Hagit Eldar-Finkelman

Tel Aviv University, Tel Aviv-Yafo, Tel Aviv District, Israel 
"Hagit Eldar-Finkelman"
Mean distance: 16.88 (cluster 6)
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Eldar-Finkelman H, VanHook AM. (2016) Science Signaling Podcast for 15 November 2016: A new type of kinase inhibitor. Science Signaling. 9: pc22
Grieco SF, Velmeshev D, Magistri M, et al. (2016) Ketamine up-regulates a cluster of intronic miRNAs within the serotonin receptor 2C gene by inhibiting glycogen synthase kinase-3. The World Journal of Biological Psychiatry : the Official Journal of the World Federation of Societies of Biological Psychiatry. 1-12
Lo Monte F, Kramer T, Gu J, et al. (2012) Identification of glycogen synthase kinase-3 inhibitors with a selective sting for glycogen synthase kinase-3α. Journal of Medicinal Chemistry. 55: 4407-24
Alon LT, Pietrokovski S, Barkan S, et al. (2011) Selective loss of glycogen synthase kinase-3α in birds reveals distinct roles for GSK-3 isozymes in tau phosphorylation. Febs Letters. 585: 1158-62
Leng S, Zhang W, Zheng Y, et al. (2010) Glycogen synthase kinase 3 beta mediates high glucose-induced ubiquitination and proteasome degradation of insulin receptor substrate 1. The Journal of Endocrinology. 206: 171-81
Eldar-Finkelman H, Licht-Murava A, Pietrokovski S, et al. (2010) Substrate competitive GSK-3 inhibitors - strategy and implications. Biochimica Et Biophysica Acta. 1804: 598-603
Eldar-Finkelman H, Eisenstein M. (2009) Peptide inhibitors targeting protein kinases. Current Pharmaceutical Design. 15: 2463-70
Ilouz R, Pietrokovski S, Eisenstein M, et al. (2008) New insights into the autoinhibition mechanism of glycogen synthase kinase-3beta. Journal of Molecular Biology. 383: 999-1007
Liberman Z, Plotkin B, Tennenbaum T, et al. (2008) Coordinated phosphorylation of insulin receptor substrate-1 by glycogen synthase kinase-3 and protein kinase C betaII in the diabetic fat tissue. American Journal of Physiology. Endocrinology and Metabolism. 294: E1169-77
Spalding A, Watson R, Zielske S, et al. (2007) Glycogen Synthase Kinase 3β Inhibition and βcatenin Activation Contribute to Enhanced Survival After Radiation in Pancreatic Cancer Cells International Journal of Radiation Oncology*Biology*Physics. 69: S608
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