) Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation. The Journal of Experimental Medicine
Keebaugh AC, Mitchell HA, Gaval-Cruz M, et al. (2011) PRTFDC1 is a genetic modifier of HPRT-deficiency in the mouse. Plos One. 6: e22381
Mitchell HA, Weinshenker D. (2010) Good night and good luck: norepinephrine in sleep pharmacology. Biochemical Pharmacology. 79: 801-9
Mitchell HA, Bogenpohl JW, Liles LC, et al. (2009) Corrigendum to "Behavioral responses of dopamine β-hydroxylase knockout mice to modafinil suggest a dual noradrenergic-dopaminergic mechanism of action" (DOI:10.1016/j.pbb.2008.07.014) Pharmacology Biochemistry and Behavior. 92: 197
Mitchell HA, Bogenpohl JW, Liles LC, et al. (2008) Behavioral responses of dopamine beta-hydroxylase knockout mice to modafinil suggest a dual noradrenergic-dopaminergic mechanism of action. Pharmacology, Biochemistry, and Behavior. 91: 217-22
Mitchell HA, Ahern TH, Liles LC, et al. (2006) The effects of norepinephrine transporter inactivation on locomotor activity in mice. Biological Psychiatry. 60: 1046-52
Ahern TH, Javors MA, Eagles DA, et al. (2006) The effects of chronic norepinephrine transporter inactivation on seizure susceptibility in mice. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 31: 730-8
Wang HW, Babic AM, Mitchell HA, et al. (2005) Elevated soluble ICAM-1 levels induce immune deficiency and increase adiposity in mice. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 19: 1018-20
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