Ranmal A. Samarasinghe, Ph.D.

Affiliations: 
University of Pittsburgh, Pittsburgh, PA, United States 
Area:
Zinc, Potassium Channels, Apoptosis, Redox, NMDA, TOPA
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"Ranmal Samarasinghe"
Mean distance: 15.28 (cluster 11)
 
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Parents

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Elias Aizenman grad student 2011 University of Pittsburgh
 (Non-classical glucocorticoid receptor action regulates gap junction intercellular communication and neural progenitor cell proliferation.)
Bennett Novitch post-doc 2018-2021 UCLA

Children

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Daniel Toker grad student
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Publications

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Atamian A, Birtele M, Hosseini N, et al. (2024) Human cerebellar organoids with functional Purkinje cells. Cell Stem Cell. 31: 39-51.e6
Allison T, Langerman J, Sabri S, et al. (2021) Defining the nature of human pluripotent stem cell-derived interneurons via single-cell analysis. Stem Cell Reports
Samarasinghe RA, Miranda OA, Buth JE, et al. (2021) Identification of neural oscillations and epileptiform changes in human brain organoids. Nature Neuroscience
Samarasinghe RA, Kanuparthi PS, Timothy Greenamyre J, et al. (2014) Transient muscarinic and glutamatergic stimulation of neural stem cells triggers acute and persistent changes in differentiation. Neurobiology of Disease. 70: 252-61
Samarasinghe RA, Kanuparthi PS, Timothy Greenamyre J, et al. (2014) Corrigendum to "Transient muscarinic and glutamatergic stimulation of neural stem cells triggers acute and persistent changes in differentiation" [Neurobiol. Dis. 70 (2014) 252-261] Neurobiology of Disease. 71: 43
Samarasinghe RA, Di Maio R, Volonte D, et al. (2011) Nongenomic glucocorticoid receptor action regulates gap junction intercellular communication and neural progenitor cell proliferation. Proceedings of the National Academy of Sciences of the United States of America. 108: 16657-62
Bales JW, Yan HQ, Ma X, et al. (2011) The dopamine and cAMP regulated phosphoprotein, 32 kDa (DARPP-32) signaling pathway: a novel therapeutic target in traumatic brain injury. Experimental Neurology. 229: 300-7
Ho Y, Samarasinghe R, Knoch ME, et al. (2008) Selective inhibition of mitogen-activated protein kinase phosphatases by zinc accounts for extracellular signal-regulated kinase 1/2-dependent oxidative neuronal cell death. Molecular Pharmacology. 74: 1141-51
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