Andrew A. Welcher

Amgen Inc., Thousand Oaks, CA, United States 
Neurotrophins, tyrosine kinase receptors, myelin proteins, peripheral neuropathy
"Andrew Welcher"
Mean distance: 15.79 (cluster 11)
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Kraus DM, Elliott GS, Chute H, et al. (2006) CSMD1 is a novel multiple domain complement-regulatory protein highly expressed in the central nervous system and epithelial tissues. Journal of Immunology (Baltimore, Md. : 1950). 176: 4419-30
Schweitzer B, Taylor V, Welcher AA, et al. (1998) Neural membrane protein 35 (NMP35): a novel member of a gene family which is highly expressed in the adult nervous system. Molecular and Cellular Neurosciences. 11: 260-73
Zhou H, Welcher AA, Shooter EM. (1997) BDNF/NT4-5 receptor TrkB and cadherin participate in cell-cell adhesion. Journal of Neuroscience Research. 49: 281-91
Lobsiger CS, Magyar JP, Taylor V, et al. (1996) Identification and characterization of a cDNA and the structural gene encoding the mouse epithelial membrane protein-1. Genomics. 36: 379-87
De León M, Welcher AA, Nahin RH, et al. (1996) Fatty acid binding protein is induced in neurons of the dorsal root ganglia after peripheral nerve injury. Journal of Neuroscience Research. 44: 283-92
Snipes GJ, Suter U, Welcher AA, et al. (1995) The molecular basis of the neuropathies of mouse and human. Progress in Brain Research. 105: 319-25
Taylor V, Welcher AA, Program AE, et al. (1995) Epithelial membrane protein-1, peripheral myelin protein 22, and lens membrane protein 20 define a novel gene family. The Journal of Biological Chemistry. 270: 28824-33
Suter U, Snipes GJ, Schoener-Scott R, et al. (1994) Regulation of tissue-specific expression of alternative peripheral myelin protein-22 (PMP22) gene transcripts by two promoters. The Journal of Biological Chemistry. 269: 25795-808
Roa BB, Garcia CA, Suter U, et al. (1994) Charcot-Marie-tooth disease type 1A Association with a spontaneous point mutation in the PMP22 gene Pediatric Nephrology. 8: 284
Roa BB, Garcia CA, Suter U, et al. (1993) Charcot-Marie-Tooth disease type 1A. Association with a spontaneous point mutation in the PMP22 gene. The New England Journal of Medicine. 329: 96-101
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