Carl Weitlauf
Affiliations: | Vanderbilt University, Nashville, TN |
Area:
electrophysiology, addiction, epilepsyGoogle:
"Carl Weitlauf"Mean distance: 14.35 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Danny Winder | grad student | 2000-2005 | Vanderbilt | |
| (Ethanol-sensitive NMDAR-dependent synaptic plasticity and synaptic transmission in the bed nucleus of the stria terminals.) | ||||
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Publications
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| Wills TA, Klug JR, Silberman Y, et al. (2012) GluN2B subunit deletion reveals key role in acute and chronic ethanol sensitivity of glutamate synapses in bed nucleus of the stria terminalis. Proceedings of the National Academy of Sciences of the United States of America. 109: E278-87 |
| Weitlauf C, Woodward JJ. (2008) Ethanol selectively attenuates NMDAR-mediated synaptic transmission in the prefrontal cortex. Alcoholism, Clinical and Experimental Research. 32: 690-8 |
| Weitlauf C, Honse Y, Auberson YP, et al. (2005) Activation of NR2A-containing NMDA receptors is not obligatory for NMDA receptor-dependent long-term potentiation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 8386-90 |
| Weitlauf C, Egli RE, Grueter BA, et al. (2004) High-frequency stimulation induces ethanol-sensitive long-term potentiation at glutamatergic synapses in the dorsolateral bed nucleus of the stria terminalis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 24: 5741-7 |
| Weitlauf C, Winder D. (2001) Calcineurin, synaptic plasticity, and memory. Thescientificworldjournal. 1: 530-3 |
| Malleret G, Haditsch U, Genoux D, et al. (2001) Inducible and reversible enhancement of learning, memory, and long-term potentiation by genetic inhibition of calcineurin. Cell. 104: 675-86 |