Natalie Landman, Ph.D.
|2007||Columbia University, New York, NY|
Area:Neurogenetics, Neural Degeneration and Repair, Disease
Mean distance: 35622
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|McIntire LB, Landman N, Kang MS, et al. (2013) Phenotypic assays for β-amyloid in mouse embryonic stem cell-derived neurons. Chemistry & Biology. 20: 956-67|
|Kang MS, Baek SH, Chun YS, et al. (2013) Modulation of lipid kinase PI4KIIÎ± activity and lipid raft association of presenilin 1 underlies Î³-secretase inhibition by ginsenoside (20S)-Rg3. The Journal of Biological Chemistry. 288: 20868-82|
|Landman N, Jeong SY, Shin SY, et al. (2006) Presenilin mutations linked to familial Alzheimer's disease cause an imbalance in phosphatidylinositol 4,5-bisphosphate metabolism. Proceedings of the National Academy of Sciences of the United States of America. 103: 19524-9|
|Landman N, Kim TW. (2004) Got RIP? Presenilin-dependent intramembrane proteolysis in growth factor receptor signaling. Cytokine & Growth Factor Reviews. 15: 337-51|
|Jung KM, Tan S, Landman N, et al. (2003) Regulated intramembrane proteolysis of the p75 neurotrophin receptor modulates its association with the TrkA receptor. The Journal of Biological Chemistry. 278: 42161-9|