Haley E. Speed
Affiliations: | University of Texas Southwestern, Dallas, TX, United States |
Area:
short-term plasticityGoogle:
"Haley Speed"Mean distance: 14.12 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorLynn E. Dobrunz | grad student | 2008 | UAB | |
(Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus.) | ||||
Craig M. Powell | post-doc | 2008- | UT Southwestern | |
(Now Instructor at UT Southwestern Medical Center) |
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Publications
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Monson N, Smith C, Greenberg H, et al. (2023) VH2+ Antigen-Experienced B Cells in the Cerebrospinal Fluid Are Expanded and Enriched in Pediatric Anti-NMDA Receptor Encephalitis. Journal of Immunology (Baltimore, Md. : 1950) |
Umfress A, Speed HE, Tan C, et al. (2021) Neuropathological Effects of Chemotherapeutic Drugs. Acs Chemical Neuroscience |
Heavner WE, Lautz JD, Speed HE, et al. (2021) Remodeling of the Homer-Shank interactome mediates homeostatic plasticity. Science Signaling. 14 |
Speed HE, Kouser M, Xuan Z, et al. (2019) Apparent Genetic Rescue of Adult Exon 21 Insertion Mutation Mice Tempered by Appropriate Control Experiments. Eneuro |
Escamilla CO, Filonova I, Walker AK, et al. (2017) Kctd13 deletion reduces synaptic transmission via increased RhoA. Nature |
Jaramillo TC, Speed HE, Xuan Z, et al. (2016) Novel Shank3 mutant exhibits behaviors with face validity for autism and altered striatal and hippocampal function. Autism Research : Official Journal of the International Society For Autism Research |
Jaramillo TC, Speed HE, Xuan Z, et al. (2015) Altered Striatal Synaptic Function and Abnormal Behaviour in Shank3 Exon4-9 Deletion Mouse Model of Autism. Autism Research : Official Journal of the International Society For Autism Research |
Speed HE, Masiulis I, Gibson JR, et al. (2015) Increased Cortical Inhibition in Autism-Linked Neuroligin-3R451C Mice Is Due in Part to Loss of Endocannabinoid Signaling. Plos One. 10: e0140638 |
Speed HE, Kouser M, Xuan Z, et al. (2015) Autism-Associated Insertion Mutation (InsG) of Shank3 Exon 21 Causes Impaired Synaptic Transmission and Behavioral Deficits. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 9648-65 |
Kouser M, Speed HE, Dewey CM, et al. (2013) Loss of predominant Shank3 isoforms results in hippocampus-dependent impairments in behavior and synaptic transmission. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 18448-68 |