William T. Dauer
Affiliations: | Columbia University, New York, NY |
Area:
neurologyGoogle:
"William Dauer"Mean distance: 17.84 (cluster 19) | S | N | B | C | P |
Children
Sign in to add trainee| Cherry C. Ho | grad student | 2009 | Columbia |
| Connie E. Kim | grad student | 2009 | Columbia |
| Lauren M. Tanabe | grad student | 2010 | Columbia |
| Jamie Ann Johansen | post-doc | 2010- | Columbia, University of Michigan |
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Publications
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| Li J, Levin DS, Kim AJ, et al. (2021) TorsinA restoration in a mouse model identifies a critical therapeutic window for DYT1 dystonia. The Journal of Clinical Investigation |
| Li J, Liang CC, Pappas SS, et al. (2020) TorsinB overexpression prevents abnormal twisting in DYT1 dystonia mouse models. Elife. 9 |
| Shin JY, Hernandez-Ono A, Fedotova T, et al. (2019) Nuclear envelope-localized torsinA-LAP1 complex regulates hepatic VLDL secretion and steatosis. The Journal of Clinical Investigation. 130 |
| Bohnen NI, Kanel P, Zhou Z, et al. (2019) Cholinergic system changes of falls and freezing of gait in Parkinson disease. Annals of Neurology |
| Albin RL, Surmeier DJ, Tubert C, et al. (2018) Targeting the pedunculopontine nucleus in Parkinson's disease: Time to go back to the drawing board. Movement Disorders : Official Journal of the Movement Disorder Society |
| Pappas SS, Li J, LeWitt TM, et al. (2018) A cell autonomous torsinA requirement for cholinergic neuron survival and motor control. Elife. 7 |
| Antoniou N, Vlachakis D, Memou A, et al. (2018) A motif within the armadillo repeat of Parkinson's-linked LRRK2 interacts with FADD to hijack the extrinsic death pathway. Scientific Reports. 8: 3455 |
| Weisheit CE, Pappas SS, Dauer WT. (2018) Inherited dystonias: clinical features and molecular pathways. Handbook of Clinical Neurology. 147: 241-254 |
| Pappas SS, Li J, LeWitt TM, et al. (2018) Author response: A cell autonomous torsinA requirement for cholinergic neuron survival and motor control Elife |
| Pappas SS, Liang CC, Kim S, et al. (2017) TorsinA dysfunction causes persistent neuronal nuclear pore defects. Human Molecular Genetics |