Ning Gu
Affiliations: | University of Oslo, Norway, Oslo, Oslo, Norway |
Area:
electrophysiology, potassium channels, hippocampusGoogle:
"Ning Gu"Mean distance: 14.96 (cluster 6) | S | N | B | C | P |
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Publications
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| Gu Y, Li Y, Ma B, et al. (2023) Probing Conformational Transition of TRPV5 Induced by Mechanical Force Using Coarse-Grained Molecular Dynamics. Journal of Chemical Information and Modeling |
| Liao Y, Kristiansen AM, Oksvold CP, et al. (2010) Neuronal Ca2+-activated K+ channels limit brain infarction and promote survival. Plos One. 5: e15601 |
| Gu N, Hu H, Vervaeke K, et al. (2008) SK (KCa2) channels do not control somatic excitability in CA1 pyramidal neurons but can be activated by dendritic excitatory synapses and regulate their impact. Journal of Neurophysiology. 100: 2589-604 |
| Gu N, Vervaeke K, Storm JF. (2007) BK potassium channels facilitate high-frequency firing and cause early spike frequency adaptation in rat CA1 hippocampal pyramidal cells. The Journal of Physiology. 580: 859-82 |
| Vervaeke K, Gu N, Agdestein C, et al. (2006) Kv7/KCNQ/M-channels in rat glutamatergic hippocampal axons and their role in regulation of excitability and transmitter release. The Journal of Physiology. 576: 235-56 |
| Gu N, Vervaeke K, Hu H, et al. (2005) Kv7/KCNQ/M and HCN/h, but not KCa2/SK channels, contribute to the somatic medium after-hyperpolarization and excitability control in CA1 hippocampal pyramidal cells. The Journal of Physiology. 566: 689-715 |
| Hu H, Shao LR, Chavoshy S, et al. (2001) Presynaptic Ca2+-activated K+ channels in glutamatergic hippocampal terminals and their role in spike repolarization and regulation of transmitter release. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 21: 9585-97 |