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Ann T. Poblenz, Ph.D.

Affiliations: 
University of Houston, Houston, TX, United States 
Area:
retina, development, apoptosis, mitochondria, chemicals, drugs
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"Ann Poblenz"
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Donald A. Fox grad student 2000 University of Houston
 (Mechanisms underlying lead- and calcium -induced rod photoreceptor-selective apoptosis: In vivo and in vitro studies.)

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Publications

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Fox DA, Poblenz AT, He L, et al. (2003) Pharmacological strategies to block rod photoreceptor apoptosis caused by calcium overload: A mechanistic target-site approach to neuroprotection. European Journal of Ophthalmology. 13: 44-56
Fox DA, Poblenz AT, He L, et al. (2003) Pharmacological strategies to block rod photoreceptor apoptosis caused by calcium overload: a mechanistic target-site approach to neuroprotection. European Journal of Ophthalmology. 13: S44-56
He L, Perkins GA, Poblenz AT, et al. (2003) Bcl-xL overexpression blocks bax-mediated mitochondrial contact site formation and apoptosis in rod photoreceptors of lead-exposed mice. Proceedings of the National Academy of Sciences of the United States of America. 100: 1022-7
He L, Poblenz AT, Medrano CJ, et al. (2000) Lead and calcium produce rod photoreceptor cell apoptosis by opening the mitochondrial permeability transition pore. The Journal of Biological Chemistry. 275: 12175-84
Fox DA, Poblenz AT, He L. (1999) Calcium overload triggers rod photoreceptor apoptotic cell death in chemical-induced and inherited retinal degenerations. Annals of the New York Academy of Sciences. 893: 282-5
Fox DA, Srivastava D, Poblenz A, et al. (1998) Lead-induced Alterations in Gene Expression and Activity of Retinal cGMP PDE Results in Calcium Overload and Rod-selective Apoptosis. Toxicology in Vitro : An International Journal Published in Association With Bibra. 12: 597-8
Fox DA, He L, Poblenz AT, et al. (1998) Lead-induced alterations in retinal cGMP phosphodiesterase trigger calcium overload, mitochondrial dysfunction and rod photoreceptor apoptosis. Toxicology Letters. 102: 359-61
Fox D, Srivastava D, Poblenz A, et al. (1998) Lead-induced alterations in retinal cGMP PDE gene expression and activity result in cell-specific apoptosis during development Toxicology Letters. 95: 18
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