Mochtar Pribadi, Ph.D.
Affiliations: | 2011 | University of California, Los Angeles, Los Angeles, CA |
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Lopez-Gonzalez R, Yang D, Pribadi M, et al. (2019) Partial inhibition of the overactivated Ku80-dependent DNA repair pathway rescues neurodegeneration in -ALS/FTD. Proceedings of the National Academy of Sciences of the United States of America |
Lee SE, Sias AC, Mandelli ML, et al. (2017) Network degeneration and dysfunction in presymptomatic C9ORF72 expansion carriers. Neuroimage. Clinical. 14: 286-297 |
Wang X, Hao L, Saur T, et al. (2016) Forward Genetic Screen in Caenorhabditis elegans Suggests F57A10.2 and acp-4 As Suppressors of C9ORF72 Related Phenotypes. Frontiers in Molecular Neuroscience. 9: 113 |
Vatsavayai SC, Yoon SJ, Gardner RC, et al. (2016) Timing and significance of pathological features in C9orf72 expansion-associated frontotemporal dementia. Brain : a Journal of Neurology |
Watson A, Pribadi M, Chowdari K, et al. (2015) C9orf72 repeat expansions that cause frontotemporal dementia are detectable among patients with psychosis. Psychiatry Research |
Sha SJ, Khazenzon AM, Ghosh PM, et al. (2015) Early-onset Alzheimer's disease versus frontotemporal dementia: resolution with genetic diagnoses? Neurocase. 1-7 |
Naasan G, Rabinovici GD, Ghosh P, et al. (2015) Amyloid in dementia associated with familial FTLD: not an innocent bystander. Neurocase. 1-8 |
Shinagawa S, Naasan G, Karydas AM, et al. (2015) Clinicopathological Study of Patients With C9ORF72-Associated Frontotemporal Dementia Presenting With Delusions. Journal of Geriatric Psychiatry and Neurology. 28: 99-107 |
Lee SE, Khazenzon AM, Trujillo AJ, et al. (2014) Altered network connectivity in frontotemporal dementia with C9orf72 hexanucleotide repeat expansion. Brain : a Journal of Neurology. 137: 3047-60 |
Fogel BL, Pribadi M, Pi S, et al. (2012) C9ORF72 expansion is not a significant cause of sporadic spinocerebellar ataxia. Movement Disorders : Official Journal of the Movement Disorder Society. 27: 1832-3 |