Laura Manuelidis
Affiliations: | Yale University, New Haven, CT |
Area:
dementiasGoogle:
"Laura Manuelidis"Bio:
MD
PROF HEAD NEUROPATH
NEUROPHATHOLOGY SURGERY SEC
333 CEDAR ST
NEW HAVEN, CT 65103206
United States
Mean distance: 18.72 (cluster 6)
Children
Sign in to add trainee| Christopher A. Baker | grad student | 2002 | Yale |
| Thomas Cremer | research scientist | 1986-1988 | Yale (Chemistry Tree) |
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Publications
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| Szigeti-Buck K, Manuelidis L. (2019) Prokaryotic SPHINX replication sequences are conserved in mammalian brain and participate in neurodegeneration. Journal of Cellular Biochemistry |
| Manuelidis L. (2018) Prokaryotic SPHINX 1.8 REP protein is tissue-specific and expressed in human germline cells. Journal of Cellular Biochemistry |
| Yeh YH, Gunasekharan V, Manuelidis L. (2017) A prokaryotic viral sequence is expressed and conserved in mammalian brain. Proceedings of the National Academy of Sciences of the United States of America |
| Botsios S, Manuelidis L. (2016) CJD and Scrapie Require Agent-Associated Nucleic Acids for Infection. Journal of Cellular Biochemistry |
| Botsios S, Tittman S, Manuelidis L. (2015) Rapid chemical decontamination of infectious CJD and scrapie particles parallels treatments known to disrupt microbes and biofilms. Virulence. 0 |
| Kipkorir T, Colangelo CM, Manuelidis L. (2015) Proteomic analysis of host brain components that bind to infectious particles in Creutzfeldt-Jakob disease. Proteomics. 15: 2983-98 |
| Kipkorir T, Tittman S, Botsios S, et al. (2014) Highly infectious CJD particles lack prion protein but contain many viral-linked peptides by LC-MS/MS. Journal of Cellular Biochemistry. 115: 2012-21 |
| Manuelidis L. (2013) Infectious particles, stress, and induced prion amyloids: a unifying perspective. Virulence. 4: 373-83 |
| Miyazawa K, Kipkorir T, Tittman S, et al. (2012) Continuous production of prions after infectious particles are eliminated: implications for Alzheimer's disease. Plos One. 7: e35471 |
| Miyazawa K, Emmerling K, Manuelidis L. (2011) High CJD infectivity remains after prion protein is destroyed. Journal of Cellular Biochemistry. 112: 3630-7 |