Kathleen Dunlap
Affiliations: | Tufts University, Boston |
Area:
Calcium channel modulationWebsite:
http://www.tufts.edu/sackler/facultyIntros/dunlapK.htmlGoogle:
"Kathleen Dunlap"Mean distance: 14.09 (cluster 11)
Children
Sign in to add trainee| M. S. AtKisson | grad student | Tufts | |
| Daniel H. Cox | grad student | Tufts Medical School | |
| Yu-Dong Zhou | grad student | 1997-2003 | Tufts |
| George G. Holz | post-doc | Tufts Medical School |
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Publications
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| Damiano CR, Cockrell DC, Dunlap K, et al. (2015) Neural mechanisms of negative reinforcement in children and adolescents with autism spectrum disorders. Journal of Neurodevelopmental Disorders. 7: 12 |
| Damiano CR, Aloi J, Dunlap K, et al. (2014) Association between the oxytocin receptor (OXTR) gene and mesolimbic responses to rewards. Molecular Autism. 5: 7 |
| Erickson MA, Haburćák M, Smukler L, et al. (2007) Altered functional expression of Purkinje cell calcium channels precedes motor dysfunction in tottering mice. Neuroscience. 150: 547-55 |
| Dunlap K. (2007) Calcium channels are models of self-control. The Journal of General Physiology. 129: 379-83 |
| Ikeda SR, Dunlap K. (2007) Calcium channels diversify their signaling portfolio. Nature Neuroscience. 10: 269-71 |
| Olsen DP, Dunlap K, Jacob MH. (2007) Kainate receptors and RNA editing in cholinergic neurons. Journal of Neurochemistry. 101: 327-41 |
| Tosetti P, Dunlap K. (2004) Assays of RGS3 activation and modulation. Methods in Enzymology. 390: 99-119 |
| Tosetti P, Pathak N, Jacob MH, et al. (2003) RGS3 mediates a calcium-dependent termination of G protein signaling in sensory neurons. Proceedings of the National Academy of Sciences of the United States of America. 100: 7337-42 |
| Tosetti P, Parente V, Taglietti V, et al. (2003) Chick RGS2L demonstrates concentration-dependent selectivity for pertussis toxin-sensitive and -insensitive pathways that inhibit L-type Ca2+ channels. The Journal of Physiology. 549: 157-69 |
| Zhou YD, Turner TJ, Dunlap K. (2003) Enhanced G protein-dependent modulation of excitatory synaptic transmission in the cerebellum of the Ca2+ channel-mutant mouse, tottering. The Journal of Physiology. 547: 497-507 |