Wayne W. Poon, Ph.D.

Affiliations: 
2001 University of California, Los Angeles, Los Angeles, CA 
Area:
Biochemistry, Chemical Biology, Proteomics and Bioinformatics, Systems Biology and Biological Regulation, Metabolism, Aging and Development
Google:
"Wayne Poon"
Mean distance: 5308.4
 

Parents

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Catherine F. Clarke grad student 2001 UCLA
 (Studies on ubiquinone biosynthesis in Escherichia coli and Saccharomyces cerevisiae.)
Carl W. Cotman post-doc UC Irvine (Neurotree)
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Publications

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Rajabli F, Benchek P, Tosto G, et al. (2023) Multi-ancestry genome-wide meta-analysis of 56,241 individuals identifies and nominates ancestry-specific loci , , and as novel risk loci for Alzheimer's disease: the Alzheimer's Disease Genetics Consortium. Medrxiv : the Preprint Server For Health Sciences
Reyes-Ortiz AM, Abud EM, Burns MS, et al. (2022) Single-nuclei transcriptome analysis of Huntington disease iPSC and mouse astrocytes implicates maturation and functional deficits. Iscience. 26: 105732
Tcw J, Qian L, Pipalia NH, et al. (2022) Cholesterol and matrisome pathways dysregulated in astrocytes and microglia. Cell. 185: 2213-2233.e25
You Y, Muraoka S, Jedrychowski MP, et al. (2022) Human neural cell type-specific extracellular vesicle proteome defines disease-related molecules associated with activated astrocytes in Alzheimer's disease brain. Journal of Extracellular Vesicles. 11: e12183
Farrell K, Kim S, Han N, et al. (2021) Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta Neuropathologica
Novikova G, Kapoor M, Tcw J, et al. (2021) Integration of Alzheimer's disease genetics and myeloid genomics identifies disease risk regulatory elements and genes. Nature Communications. 12: 1610
Walker JM, Richardson TE, Farrell K, et al. (2020) Early Selective Vulnerability of the CA2 Hippocampal Subfield in Primary Age-Related Tauopathy. Journal of Neuropathology and Experimental Neurology
Bilousova T, Melnik M, Miyoshi E, et al. (2019) Apolipoprotein E/Aβ Complex Accumulates in AD Cortical Synapses via apoE Receptors and Is Enhanced by APOE4. The American Journal of Pathology
Anttila V, Bulik-Sullivan B, et al. (2018) Analysis of shared heritability in common disorders of the brain. Science (New York, N.Y.). 360
Rajic AJ, Pressman PS, Poon WW. (2018) P1-491: Comparative Analysis Of Clinicopathological Threshold Modeling Using Quantitative Nft Loads, Vascular Disease, And Age-Related Gliosis Against Braak Tangle Stage In Pure Alzheimer Disease Autopsy Cases Alzheimers & Dementia. 14
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