Marcello D'Amelio

Affiliations: 
IRCCS Santa Lucia Foundation, Rome, Italy, Roma, Lazio, Italy 
Area:
Neurodegeneration
Google:
"Marcello D'Amelio"
Mean distance: 20.05 (cluster 32)
 
SNBCP
Cross-listing: Neuropathology Tree

BETA: Related publications

Publications

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Pignataro A, Krashia P, De Introna M, et al. (2023) Chemogenetic rectification of the inhibitory tone onto hippocampal neurons reverts autistic-like traits and normalizes local expression of estrogen receptors in the Ambra1+/- mouse model of female autism. Translational Psychiatry. 13: 63
Klionsky DJ, Abdel-Aziz AK, Abdelfatah S, et al. (2021) Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition). Autophagy. 1-382
Nobili A, Krashia P, D'Amelio M. (2020) Cisd2: A promising new target in Alzheimer's disease. The Journal of Pathology
Borreca A, Valeri F, De Luca M, et al. (2020) Transient upregulation of translational efficiency in prodromal and early symptomatic Tg2576 mice contributes to Aβ pathology. Neurobiology of Disease. 104787
Becher J, Simula L, Volpe E, et al. (2018) AMBRA1 Controls Regulatory T-Cell Differentiation and Homeostasis Upstream of the FOXO3-FOXP3 Axis. Developmental Cell. 47: 592-607.e6
Cordella A, Krashia P, Nobili A, et al. (2018) Dopamine loss alters the hippocampus-nucleus accumbens synaptic transmission in the Tg2576 mouse model of Alzheimer's disease. Neurobiology of Disease
D'Amelio M, Serra L, Bozzali M. (2018) Ventral Tegmental Area in Prodromal Alzheimer's Disease: Bridging the Gap between Mice and Humans. Journal of Alzheimer's Disease : Jad. 63: 181-183
Nobili A, Krashia P, Cordella A, et al. (2018) Ambra1 Shapes Hippocampal Inhibition/Excitation Balance: Role in Neurodevelopmental Disorders. Molecular Neurobiology
Landrock KK, Sullivan P, Martini-Stoica H, et al. (2017) Pleiotropic Neuropathological and Biochemical Alterations Associated with Myo5a Mutation in a Rat Model. Brain Research
Nobili A, Latagliata EC, Viscomi MT, et al. (2017) Dopamine neuronal loss contributes to memory and reward dysfunction in a model of Alzheimer's disease. Nature Communications. 8: 14727
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