Kan Cao, Ph.D.
Institution:
Johns Hopkins University, Baltimore, MDArea:
Cell biologyGoogle:
"Kan Cao"Mean distance: 18.83 (cluster 46)
Parents
Sign in to add mentor| Yixian Zheng | grad student | 2005 | Johns Hopkins | |
| (The regulation of spindle morphogenesis and chromosome segregation during mitosis.) | ||||
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Publications
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| Sun L, Chiang JY, Choi JY, et al. (2019) Transient induction of telomerase expression mediates senescence and reduces tumorigenesis in primary fibroblasts. Proceedings of the National Academy of Sciences of the United States of America |
| Tariq Z, Zhang H, Chia-Liu A, et al. (2017) Lamin A and microtubules collaborate to maintain nuclear morphology. Nucleus (Austin, Tex.). 1-14 |
| Zhang H, Cao K. (2017) Mechanisms of genome instability in Hutchinson-Gilford progeria Frontiers of Biology in China. 12: 49-62 |
| Wu D, Yates PA, Zhang H, et al. (2016) Comparing lamin proteins post-translational relative stability using a 2A peptide-based system reveals elevated resistance of progerin to cellular degradation. Nucleus (Austin, Tex.). 0 |
| Zhang H, Sun L, Wang K, et al. (2016) Loss of H3K9me3 Correlates with ATM Activation and Histone H2AX Phosphorylation Deficiencies in Hutchinson-Gilford Progeria Syndrome. Plos One. 11: e0167454 |
| Zhang H, Xiong ZM, Cao K. (2014) Mechanisms controlling the smooth muscle cell death in progeria via down-regulation of poly(ADP-ribose) polymerase 1. Proceedings of the National Academy of Sciences of the United States of America. 111: E2261-70 |
| Wu D, Flannery AR, Cai H, et al. (2014) Nuclear localization signal deletion mutants of lamin A and progerin reveal insights into lamin A processing and emerin targeting. Nucleus (Austin, Tex.). 5: 66-74 |
| Zhang H, Kieckhaefer JE, Cao K. (2013) Mouse models of laminopathies. Aging Cell. 12: 2-10 |
| Cao K, Graziotto JJ, Blair CD, et al. (2011) Rapamycin reverses cellular phenotypes and enhances mutant protein clearance in Hutchinson-Gilford progeria syndrome cells. Science Translational Medicine. 3: 89ra58 |
| Cao K, Blair CD, Faddah DA, et al. (2011) Progerin and telomere dysfunction collaborate to trigger cellular senescence in normal human fibroblasts. The Journal of Clinical Investigation. 121: 2833-44 |