Samuel S. Pappas, Ph.D.

Affiliations: 
Physiology Michigan State University, East Lansing, MI 
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Keith Lookingland grad student 2011 Michigan State
 (Sex differences in the anatomical distribution, regulation, and anti-nociceptive function of the A11 diencephalospinal dopamine neuronal system.)
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Morrison LM, Huang H, Handler HP, et al. (2023) Increased intrinsic membrane excitability is associated with hypertrophic olivary degeneration in spinocerebellar ataxia type 1. Biorxiv : the Preprint Server For Biology
Yellajoshyula D, Opeyemi S, Dauer WT, et al. (2022) Genetic evidence of aberrant striatal synaptic maturation and secretory pathway alteration in a dystonia mouse model. Dystonia. 1
Li J, Liang CC, Pappas SS, et al. (2020) TorsinB overexpression prevents abnormal twisting in DYT1 dystonia mouse models. Elife. 9
Pappas SS, Li J, LeWitt TM, et al. (2018) A cell autonomous torsinA requirement for cholinergic neuron survival and motor control. Elife. 7
Weisheit CE, Pappas SS, Dauer WT. (2018) Inherited dystonias: clinical features and molecular pathways. Handbook of Clinical Neurology. 147: 241-254
Pappas SS, Li J, LeWitt TM, et al. (2018) Author response: A cell autonomous torsinA requirement for cholinergic neuron survival and motor control Elife
Pappas SS, Liang CC, Kim S, et al. (2017) TorsinA dysfunction causes persistent neuronal nuclear pore defects. Human Molecular Genetics
DeSimone JC, Pappas SS, Febo M, et al. (2017) Forebrain knock-out of torsinA reduces striatal free-water and impairs whole-brain functional connectivity in a symptomatic mouse model of DYT1 dystonia. Neurobiology of Disease
Pappas SS, Darr K, Holley SM, et al. (2015) Forebrain deletion of the dystonia protein torsinA causes dystonic-like movements and loss of striatal cholinergic neurons. Elife. 4
Pappas SS, Darr K, Holley SM, et al. (2015) Author response: Forebrain deletion of the dystonia protein torsinA causes dystonic-like movements and loss of striatal cholinergic neurons Elife
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