Jacquelyn A. Brown, Ph.D.

cell biology, motility,

Paul Bridgman	grad student	2008	Washington University
(The role of growth cone myosin II and actin associated proteins in the repulsive response induced by the guidance factor semaphorin 3A.)

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Brown JA, Diggs-Andrews KA, Gianino SM, et al. (2012) Neurofibromatosis-1 heterozygosity impairs CNS neuronal morphology in a cAMP/PKA/ROCK-dependent manner. Molecular and Cellular Neurosciences. 49: 13-22
Brown JA, Emnett RJ, White CR, et al. (2010) Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice. Human Molecular Genetics. 19: 4515-28
Brown JA, Gianino SM, Gutmann DH. (2010) Defective cAMP generation underlies the sensitivity of CNS neurons to neurofibromatosis-1 heterozygosity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 5579-89
Brown JA, Bridgman PC. (2009) Disruption of the cytoskeleton during Semaphorin 3A induced growth cone collapse correlates with differences in actin organization and associated binding proteins. Developmental Neurobiology. 69: 633-46
Brown JA, Wysolmerski RB, Bridgman PC. (2009) Dorsal root ganglion neurons react to semaphorin 3A application through a biphasic response that requires multiple myosin II isoforms. Molecular Biology of the Cell. 20: 1167-79
Brown J, Bridgman PC. (2003) Role of myosin II in axon outgrowth. The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society. 51: 421-8