Jeffrey L. Elliott
Affiliations: | University of Texas Southwestern Medical Center, Dallas, TX, United States |
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"Jeffrey Elliott"Mean distance: 15.83 (cluster 24) | S | N | B | C | P |
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Publications
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| Dubey D, Khemani P, Remster E, et al. (2016) Gradually Progressive Spastic Ataxia in a Young Man: Steadily Unsteady. Jama Neurology |
| Williams JR, Trias E, Beilby PR, et al. (2016) Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SOD(G93A) mice co-expressing the copper-chaperone-for-SOD. Neurobiology of Disease |
| Son M, Elliott JL. (2014) Mitochondrial defects in transgenic mice expressing Cu,Zn superoxide dismutase mutations: the role of copper chaperone for SOD1. Journal of the Neurological Sciences. 336: 1-7 |
| Son M, Srikanth U, Puttaparthi K, et al. (2011) Biochemical properties and in vivo effects of the SOD1 zinc-binding site mutant (H80G). Journal of Neurochemistry. 118: 891-901 |
| Sadeghian H, O'Suilleabhain PE, Battiste J, et al. (2011) Huntington chorea presenting with motor neuron disease. Archives of Neurology. 68: 650-2 |
| Stallings NR, Puttaparthi K, Luther CM, et al. (2010) Progressive motor weakness in transgenic mice expressing human TDP-43. Neurobiology of Disease. 40: 404-14 |
| Williams AH, Valdez G, Moresi V, et al. (2009) MicroRNA-206 delays ALS progression and promotes regeneration of neuromuscular synapses in mice. Science (New York, N.Y.). 326: 1549-54 |
| Son M, Fu Q, Puttaparthi K, et al. (2009) Redox susceptibility of SOD1 mutants is associated with the differential response to CCS over-expression in vivo. Neurobiology of Disease. 34: 155-62 |
| Proescher JB, Son M, Elliott JL, et al. (2008) Biological effects of CCS in the absence of SOD1 enzyme activation: implications for disease in a mouse model for ALS. Human Molecular Genetics. 17: 1728-37 |
| Son M, Leary SC, Romain N, et al. (2008) Isolated cytochrome c oxidase deficiency in G93A SOD1 mice overexpressing CCS protein. The Journal of Biological Chemistry. 283: 12267-75 |