Oscar Solis

Cajal Institute, CSIC, Madrid 
Parkinson disease, dyskinesia, dendritic spines
"Oscar Solis"
Mean distance: 14.97 (cluster 11)
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Kouvaros S, Bizup B, Solis O, et al. (2023) A CRE/DRE dual recombinase transgenic mouse reveals synaptic zinc-mediated thalamocortical neuromodulation. Science Advances. 9: eadf3525
Espadas I, Ortiz O, García-Sanz P, et al. (2020) Dopamine D2R is Required for Hippocampal-dependent Memory and Plasticity at the CA3-CA1 Synapse. Cerebral Cortex (New York, N.Y. : 1991)
Suarez LM, Solis O, Sanz-Magro A, et al. (2020) Dopamine D1 Receptors Regulate Spines in Striatal Direct-Pathway and Indirect-Pathway Neurons. Movement Disorders : Official Journal of the Movement Disorder Society
Solís O, García-Sanz P, Martín AB, et al. (2019) Behavioral sensitization and cellular responses to psychostimulants are reduced in D2R knockout mice. Addiction Biology. e12840
Keifman E, Ruiz de Diego I, Pafundo DE, et al. (2019) Optostimulation of striatonigral terminals in substantia nigra induces dyskinesia that increases after L-DOPA in a mouse model of Parkinson's disease. British Journal of Pharmacology
Ruiz-DeDiego I, Fasano S, Solís O, et al. (2018) Genetic enhancement of Ras-ERK pathway does not aggravate L-DOPA-induced dyskinesia in mice but prevents the decrease induced by lovastatin. Scientific Reports. 8: 15381
García-Montes JR, Solís O, Enríquez-Traba J, et al. (2018) Genetic Knockdown of mGluR5 in Striatal D1R-Containing Neurons Attenuates L-DOPA-Induced Dyskinesia in Aphakia Mice. Molecular Neurobiology
Solís O, Moratalla R. (2018) Dopamine receptors: homomeric and heteromeric complexes in L-DOPA-induced dyskinesia. Journal of Neural Transmission (Vienna, Austria : 1996)
Solís O, García-Montes JR, Garcia-Sanz P, et al. (2017) Human COMT over-expression confers a heightened susceptibility to dyskinesia in mice. Neurobiology of Disease
Moratalla R, Solís O, Suárez LM. (2017) Morphological Plasticity in the Striatum Associated With Dopamine Dysfunction Handbook of Behavioral Neuroscience. 24: 755-770
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