Dena A. Jacob, Ph.D.

Affiliations: 
2008 Neuroscience & Behavior University of Massachusetts, Amherst, Amherst, MA 
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"Dena Jacob"
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Nancy G. Forger grad student 2008 U Mass Amherst
 (The role of cell death in the development of a sexually dimorphic neuromuscular system.)
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Publications

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Jablonski MR, Jacob DA, Campos C, et al. (2012) Selective increase of two ABC drug efflux transporters at the blood-spinal cord barrier suggests induced pharmacoresistance in ALS. Neurobiology of Disease. 47: 194-200
Jablonski MR, Cooper L, Jacob DA. (2011) NMDA receptor excitotoxicity: impact on phosphatase activity and phosphorylation of huntingtin. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 4357-9
Jacob DA, Ray T, Bengston CL, et al. (2008) The role of cell death in sexually dimorphic muscle development: male-specific muscles are retained in female bax/bak knockout mice. Developmental Neurobiology. 68: 1303-14
Gotsiridze T, Kang N, Jacob D, et al. (2007) Development of sex differences in the principal nucleus of the bed nucleus of the stria terminalis of mice: role of Bax-dependent cell death. Developmental Neurobiology. 67: 355-62
Jacob DA, Bengston CL, Forger NG. (2005) Effects of Bax gene deletion on muscle and motoneuron degeneration in a sexually dimorphic neuromuscular system. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 5638-44
Forger NG, Rosen GJ, Waters EM, et al. (2004) Deletion of Bax eliminates sex differences in the mouse forebrain. Proceedings of the National Academy of Sciences of the United States of America. 101: 13666-71
McAnally JL, Xu L, Villain M, et al. (2001) The role of adjuvants in the efficacy of a peptide vaccine for myasthenia gravis. Experimental Biology and Medicine (Maywood, N.J.). 226: 307-11
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