Jennifer A. Bendiske, Ph.D.
Affiliations: | University of Connecticut, Storrs, CT, United States |
Area:
Neuroprotection in models of Alzheimer-type pathogenesisGoogle:
"Jennifer Bendiske"Mean distance: 30533.1
Parents
Sign in to add mentor| Ben A. Bahr | grad student | 2002 | University of Connecticut | |
| (Pathogenic changes and compensatory responses in a model of age-related neurodegeneration.) | ||||
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Publications
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| Kanju PM, Parameshwaran K, Vaithianathan T, et al. (2007) Lysosomal dysfunction produces distinct alterations in synaptic alpha-amino-3-hydroxy-5-methylisoxazolepropionic acid and N-methyl-D-aspartate receptor currents in hippocampus. Journal of Neuropathology and Experimental Neurology. 66: 779-88 |
| Butler D, Bendiske J, Michaelis ML, et al. (2007) Microtubule-stabilizing agent prevents protein accumulation-induced loss of synaptic markers. European Journal of Pharmacology. 562: 20-7 |
| Bendiske J, Bahr BA. (2003) Lysosomal activation is a compensatory response against protein accumulation and associated synaptopathogenesis--an approach for slowing Alzheimer disease? Journal of Neuropathology and Experimental Neurology. 62: 451-63 |
| Bahr BA, Bendiske J. (2002) The neuropathogenic contributions of lysosomal dysfunction. Journal of Neurochemistry. 83: 481-9 |
| Bendiske J, Caba E, Brown QB, et al. (2002) Intracellular deposition, microtubule destabilization, and transport failure: an "early" pathogenic cascade leading to synaptic decline. Journal of Neuropathology and Experimental Neurology. 61: 640-50 |
| Bahr BA, Bendiske J, Brown QB, et al. (2002) Survival signaling and selective neuroprotection through glutamatergic transmission. Experimental Neurology. 174: 37-47 |