Davide Tampellini
Affiliations: | Inserm - Université Paris-Saclay |
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"Davide Tampellini"Mean distance: 15.66 (cluster 28) | S | N | B | C | P |
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Publications
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Cascella R, Banchelli M, Abolghasem Ghadami S, et al. (2022) An and investigation of cytoplasmic TDP-43 inclusions reveals the absence of a clear amyloid signature. Annals of Medicine. 55: 72-88 |
Akwa Y, Di Malta C, Zallo F, et al. (2022) Stimulation of synaptic activity promotes TFEB-mediated clearance of pathological MAPT/Tau in cellular and mouse models of tauopathies. Autophagy. 1-18 |
Mann A, Gondard E, Tampellini D, et al. (2017) Chronic deep brain stimulation in an Alzheimer's disease mouse model enhances memory and reduces pathological hallmarks. Brain Stimulation |
Tampellini D. (2015) Synaptic activity and Alzheimer's disease: a critical update. Frontiers in Neuroscience. 9: 423 |
Stack C, Jainuddin S, Elipenahli C, et al. (2014) Methylene blue upregulates Nrf2/ARE genes and prevents tau-related neurotoxicity. Human Molecular Genetics. 23: 3716-32 |
Dumont M, Stack C, Elipenahli C, et al. (2014) PGC-1α overexpression exacerbates β-amyloid and tau deposition in a transgenic mouse model of Alzheimer's disease. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 1745-55 |
Dumont M, Stack C, Elipenahli C, et al. (2012) Bezafibrate administration improves behavioral deficits and tau pathology in P301S mice. Human Molecular Genetics. 21: 5091-105 |
Gouras GK, Willén K, Tampellini D. (2012) Critical role of intraneuronal Aβ in Alzheimer's disease: technical challenges in studying intracellular Aβ. Life Sciences. 91: 1153-8 |
Capetillo-Zarate E, Gracia L, Tampellini D, et al. (2012) Intraneuronal Aβ accumulation, amyloid plaques, and synapse pathology in Alzheimer's disease. Neuro-Degenerative Diseases. 10: 56-9 |
Tampellini D, Rahman N, Lin MT, et al. (2011) Impaired β-amyloid secretion in Alzheimer's disease pathogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 15384-90 |